Enhanced susceptibility of triple transgenic Alzheimer’s disease (3xTg-AD) mice to acute infection
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1742-2094
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Enhanced susceptibility of triple transgenic Alzheimer’s disease (3xTg-AD) mice to acute infection ; volume:14 ; number:1 ; day:11 ; month:3 ; year:2017 ; pages:1-13 ; date:12.2017
Journal of neuroinflammation ; 14, Heft 1 (11.3.2017), 1-13, 12.2017
- Keyword
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Infektion
Infektiologie
- Creator
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Montacute, Rebecca
- Contributor
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Foley, Kerry
Forman, Ruth
Else, Kathryn Jane
Cruickshank, Sheena Margaret
Allan, Stuart McRae
SpringerLink (Online service)
- DOI
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10.1186/s12974-017-0826-5
- URN
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urn:nbn:de:1111-201704027145
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:50 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Montacute, Rebecca
- Foley, Kerry
- Forman, Ruth
- Else, Kathryn Jane
- Cruickshank, Sheena Margaret
- Allan, Stuart McRae
- SpringerLink (Online service)
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Short-term resistance exercise inhibits neuroinflammation and attenuates neuropathological changes in 3xTg Alzheimer’s disease mice
Exercise and resveratrol increase fracture resistance in the 3xTg-AD mouse model of Alzheimer’s disease
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Sex differences in metabolic phenotype and hypothalamic inflammation in the 3xTg-AD mouse model of Alzheimer’s disease
Abundance of Aβ5-xlike immunoreactivity in transgenic 5XFAD, APP/PS1KI and 3xTG mice, sporadic and familial Alzheimer’s disease
Impaired Spatial Reorientation in the 3xTg-AD Mouse Model of Alzheimer’s Disease
A multi-targeting immunotherapy ameliorates multiple facets of Alzheimer’s disease in 3xTg mice
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Short-term resistance exercise inhibits neuroinflammation and attenuates neuropathological changes in 3xTg Alzheimer’s disease mice
Exercise and resveratrol increase fracture resistance in the 3xTg-AD mouse model of Alzheimer’s disease
Fingolimod Rescues Memory and Improves Pathological Hallmarks in the 3xTg-AD Model of Alzheimer’s Disease
IVIg protects the 3xTg-AD mouse model of Alzheimer’s disease from memory deficit and Aβ pathology
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Immunomodulation targeting of both Aβ and tau pathological conformers ameliorates Alzheimer’s disease pathology in TgSwDI and 3xTg mouse models
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