Pharmacologic antagonism of CB1 receptors improves electrophysiological alterations in Purkinje cells exposed to 3-AP
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
-
Pharmacologic antagonism of CB1 receptors improves electrophysiological alterations in Purkinje cells exposed to 3-AP ; volume:24 ; number:1 ; day:3 ; month:3 ; year:2023 ; pages:1-11 ; date:12.2023
BMC neuroscience ; 24, Heft 1 (3.3.2023), 1-11, 12.2023
- Creator
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Ranjbar, Hoda
Soti, Monavareh
Kohlmeier, Kristi A.
Janahmadi, Mahyar
Shabani, Mohammad
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12868-023-00786-4
- URN
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urn:nbn:de:101:1-2024030108334612201193
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:50 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Ranjbar, Hoda
- Soti, Monavareh
- Kohlmeier, Kristi A.
- Janahmadi, Mahyar
- Shabani, Mohammad
- SpringerLink (Online service)
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Impression C.B.1
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Impression C.B.1
Electrophysiological in-vivo characterization of regenerating Purkinje neurons in the larval zebrafish cerebellum
Mitragynine (Kratom)-Induced Cognitive Impairments in Mice Resemble Δ9-THC and Morphine Effects: Reversal by Cannabinoid CB1 Receptor Antagonism
Das Purkinje-Phänomen.
Cannabinoid CB1 receptors regulate salivation
Non-pharmacologic therapy of hypertension
mGluR5 antagonism inhibits cocaine reinforcement and relapse by elevation of extracellular glutamate in the nucleus accumbens via a CB1 receptor mechanism
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