Dysfunctional adipocytes promote tumor progression through YAP/TAZ-dependent cancer-associated adipocyte transformation
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Dysfunctional adipocytes promote tumor progression through YAP/TAZ-dependent cancer-associated adipocyte transformation ; volume:15 ; number:1 ; day:14 ; month:5 ; year:2024 ; pages:1-16 ; date:12.2024
Nature Communications ; 15, Heft 1 (14.5.2024), 1-16, 12.2024
- Creator
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Song, Yaechan
Na, Heeju
Lee, Seung Eon
Kim, You Min
Moon, Jihyun
Nam, Tae Wook
Ji, Yul
Jin, Young
Park, Jae Hyung
Cho, Seok Chan
Lee, Jaehoon
Hwang, Daehee
Ha, Sang-Jun
Park, Hyun Woo
Kim, Jae Bum
Lee, Han-Woong
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41467-024-48179-3
- URN
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urn:nbn:de:101:1-2409232103206.756915357729
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:24 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Song, Yaechan
- Na, Heeju
- Lee, Seung Eon
- Kim, You Min
- Moon, Jihyun
- Nam, Tae Wook
- Ji, Yul
- Jin, Young
- Park, Jae Hyung
- Cho, Seok Chan
- Lee, Jaehoon
- Hwang, Daehee
- Ha, Sang-Jun
- Park, Hyun Woo
- Kim, Jae Bum
- Lee, Han-Woong
- SpringerLink (Online service)
Other Objects (12)
Author Correction: Dysfunctional adipocytes promote tumor progression through YAP/TAZ-dependent cancer-associated adipocyte transformation
YAP/TAZ drives Notch and angiogenesis mechanoregulation in silico
Hippo-YAP/TAZ signaling in neural crest development and neuroblastoma
Role of YAP/TAZ in endothelial cell growth and metabolism
DNMT3A Cooperates with YAP/TAZ to Drive Gallbladder Cancer Metastasis
The role of YAP/TAZ activity in cancer metabolic reprogramming
AMPK stimulation inhibits YAP/TAZ signaling to ameliorate hepatic fibrosis
YAP and TAZ are dispensable for physiological and malignant haematopoiesis
A feed forward loop enforces YAP/TAZ signaling during tumorigenesis
YAP/TAZ interacts with RBM39 to confer resistance against indisulam
Advanced glycation end-products accelerate amyloid deposits in adipocyte’s lipid droplets
Targeting the YAP/TAZ Hippo signaling pathway for oral cancer treatment
Author Correction: Dysfunctional adipocytes promote tumor progression through YAP/TAZ-dependent cancer-associated adipocyte transformation
YAP/TAZ drives Notch and angiogenesis mechanoregulation in silico
Hippo-YAP/TAZ signaling in neural crest development and neuroblastoma
Role of YAP/TAZ in endothelial cell growth and metabolism
DNMT3A Cooperates with YAP/TAZ to Drive Gallbladder Cancer Metastasis
The role of YAP/TAZ activity in cancer metabolic reprogramming
AMPK stimulation inhibits YAP/TAZ signaling to ameliorate hepatic fibrosis
YAP and TAZ are dispensable for physiological and malignant haematopoiesis
A feed forward loop enforces YAP/TAZ signaling during tumorigenesis
YAP/TAZ interacts with RBM39 to confer resistance against indisulam
Advanced glycation end-products accelerate amyloid deposits in adipocyte’s lipid droplets
Targeting the YAP/TAZ Hippo signaling pathway for oral cancer treatment
Author Correction: Dysfunctional adipocytes promote tumor progression through YAP/TAZ-dependent cancer-associated adipocyte transformation
YAP/TAZ drives Notch and angiogenesis mechanoregulation in silico
Hippo-YAP/TAZ signaling in neural crest development and neuroblastoma
Role of YAP/TAZ in endothelial cell growth and metabolism
DNMT3A Cooperates with YAP/TAZ to Drive Gallbladder Cancer Metastasis
The role of YAP/TAZ activity in cancer metabolic reprogramming
AMPK stimulation inhibits YAP/TAZ signaling to ameliorate hepatic fibrosis
YAP and TAZ are dispensable for physiological and malignant haematopoiesis
A feed forward loop enforces YAP/TAZ signaling during tumorigenesis
YAP/TAZ interacts with RBM39 to confer resistance against indisulam
Advanced glycation end-products accelerate amyloid deposits in adipocyte’s lipid droplets