Amyloid-β with isomerized Asp7 cytotoxicity is coupled to protein phosphorylation

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2045-2322

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Amyloid-β with isomerized Asp7 cytotoxicity is coupled to protein phosphorylation ; volume:8 ; number:1 ; day:23 ; month:2 ; year:2018 ; pages:1-10 ; date:12.2018
Scientific reports ; 8, Heft 1 (23.2.2018), 1-10, 12.2018

Keyword: Krebs
Onkologie

Creator: Zatsepina, O. G.

Contributor: Kechko, O. I.
Mitkevich, V. A.
Kozin, S. A.
Yurinskaya, M. M.
Vinokurov, M. G.
Serebryakova, M. V.
Rezvykh, A. P.
Evgen’ev, M. B.
Makarov, A. A.
SpringerLink (Online service)

DOI: 10.1038/s41598-018-21815-x

URN: urn:nbn:de:1111-2018041219047

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:59 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Zatsepina, O. G.
Kechko, O. I.
Mitkevich, V. A.
Kozin, S. A.
Yurinskaya, M. M.
Vinokurov, M. G.
Serebryakova, M. V.
Rezvykh, A. P.
Evgen’ev, M. B.
Makarov, A. A.
SpringerLink (Online service)

Other Objects (12)

Bifurcations in coupled amyloid-β aggregation-inflammation systems

Origins of amyloid-β

Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo

Selective amyloid-β lowering agents

Hochschulschrift

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

G-protein coupled estrogen receptor 1, amyloid-β, and tau tangles in older adults

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

Loss of glucocorticoid receptor phosphorylation contributes to cognitive and neurocentric damages of the amyloid-β pathway

The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro

Copper mediated amyloid-β binding to Transthyretin

Corynoxine isomers decrease levels of amyloid-β peptide and amyloid-β precursor protein by promoting autophagy and lysosome biogenesis

Amyloid-β 1–24 C-terminal truncated fragment promotes amyloid-β 1–42 aggregate formation in the healthy brain

Bifurcations in coupled amyloid-β aggregation-inflammation systems

Origins of amyloid-β

Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo

Selective amyloid-β lowering agents

Hochschulschrift

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

G-protein coupled estrogen receptor 1, amyloid-β, and tau tangles in older adults

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

Loss of glucocorticoid receptor phosphorylation contributes to cognitive and neurocentric damages of the amyloid-β pathway

The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro

Copper mediated amyloid-β binding to Transthyretin

Corynoxine isomers decrease levels of amyloid-β peptide and amyloid-β precursor protein by promoting autophagy and lysosome biogenesis

Amyloid-β 1–24 C-terminal truncated fragment promotes amyloid-β 1–42 aggregate formation in the healthy brain

Bifurcations in coupled amyloid-β aggregation-inflammation systems

Origins of amyloid-β

Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo

Selective amyloid-β lowering agents

Hochschulschrift

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

G-protein coupled estrogen receptor 1, amyloid-β, and tau tangles in older adults

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

Loss of glucocorticoid receptor phosphorylation contributes to cognitive and neurocentric damages of the amyloid-β pathway

The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro

Copper mediated amyloid-β binding to Transthyretin

Corynoxine isomers decrease levels of amyloid-β peptide and amyloid-β precursor protein by promoting autophagy and lysosome biogenesis

Amyloid-β 1–24 C-terminal truncated fragment promotes amyloid-β 1–42 aggregate formation in the healthy brain

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read and agree to the privacy policy for the collection of personal data.*

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

Amyloid-β with isomerized Asp7 cytotoxicity is coupled to protein phosphorylation

Show object

References and Relationships

Classification and Topics

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

Bifurcations in coupled amyloid-β aggregation-inflammation systems

Origins of amyloid-β

Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo

Selective amyloid-β lowering agents

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

G-protein coupled estrogen receptor 1, amyloid-β, and tau tangles in older adults

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

Loss of glucocorticoid receptor phosphorylation contributes to cognitive and neurocentric damages of the amyloid-β pathway

The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro

Copper mediated amyloid-β binding to Transthyretin

Corynoxine isomers decrease levels of amyloid-β peptide and amyloid-β precursor protein by promoting autophagy and lysosome biogenesis

Amyloid-β 1–24 C-terminal truncated fragment promotes amyloid-β 1–42 aggregate formation in the healthy brain

Bifurcations in coupled amyloid-β aggregation-inflammation systems

Origins of amyloid-β

Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo

Selective amyloid-β lowering agents

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

G-protein coupled estrogen receptor 1, amyloid-β, and tau tangles in older adults

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

Loss of glucocorticoid receptor phosphorylation contributes to cognitive and neurocentric damages of the amyloid-β pathway

The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro

Copper mediated amyloid-β binding to Transthyretin

Corynoxine isomers decrease levels of amyloid-β peptide and amyloid-β precursor protein by promoting autophagy and lysosome biogenesis

Amyloid-β 1–24 C-terminal truncated fragment promotes amyloid-β 1–42 aggregate formation in the healthy brain

Bifurcations in coupled amyloid-β aggregation-inflammation systems

Origins of amyloid-β

Redundant Gs-coupled serotonin receptors regulate amyloid-β metabolism in vivo

Selective amyloid-β lowering agents

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

G-protein coupled estrogen receptor 1, amyloid-β, and tau tangles in older adults

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

Loss of glucocorticoid receptor phosphorylation contributes to cognitive and neurocentric damages of the amyloid-β pathway

The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro

Copper mediated amyloid-β binding to Transthyretin

Corynoxine isomers decrease levels of amyloid-β peptide and amyloid-β precursor protein by promoting autophagy and lysosome biogenesis

Amyloid-β 1–24 C-terminal truncated fragment promotes amyloid-β 1–42 aggregate formation in the healthy brain

Related objects

Reset password