ß-hydroxybutyrate improves mitochondrial function after transient ischemia in the mouse

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: In: Neurochemical research, 47.2022, 11, S. 3241-3249

Event: Veröffentlichung

(where): Frankfurt am Main

(who): Universitätsbibliothek Johann Christian Senckenberg

(when): 2022

Creator: Lehto, Alina Barbara
Koch, Konrad
Brandes, Johanna
Viel, Christian
Fuchs, Marius
Klein, Jochen

DOI: 10.1007/s11064-022-03637-6

URN: urn:nbn:de:hebis:30:3-696401

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:26 AM CEST

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Associated

Lehto, Alina Barbara
Koch, Konrad
Brandes, Johanna
Viel, Christian
Fuchs, Marius
Klein, Jochen
Universitätsbibliothek Johann Christian Senckenberg

Time of origin

2022

Other Objects (12)

ß-Hydroxybutyrate Improves Mitochondrial Function After Transient Ischemia in the Mouse

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Inflation using hydrogen improves donor lung quality by regulating mitochondrial function during cold ischemia phase

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Mitochondrial involvement in ischemia-reperfusion injury

Regular transient limb ischemia improves endothelial function and inhibits endothelial cell apoptosis to prevent atherosclerosis in rabbit

Diallyl trisulfide improves spinal cord ischemia–reperfusion injury damage by activating AMPK to stabilize mitochondrial function

Genetic deletion of mGlu2 metabotropic glutamate receptors improves the short-term outcome of cerebral transient focal ischemia

Subcutaneous administration of β-hydroxybutyrate improves learning and memory of sepsis surviving mice

Simulated transient hearing loss improves auditory sensitivity

Partial loss of VE-cadherin improves long-term outcome and cerebral blood flow after transient brain ischemia in mice

Isolated transient vertigo: posterior circulation ischemia or benign origin?

Empagliflozin improves renal ischemia–reperfusion injury by reducing inflammation and enhancing mitochondrial fusion through AMPK–OPA1 pathway promotion

ß-Hydroxybutyrate Improves Mitochondrial Function After Transient Ischemia in the Mouse

Ketone body β-hydroxybutyrate (BHB) preserves mitochondrial bioenergetics

Inflation using hydrogen improves donor lung quality by regulating mitochondrial function during cold ischemia phase

Aufsatzsammlung

Mitochondrial involvement in ischemia-reperfusion injury

Regular transient limb ischemia improves endothelial function and inhibits endothelial cell apoptosis to prevent atherosclerosis in rabbit

Diallyl trisulfide improves spinal cord ischemia–reperfusion injury damage by activating AMPK to stabilize mitochondrial function

Genetic deletion of mGlu2 metabotropic glutamate receptors improves the short-term outcome of cerebral transient focal ischemia

Subcutaneous administration of β-hydroxybutyrate improves learning and memory of sepsis surviving mice

Simulated transient hearing loss improves auditory sensitivity

Partial loss of VE-cadherin improves long-term outcome and cerebral blood flow after transient brain ischemia in mice

Isolated transient vertigo: posterior circulation ischemia or benign origin?

Empagliflozin improves renal ischemia–reperfusion injury by reducing inflammation and enhancing mitochondrial fusion through AMPK–OPA1 pathway promotion

ß-Hydroxybutyrate Improves Mitochondrial Function After Transient Ischemia in the Mouse

Ketone body β-hydroxybutyrate (BHB) preserves mitochondrial bioenergetics

Inflation using hydrogen improves donor lung quality by regulating mitochondrial function during cold ischemia phase

Aufsatzsammlung

Mitochondrial involvement in ischemia-reperfusion injury

Regular transient limb ischemia improves endothelial function and inhibits endothelial cell apoptosis to prevent atherosclerosis in rabbit

Diallyl trisulfide improves spinal cord ischemia–reperfusion injury damage by activating AMPK to stabilize mitochondrial function

Genetic deletion of mGlu2 metabotropic glutamate receptors improves the short-term outcome of cerebral transient focal ischemia

Subcutaneous administration of β-hydroxybutyrate improves learning and memory of sepsis surviving mice

Simulated transient hearing loss improves auditory sensitivity

Partial loss of VE-cadherin improves long-term outcome and cerebral blood flow after transient brain ischemia in mice

Isolated transient vertigo: posterior circulation ischemia or benign origin?

