- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1474-760X
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Taking the RISC of exiting naïve pluripotency ; volume:17 ; number:1 ; day:16 ; month:5 ; year:2016 ; pages:1-4 ; date:12.2016
Genome biology ; 17, Heft 1 (16.5.2016), 1-4, 12.2016
- Creator
-
Guallar, Diana
- Contributor
-
Wang, Jianlong
SpringerLink (Online service)
- DOI
-
10.1186/s13059-016-0979-z
- URN
-
urn:nbn:de:1111-2016051712021
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:56 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Guallar, Diana
- Wang, Jianlong
- SpringerLink (Online service)
Other Objects (12)
Epigenetic inheritance is gated by naïve pluripotency and Dppa2
Cpt1a Drives primed-to-naïve pluripotency transition through lipid remodeling
Risc
RISC-mediated control of selected chromatin regulators stabilizes ground state pluripotency of mouse embryonic stem cells
Negative feedback via RSK modulates Erk‐dependent progression from naïve pluripotency
Esrrb extinction triggers dismantling of naïve pluripotency and marks commitment to differentiation
Transient upregulation of IRF1 during exit from naive pluripotency confers viral protection
MTCH2-mediated mitochondrial fusion drives exit from naïve pluripotency in embryonic stem cells
Cooperative genetic networks drive embryonic stem cell transition from naïve to formative pluripotency
Das RISC-Konzept
Prdm14 promotes germline fate and naive pluripotency by repressing FGF signalling and DNA methylation
Stat3 promotes mitochondrial transcription and oxidative respiration during maintenance and induction of naive pluripotency
Epigenetic inheritance is gated by naïve pluripotency and Dppa2
Cpt1a Drives primed-to-naïve pluripotency transition through lipid remodeling
Risc
RISC-mediated control of selected chromatin regulators stabilizes ground state pluripotency of mouse embryonic stem cells
Negative feedback via RSK modulates Erk‐dependent progression from naïve pluripotency
Esrrb extinction triggers dismantling of naïve pluripotency and marks commitment to differentiation
Transient upregulation of IRF1 during exit from naive pluripotency confers viral protection
MTCH2-mediated mitochondrial fusion drives exit from naïve pluripotency in embryonic stem cells
Cooperative genetic networks drive embryonic stem cell transition from naïve to formative pluripotency
Das RISC-Konzept
Prdm14 promotes germline fate and naive pluripotency by repressing FGF signalling and DNA methylation
Stat3 promotes mitochondrial transcription and oxidative respiration during maintenance and induction of naive pluripotency
Epigenetic inheritance is gated by naïve pluripotency and Dppa2
Cpt1a Drives primed-to-naïve pluripotency transition through lipid remodeling
Risc
RISC-mediated control of selected chromatin regulators stabilizes ground state pluripotency of mouse embryonic stem cells
Negative feedback via RSK modulates Erk‐dependent progression from naïve pluripotency
Esrrb extinction triggers dismantling of naïve pluripotency and marks commitment to differentiation
Transient upregulation of IRF1 during exit from naive pluripotency confers viral protection
MTCH2-mediated mitochondrial fusion drives exit from naïve pluripotency in embryonic stem cells
Cooperative genetic networks drive embryonic stem cell transition from naïve to formative pluripotency
Das RISC-Konzept
Prdm14 promotes germline fate and naive pluripotency by repressing FGF signalling and DNA methylation