Contact hypersensitivity reaction to Oxazolone is independent whereas contact hypersensitivity reaction to DNCB is dependent on Il4 [Abstract]
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Bibliographic citation
-
In: Journal of Dermatological Science, 16, Supplement 1, S. S177-
- Classification
-
Medizin, Gesundheit
- Event
-
Veröffentlichung
- (where)
-
Augsburg
- (who)
-
Universität Augsburg
- (when)
-
1998
- Event
-
Veröffentlichung
- (where)
-
Amsterdam
- (who)
-
Elsevier
- (when)
-
1998
- Creator
- DOI
-
10.1016/S0923-1811(98)84056-5
- URN
-
urn:nbn:de:bvb:384-opus4-863628
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:24 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Traidl-Hoffmann, Claudia
- Jugert, Frank
- Merk, Hans
- Hunzelmann, Nicolas
- Universität Augsburg
- Elsevier
Time of origin
- 1998
Other Objects (12)
Cutaneous Metal Hypersensitivity Reaction
Contact hypersensitivity in experimental animals : 47 tab.
Is cancer a severe delayed hypersensitivity reaction and histamine a blueprint?
Immediate hypersensitivity reaction followed by successful oral desensitization to ursodiol
Contact hypersensitivity to nickel : experimental induction of allergy and tolerance, respectively
Contact hypersensitivity to nickel: experimental induction of allergy and tolerance, respectively
Tolerance to DNFB-induced contact hypersensitivity reactions is mediated by CD73+ effector memory regulatory T cells
Role of innate immune receptors in chemical-induced contact hypersensitivity : identification and modulation of novel inflammatory pathways in contact hypersensitivity
IL-31 is crucial for induction of pruritus, but not inflammation, in contact hypersensitivity
Hepatic NK cell education in monobenzone-induced contact hypersensitivity
IL-24 contributes to skin inflammation in Para-Phenylenediamine-induced contact hypersensitivity
Delayed hypersensitivity reaction to orodispersible budesonide in a case with eosinophilic esophagitis
Cutaneous Metal Hypersensitivity Reaction
Contact hypersensitivity in experimental animals : 47 tab.
Is cancer a severe delayed hypersensitivity reaction and histamine a blueprint?
Immediate hypersensitivity reaction followed by successful oral desensitization to ursodiol
Contact hypersensitivity to nickel : experimental induction of allergy and tolerance, respectively
Contact hypersensitivity to nickel: experimental induction of allergy and tolerance, respectively
Tolerance to DNFB-induced contact hypersensitivity reactions is mediated by CD73+ effector memory regulatory T cells
Role of innate immune receptors in chemical-induced contact hypersensitivity : identification and modulation of novel inflammatory pathways in contact hypersensitivity
IL-31 is crucial for induction of pruritus, but not inflammation, in contact hypersensitivity
Hepatic NK cell education in monobenzone-induced contact hypersensitivity
IL-24 contributes to skin inflammation in Para-Phenylenediamine-induced contact hypersensitivity
Delayed hypersensitivity reaction to orodispersible budesonide in a case with eosinophilic esophagitis
Cutaneous Metal Hypersensitivity Reaction
Contact hypersensitivity in experimental animals : 47 tab.
Is cancer a severe delayed hypersensitivity reaction and histamine a blueprint?
Immediate hypersensitivity reaction followed by successful oral desensitization to ursodiol
Contact hypersensitivity to nickel : experimental induction of allergy and tolerance, respectively
Contact hypersensitivity to nickel: experimental induction of allergy and tolerance, respectively
Tolerance to DNFB-induced contact hypersensitivity reactions is mediated by CD73+ effector memory regulatory T cells
Role of innate immune receptors in chemical-induced contact hypersensitivity : identification and modulation of novel inflammatory pathways in contact hypersensitivity
IL-31 is crucial for induction of pruritus, but not inflammation, in contact hypersensitivity
Hepatic NK cell education in monobenzone-induced contact hypersensitivity
IL-24 contributes to skin inflammation in Para-Phenylenediamine-induced contact hypersensitivity