Empagliflozin improves renal ischemia–reperfusion injury by reducing inflammation and enhancing mitochondrial fusion through AMPK–OPA1 pathway promotion

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ß-hydroxybutyrate improves mitochondrial function after transient ischemia in the mouse

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Other Objects (12)

ß-Hydroxybutyrate Improves Mitochondrial Function After Transient Ischemia in the Mouse

Ketone body β-hydroxybutyrate (BHB) preserves mitochondrial bioenergetics

Inflation using hydrogen improves donor lung quality by regulating mitochondrial function during cold ischemia phase

Mitochondrial involvement in ischemia-reperfusion injury

Regular transient limb ischemia improves endothelial function and inhibits endothelial cell apoptosis to prevent atherosclerosis in rabbit

Diallyl trisulfide improves spinal cord ischemia–reperfusion injury damage by activating AMPK to stabilize mitochondrial function

Genetic deletion of mGlu2 metabotropic glutamate receptors improves the short-term outcome of cerebral transient focal ischemia

Subcutaneous administration of β-hydroxybutyrate improves learning and memory of sepsis surviving mice

Simulated transient hearing loss improves auditory sensitivity

Partial loss of VE-cadherin improves long-term outcome and cerebral blood flow after transient brain ischemia in mice

Isolated transient vertigo: posterior circulation ischemia or benign origin?

Empagliflozin improves renal ischemia–reperfusion injury by reducing inflammation and enhancing mitochondrial fusion through AMPK–OPA1 pathway promotion

ß-Hydroxybutyrate Improves Mitochondrial Function After Transient Ischemia in the Mouse

Ketone body β-hydroxybutyrate (BHB) preserves mitochondrial bioenergetics

Inflation using hydrogen improves donor lung quality by regulating mitochondrial function during cold ischemia phase

Mitochondrial involvement in ischemia-reperfusion injury

Regular transient limb ischemia improves endothelial function and inhibits endothelial cell apoptosis to prevent atherosclerosis in rabbit

Diallyl trisulfide improves spinal cord ischemia–reperfusion injury damage by activating AMPK to stabilize mitochondrial function

Genetic deletion of mGlu2 metabotropic glutamate receptors improves the short-term outcome of cerebral transient focal ischemia

Subcutaneous administration of β-hydroxybutyrate improves learning and memory of sepsis surviving mice

Simulated transient hearing loss improves auditory sensitivity

Partial loss of VE-cadherin improves long-term outcome and cerebral blood flow after transient brain ischemia in mice

Isolated transient vertigo: posterior circulation ischemia or benign origin?

Empagliflozin improves renal ischemia–reperfusion injury by reducing inflammation and enhancing mitochondrial fusion through AMPK–OPA1 pathway promotion

ß-Hydroxybutyrate Improves Mitochondrial Function After Transient Ischemia in the Mouse

Ketone body β-hydroxybutyrate (BHB) preserves mitochondrial bioenergetics

Inflation using hydrogen improves donor lung quality by regulating mitochondrial function during cold ischemia phase

Mitochondrial involvement in ischemia-reperfusion injury

Regular transient limb ischemia improves endothelial function and inhibits endothelial cell apoptosis to prevent atherosclerosis in rabbit

Diallyl trisulfide improves spinal cord ischemia–reperfusion injury damage by activating AMPK to stabilize mitochondrial function

Genetic deletion of mGlu2 metabotropic glutamate receptors improves the short-term outcome of cerebral transient focal ischemia

Subcutaneous administration of β-hydroxybutyrate improves learning and memory of sepsis surviving mice

Simulated transient hearing loss improves auditory sensitivity

Partial loss of VE-cadherin improves long-term outcome and cerebral blood flow after transient brain ischemia in mice

Isolated transient vertigo: posterior circulation ischemia or benign origin?

Empagliflozin improves renal ischemia–reperfusion injury by reducing inflammation and enhancing mitochondrial fusion through AMPK–OPA1 pathway promotion

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