Understanding the function of NF-κB transcription factor c-Rel in pancreatic cancer
- Weitere Titel
-
Untersuchung der Funktion des NF-κB-Transkriptionsfaktors c-Rel bei Bauchspeicheldrüsenkrebs
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
München, Technische Universität München, Dissertation, 2021
- Schlagwort
-
Bauchspeicheldrüsenkrebs
Krebs
Signaltransduktion
Bauchspeicheldrüse
- Ereignis
-
Veröffentlichung
- (wo)
-
München
- (wer)
-
Universitätsbibliothek der TU München
- (wann)
-
2021
- Urheber
-
Kabacaoglu, Derya
- Beteiligte Personen und Organisationen
- URN
-
urn:nbn:de:bvb:91-diss-20211006-1596373-1-6
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
- 15.08.2025, 07:23 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Kabacaoglu, Derya
- Algül, Hana
- Klingenspor, Martin
- Universitätsbibliothek der TU München
Entstanden
- 2021
Ähnliche Objekte (12)
NF-κB/Rel Transcription factors in pancreatic cancer: focusing on RelA, c-Rel, and RelB
NF-kB c-Rel in Fibroblasten und Fibrose
NF-кB c-Rel modulates pre-fibrotic changes in human fibroblasts
NF-κB RelA is a cell-intrinsic metabolic checkpoint restricting glycolysis
![Structure of the human transcription factor NF-κB [NF-kappaB] P52 homodimer DNA complex](/assets/placeholder/searchResultMediaNoDigitisedMedia.png)
Structure of the human transcription factor NF-κB [NF-kappaB] P52 homodimer DNA complex
Accelerating corneal wound healing using exosome-mediated targeting of NF-κB c-Rel
Untersuchungen zu Struktur und Funktion des RelB-Proteins, einer Untereinheit des Transkriptionsfaktors NF-κB [NF-Kappa-B]
Multilevel Regulation of NF‐κB Signaling by NSD2 Suppresses Kras ‐Driven Pancreatic Tumorigenesis
Centrosomal protein 55 activates NF-κB signalling and promotes pancreatic cancer cells aggressiveness
GSK3β modulates NF-κB activation and RelB degradation through site-specific phosphorylation of BCL10
Signal transduction mechanisms involved in S100A4-induced activation of the transcription factor NF-κB
Breast cancer metastasis suppressor 1 (BRMS1) inhibits osteopontin transcription by abrogating NF-κB activation
NF-κB/Rel Transcription factors in pancreatic cancer: focusing on RelA, c-Rel, and RelB
NF-kB c-Rel in Fibroblasten und Fibrose
NF-кB c-Rel modulates pre-fibrotic changes in human fibroblasts
NF-κB RelA is a cell-intrinsic metabolic checkpoint restricting glycolysis
Structure of the human transcription factor NF-κB [NF-kappaB] P52 homodimer DNA complex
Accelerating corneal wound healing using exosome-mediated targeting of NF-κB c-Rel
Untersuchungen zu Struktur und Funktion des RelB-Proteins, einer Untereinheit des Transkriptionsfaktors NF-κB [NF-Kappa-B]
Multilevel Regulation of NF‐κB Signaling by NSD2 Suppresses Kras ‐Driven Pancreatic Tumorigenesis
Centrosomal protein 55 activates NF-κB signalling and promotes pancreatic cancer cells aggressiveness
GSK3β modulates NF-κB activation and RelB degradation through site-specific phosphorylation of BCL10
Signal transduction mechanisms involved in S100A4-induced activation of the transcription factor NF-κB
Breast cancer metastasis suppressor 1 (BRMS1) inhibits osteopontin transcription by abrogating NF-κB activation
NF-κB/Rel Transcription factors in pancreatic cancer: focusing on RelA, c-Rel, and RelB
NF-kB c-Rel in Fibroblasten und Fibrose
NF-кB c-Rel modulates pre-fibrotic changes in human fibroblasts
NF-κB RelA is a cell-intrinsic metabolic checkpoint restricting glycolysis
Structure of the human transcription factor NF-κB [NF-kappaB] P52 homodimer DNA complex
Accelerating corneal wound healing using exosome-mediated targeting of NF-κB c-Rel
Untersuchungen zu Struktur und Funktion des RelB-Proteins, einer Untereinheit des Transkriptionsfaktors NF-κB [NF-Kappa-B]
Multilevel Regulation of NF‐κB Signaling by NSD2 Suppresses Kras ‐Driven Pancreatic Tumorigenesis
Centrosomal protein 55 activates NF-κB signalling and promotes pancreatic cancer cells aggressiveness
GSK3β modulates NF-κB activation and RelB degradation through site-specific phosphorylation of BCL10
Signal transduction mechanisms involved in S100A4-induced activation of the transcription factor NF-κB