DNGR‐1 is dispensable for CD8 + T‐cell priming during respiratory syncytial virus infection
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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DNGR‐1 is dispensable for CD8 + T‐cell priming during respiratory syncytial virus infection ; volume:44 ; number:8 ; year:2014 ; pages:2340-2348 ; extent:9
European journal of immunology ; 44, Heft 8 (2014), 2340-2348 (gesamt 9)
- Creator
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Durant, Lydia R.
Pereira, Catherine
Boakye, Aime
Makris, Spyridon
Kausar, Fahima
Goritzka, Michelle
Johansson, Cecilia
- DOI
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10.1002/eji.201444454
- URN
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urn:nbn:de:101:1-2023011806215227713284
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:32 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Durant, Lydia R.
- Pereira, Catherine
- Boakye, Aime
- Makris, Spyridon
- Kausar, Fahima
- Goritzka, Michelle
- Johansson, Cecilia
Other Objects (12)
Compartmentalization and functional characterization of CD8 T-cell immunity in mice infected with respiratory syncytial virus
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miRNA miR-21 is largely dispensable for intrathymic T-cell development
Immunopathology caused by impaired CD8+ T‐cell responses
Immunopathology caused by impaired CD8 + T‐cell responses
The hallmarks of CMV-specific CD8 T-cell differentiation
CD8 T-cell subsets: heterogeneity, functions, and therapeutic potential
Metabolic dynamics instruct CD8 + T‐cell differentiation and functions
Die Bedeutung des Glukosestoffwechsels beim Priming humaner, antigenspezifischer CD8\(^+\) T-Zellen
CD4 T-cell help sustains distinct subpopulations in functional and dysfunctional CD8 T-cell responses.
CD8+ tissue-resident memory T-cell development depends on infection-matching regulatory T-cell types
Targeting CD40 enhances antibody- and CD8-mediated protection against respiratory syncytial virus infection
Compartmentalization and functional characterization of CD8 T-cell immunity in mice infected with respiratory syncytial virus
T-cell receptor avidity of polyclonal CD8+ T-cell populations
miRNA miR-21 is largely dispensable for intrathymic T-cell development
Immunopathology caused by impaired CD8+ T‐cell responses
Immunopathology caused by impaired CD8 + T‐cell responses
The hallmarks of CMV-specific CD8 T-cell differentiation
CD8 T-cell subsets: heterogeneity, functions, and therapeutic potential
Metabolic dynamics instruct CD8 + T‐cell differentiation and functions
Die Bedeutung des Glukosestoffwechsels beim Priming humaner, antigenspezifischer CD8\(^+\) T-Zellen
CD4 T-cell help sustains distinct subpopulations in functional and dysfunctional CD8 T-cell responses.
CD8+ tissue-resident memory T-cell development depends on infection-matching regulatory T-cell types
Targeting CD40 enhances antibody- and CD8-mediated protection against respiratory syncytial virus infection
Compartmentalization and functional characterization of CD8 T-cell immunity in mice infected with respiratory syncytial virus
T-cell receptor avidity of polyclonal CD8+ T-cell populations
miRNA miR-21 is largely dispensable for intrathymic T-cell development
Immunopathology caused by impaired CD8+ T‐cell responses
Immunopathology caused by impaired CD8 + T‐cell responses
The hallmarks of CMV-specific CD8 T-cell differentiation
CD8 T-cell subsets: heterogeneity, functions, and therapeutic potential
Metabolic dynamics instruct CD8 + T‐cell differentiation and functions
Die Bedeutung des Glukosestoffwechsels beim Priming humaner, antigenspezifischer CD8\(^+\) T-Zellen
CD4 T-cell help sustains distinct subpopulations in functional and dysfunctional CD8 T-cell responses.
CD8+ tissue-resident memory T-cell development depends on infection-matching regulatory T-cell types