- Alternative title
-
Die Rolle der EGFR / KRAS nachgeschalteten Zielproteine MEK, EGR1 und KLF5 während der Initiation und Progression des Pankreaskarzinoms
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
München, Technische Universität München, Diss., 2015
- Keyword
-
Krebs
Onkologie
- Event
-
Veröffentlichung
- (where)
-
München
- (who)
-
Universitätsbibliothek der TU München
- (when)
-
2015
- Creator
-
Briel, Thomas
- Contributor
- URN
-
urn:nbn:de:bvb:91-diss-20150206-1220374-0-1
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
25.03.2025, 1:47 PM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
- Briel, Thomas
- Schnieke, Angelika
- Siveke, Jens T.
- Universitätsbibliothek der TU München
Time of origin
- 2015
Other Objects (12)
The role of the EGFR and ERBB4 in acute pancreatitis
The role of E-cadherin in EGFR signal transduction and EGFR-inhibitory treatment in gastric cancer
The role of E-cadherin in EGFR signal transduction and EGFR-inhibitory treatment in gastric cancer
The role of ADAM17-EGFR signaling in regulation of the epidermal differentation
Mathematical model to uncover the role of receptor ubiquitination in dose dependent EGFR trafficking
Characterisation and regulation of the Egfr-Egfr ligand system in fish models for melanoma
Deciphering the mechanistic role of scaffolding protein NEDD9 in the pathogenesis and therapy of EGFR-driven lung adenocarcinoma
Transactivation of the EGFR signal in human cancer cells
Transactivation of the EGFR signal in human cancer cells
Genomic variations in the EGFR pathway in relation to skin toxicity of EGFR inhibitors analyzed by deep sequencing
Der Einfluss der EGFR Expressionsdichte auf die EGFR Antikörper gerichteten Abwehrmechanismen
Unraveling the mechanisms of EGFR-inhibitor associated cutaneous adverse effects.
The role of the EGFR and ERBB4 in acute pancreatitis
The role of E-cadherin in EGFR signal transduction and EGFR-inhibitory treatment in gastric cancer
The role of E-cadherin in EGFR signal transduction and EGFR-inhibitory treatment in gastric cancer
The role of ADAM17-EGFR signaling in regulation of the epidermal differentation
Mathematical model to uncover the role of receptor ubiquitination in dose dependent EGFR trafficking
Characterisation and regulation of the Egfr-Egfr ligand system in fish models for melanoma
Deciphering the mechanistic role of scaffolding protein NEDD9 in the pathogenesis and therapy of EGFR-driven lung adenocarcinoma
Transactivation of the EGFR signal in human cancer cells
Transactivation of the EGFR signal in human cancer cells
Genomic variations in the EGFR pathway in relation to skin toxicity of EGFR inhibitors analyzed by deep sequencing
Der Einfluss der EGFR Expressionsdichte auf die EGFR Antikörper gerichteten Abwehrmechanismen
Unraveling the mechanisms of EGFR-inhibitor associated cutaneous adverse effects.
The role of the EGFR and ERBB4 in acute pancreatitis
The role of E-cadherin in EGFR signal transduction and EGFR-inhibitory treatment in gastric cancer
The role of E-cadherin in EGFR signal transduction and EGFR-inhibitory treatment in gastric cancer
The role of ADAM17-EGFR signaling in regulation of the epidermal differentation
Mathematical model to uncover the role of receptor ubiquitination in dose dependent EGFR trafficking
Characterisation and regulation of the Egfr-Egfr ligand system in fish models for melanoma
Deciphering the mechanistic role of scaffolding protein NEDD9 in the pathogenesis and therapy of EGFR-driven lung adenocarcinoma
Transactivation of the EGFR signal in human cancer cells
Transactivation of the EGFR signal in human cancer cells
Genomic variations in the EGFR pathway in relation to skin toxicity of EGFR inhibitors analyzed by deep sequencing
Der Einfluss der EGFR Expressionsdichte auf die EGFR Antikörper gerichteten Abwehrmechanismen