BACE1 regulates expression of Clusterin in astrocytes for enhancing clearance of β-amyloid peptides
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
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Online-Ressource, 1 online resource.
- Sprache
-
Englisch
- Erschienen in
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BACE1 regulates expression of Clusterin in astrocytes for enhancing clearance of β-amyloid peptides ; volume:18 ; number:1 ; day:4 ; month:5 ; year:2023 ; pages:1-20 ; date:12.2023
Molecular neurodegeneration ; 18, Heft 1 (4.5.2023), 1-20, 12.2023
- Urheber
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Zhou, John
Singh, Neeraj
Galske, James
Hudobenko, Jacob
Hu, Xiangyou
Yan, Riqiang
- Beteiligte Personen und Organisationen
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SpringerLink (Online service)
- DOI
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10.1186/s13024-023-00611-w
- URN
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urn:nbn:de:101:1-2023090608503922415580
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
- 14.08.2025, 10:58 MESZ
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Beteiligte
- Zhou, John
- Singh, Neeraj
- Galske, James
- Hudobenko, Jacob
- Hu, Xiangyou
- Yan, Riqiang
- SpringerLink (Online service)
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Non-neuronal and neuronal BACE1 elevation in association with angiopathic and leptomeningeal β-amyloid deposition in the human brain
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BACE1 elevation engendered by GGA3 deletion increases β-amyloid pathology in association with APP elevation and decreased CHL1 processing in 5XFAD mice
The effect of BACE1-AS on β-amyloid generation by regulating BACE1 mRNA expression
BACE1 inhibition more effectively suppresses initiation than progression of β-amyloid pathology
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β-Amyloid promotes accumulation of lipid peroxides by inhibiting CD36-mediated clearance of oxidized lipoproteins
Sorting nexin 12 interacts with BACE1 and regulates BACE1-mediated APP processing
Aβ34 is a BACE1-derived degradation intermediate associated with amyloid clearance and Alzheimer’s disease progression
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