- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1750-1326
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Glycosphingolipids and neuroinflammation in Parkinson’s disease ; volume:15 ; number:1 ; day:17 ; month:10 ; year:2020 ; pages:1-16 ; date:12.2020
Molecular neurodegeneration ; 15, Heft 1 (17.10.2020), 1-16, 12.2020
- Creator
-
Belarbi, Karim
Cuvelier, Elodie
Bonte, Marie-Amandine
Desplanque, Mazarine
Gressier, Bernard
Devos, David
Chartier-Harlin, Marie-Christine
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1186/s13024-020-00408-1
- URN
-
urn:nbn:de:101:1-2020120218484840413882
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:20 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Belarbi, Karim
- Cuvelier, Elodie
- Bonte, Marie-Amandine
- Desplanque, Mazarine
- Gressier, Bernard
- Devos, David
- Chartier-Harlin, Marie-Christine
- SpringerLink (Online service)
Other Objects (12)
Neuroinflammation and protein pathology in Parkinson’s disease dementia
LRRK2 and neuroinflammation: partners in crime in Parkinson’s disease?
Neuroinflammation in Parkinson’s disease – putative pathomechanisms and targets for disease-modification
Neurodegeneration and Neuroinflammation in Parkinson’s Disease: a Self-Sustained Loop
Modeling Neuroinflammation in sporadic Parkinson ́s disease
NLRP3 Inflammasome-Mediated Neuroinflammation and Related Mitochondrial Impairment in Parkinson’s Disease
The role of Toll-like receptors and neuroinflammation in Parkinson’s disease
CXCL12 is involved in α-synuclein-triggered neuroinflammation of Parkinson’s disease
Universal clinical Parkinson’s disease axes identify a major influence of neuroinflammation
Novel immunotherapeutic approaches to target alpha-synuclein and related neuroinflammation in Parkinson’s disease
NOD-like receptor NLRC5 promotes neuroinflammation and inhibits neuronal survival in Parkinson’s disease models
Defining the contribution of neuroinflammation to Parkinson’s disease in humanized immune system mice
Neuroinflammation and protein pathology in Parkinson’s disease dementia
LRRK2 and neuroinflammation: partners in crime in Parkinson’s disease?
Neuroinflammation in Parkinson’s disease – putative pathomechanisms and targets for disease-modification
Neurodegeneration and Neuroinflammation in Parkinson’s Disease: a Self-Sustained Loop
Modeling Neuroinflammation in sporadic Parkinson ́s disease
NLRP3 Inflammasome-Mediated Neuroinflammation and Related Mitochondrial Impairment in Parkinson’s Disease
The role of Toll-like receptors and neuroinflammation in Parkinson’s disease
CXCL12 is involved in α-synuclein-triggered neuroinflammation of Parkinson’s disease
Universal clinical Parkinson’s disease axes identify a major influence of neuroinflammation
Novel immunotherapeutic approaches to target alpha-synuclein and related neuroinflammation in Parkinson’s disease
NOD-like receptor NLRC5 promotes neuroinflammation and inhibits neuronal survival in Parkinson’s disease models
Defining the contribution of neuroinflammation to Parkinson’s disease in humanized immune system mice
Neuroinflammation and protein pathology in Parkinson’s disease dementia
LRRK2 and neuroinflammation: partners in crime in Parkinson’s disease?
Neuroinflammation in Parkinson’s disease – putative pathomechanisms and targets for disease-modification
Neurodegeneration and Neuroinflammation in Parkinson’s Disease: a Self-Sustained Loop
Modeling Neuroinflammation in sporadic Parkinson ́s disease
NLRP3 Inflammasome-Mediated Neuroinflammation and Related Mitochondrial Impairment in Parkinson’s Disease
The role of Toll-like receptors and neuroinflammation in Parkinson’s disease
CXCL12 is involved in α-synuclein-triggered neuroinflammation of Parkinson’s disease
Universal clinical Parkinson’s disease axes identify a major influence of neuroinflammation
Novel immunotherapeutic approaches to target alpha-synuclein and related neuroinflammation in Parkinson’s disease
NOD-like receptor NLRC5 promotes neuroinflammation and inhibits neuronal survival in Parkinson’s disease models