A three-dimensional collagen construct to model lipopolysaccharide-induced activation of BV2 microglia
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1742-2094
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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A three-dimensional collagen construct to model lipopolysaccharide-induced activation of BV2 microglia ; volume:11 ; number:1 ; day:30 ; month:7 ; year:2014 ; pages:1-10 ; date:12.2014
Journal of neuroinflammation ; 11, Heft 1 (30.7.2014), 1-10, 12.2014
- Creator
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Haw, Randy Tatt Yhew
- Contributor
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Tong, Chih Kong
Yew, Andrew
Lee, Han Chung
Phillips, James B.
Vidyadaran, Sharmili
SpringerLink (Online service)
- DOI
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10.1186/1742-2094-11-134
- URN
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urn:nbn:de:1111-2016032337230
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.20252025, 4:51 PM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Haw, Randy Tatt Yhew
- Tong, Chih Kong
- Yew, Andrew
- Lee, Han Chung
- Phillips, James B.
- Vidyadaran, Sharmili
- SpringerLink (Online service)
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Paroxetine ameliorates lipopolysaccharide-induced microglia activation via differential regulation of MAPK signaling
EP2-PKA signaling is suppressed by triptolide in lipopolysaccharide-induced microglia activation
A Helminth Protease Inhibitor Modulates the Lipopolysaccharide-Induced Proinflammatory Phenotype of Microglia in vitro
The anti-inflammatory role of tissue inhibitor of metalloproteinase-2 in lipopolysaccharide-stimulated microglia
Paroxetine suppresses reactive microglia-mediated but not lipopolysaccharide-induced inflammatory responses in primary astrocytes
A Helminth Protease Inhibitor Modulates the Lipopolysaccharide-Induced Proinflammatory Phenotype of Microglia in vitro
Resveratrol potently reduces prostaglandin E2production and free radical formation in lipopolysaccharide-activated primary rat microglia
Differential expression of E-type prostanoid receptors 2 and 4 in microglia stimulated with lipopolysaccharide
Single-wall carbon nanohorns inhibited activation of microglia induced by lipopolysaccharide through blocking of Sirt3
Minocycline attenuation of rat corpus callosum abnormality mediated by low-dose lipopolysaccharide-induced microglia activation
Reciprocal interactions of mouse bone marrow-derived mesenchymal stem cells and BV2 microglia after lipopolysaccharide stimulation
Azithromycin Protects Oligodendrocyte Progenitor Cells against Lipopolysaccharide-Activated Microglia-Induced Damage
Paroxetine ameliorates lipopolysaccharide-induced microglia activation via differential regulation of MAPK signaling
EP2-PKA signaling is suppressed by triptolide in lipopolysaccharide-induced microglia activation
A Helminth Protease Inhibitor Modulates the Lipopolysaccharide-Induced Proinflammatory Phenotype of Microglia in vitro
The anti-inflammatory role of tissue inhibitor of metalloproteinase-2 in lipopolysaccharide-stimulated microglia
Paroxetine suppresses reactive microglia-mediated but not lipopolysaccharide-induced inflammatory responses in primary astrocytes
A Helminth Protease Inhibitor Modulates the Lipopolysaccharide-Induced Proinflammatory Phenotype of Microglia in vitro
Resveratrol potently reduces prostaglandin E2production and free radical formation in lipopolysaccharide-activated primary rat microglia
Differential expression of E-type prostanoid receptors 2 and 4 in microglia stimulated with lipopolysaccharide
Single-wall carbon nanohorns inhibited activation of microglia induced by lipopolysaccharide through blocking of Sirt3
Minocycline attenuation of rat corpus callosum abnormality mediated by low-dose lipopolysaccharide-induced microglia activation