Author Correction: Optogenetic stimulation of vagal nerves for enhanced glucose-stimulated insulin secretion and β cell proliferation
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
-
Author Correction: Optogenetic stimulation of vagal nerves for enhanced glucose-stimulated insulin secretion and β cell proliferation ; day:2 ; month:4 ; year:2024 ; pages:1
Nature biomedical engineering ; (2.4.2024), 1
- Creator
-
Kawana, Yohei
Imai, Junta
Morizawa, Yosuke M.
Ikoma, Yoko
Kohata, Masato
Komamura, Hiroshi
Sato, Toshihiro
Izumi, Tomohito
Yamamoto, Junpei
Endo, Akira
Sugawara, Hiroto
Kubo, Haremaru
Hosaka, Shinichiro
Munakata, Yuichiro
Asai, Yoichiro
Kodama, Shinjiro
Takahashi, Kei
Kaneko, Keizo
Sawada, Shojiro
Yamada, Tetsuya
Ito, Akira
Niizuma, Kuniyasu
Tominaga, Teiji
Yamanaka, Akihiro
Matsui, Ko
Katagiri, Hideki
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1038/s41551-024-01200-y
- URN
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urn:nbn:de:101:1-2406162121325.705881025732
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
- 14.08.2025, 10:52 AM CEST
Data provider
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Associated
- Kawana, Yohei
- Imai, Junta
- Morizawa, Yosuke M.
- Ikoma, Yoko
- Kohata, Masato
- Komamura, Hiroshi
- Sato, Toshihiro
- Izumi, Tomohito
- Yamamoto, Junpei
- Endo, Akira
- Sugawara, Hiroto
- Kubo, Haremaru
- Hosaka, Shinichiro
- Munakata, Yuichiro
- Asai, Yoichiro
- Kodama, Shinjiro
- Takahashi, Kei
- Kaneko, Keizo
- Sawada, Shojiro
- Yamada, Tetsuya
- Ito, Akira
- Niizuma, Kuniyasu
- Tominaga, Teiji
- Yamanaka, Akihiro
- Matsui, Ko
- Katagiri, Hideki
- SpringerLink (Online service)
Other Objects (12)
Optogenetic stimulation of vagal nerves for enhanced glucose-stimulated insulin secretion and β cell proliferation
Follicle-stimulating hormone orchestrates glucose-stimulated insulin secretion of pancreatic islets
Cortistatin hyperpolarizes pancreatic beta cell membrane and reduces glucose-stimulated insulin secretion
20-HETE promotes glucose-stimulated insulin secretion in an autocrine manner through FFAR1
20-HETE promotes glucose-stimulated insulin secretion in an autocrine manner through FFAR1
Glucose-stimulated insulin secretion depends on FFA1 and Gq in neonatal mouse islets
A pathway model of glucose-stimulated insulin secretion in the pancreatic β-cell
Effects of Hypercalcemia, Hypercalcemia and Calcitonin on Glucose Stimulated Insulin Secretion in Man
Olfactory receptors are expressed in pancreatic β-cells and promote glucose-stimulated insulin secretion
Nutrient-stimulated insulin secretion in mouse islets is critically dependent on intracellular pH
High-dose clevudine impairs mitochondrial function and glucose-stimulated insulin secretion in INS-1E cells
In situ structure of actin remodeling during glucose-stimulated insulin secretion using cryo-electron tomography
Optogenetic stimulation of vagal nerves for enhanced glucose-stimulated insulin secretion and β cell proliferation
Follicle-stimulating hormone orchestrates glucose-stimulated insulin secretion of pancreatic islets
Cortistatin hyperpolarizes pancreatic beta cell membrane and reduces glucose-stimulated insulin secretion
20-HETE promotes glucose-stimulated insulin secretion in an autocrine manner through FFAR1
20-HETE promotes glucose-stimulated insulin secretion in an autocrine manner through FFAR1
Glucose-stimulated insulin secretion depends on FFA1 and Gq in neonatal mouse islets
A pathway model of glucose-stimulated insulin secretion in the pancreatic β-cell
Effects of Hypercalcemia, Hypercalcemia and Calcitonin on Glucose Stimulated Insulin Secretion in Man
Olfactory receptors are expressed in pancreatic β-cells and promote glucose-stimulated insulin secretion
Nutrient-stimulated insulin secretion in mouse islets is critically dependent on intracellular pH
High-dose clevudine impairs mitochondrial function and glucose-stimulated insulin secretion in INS-1E cells
In situ structure of actin remodeling during glucose-stimulated insulin secretion using cryo-electron tomography
Optogenetic stimulation of vagal nerves for enhanced glucose-stimulated insulin secretion and β cell proliferation
Follicle-stimulating hormone orchestrates glucose-stimulated insulin secretion of pancreatic islets
Cortistatin hyperpolarizes pancreatic beta cell membrane and reduces glucose-stimulated insulin secretion
20-HETE promotes glucose-stimulated insulin secretion in an autocrine manner through FFAR1
20-HETE promotes glucose-stimulated insulin secretion in an autocrine manner through FFAR1
Glucose-stimulated insulin secretion depends on FFA1 and Gq in neonatal mouse islets
A pathway model of glucose-stimulated insulin secretion in the pancreatic β-cell
Effects of Hypercalcemia, Hypercalcemia and Calcitonin on Glucose Stimulated Insulin Secretion in Man
Olfactory receptors are expressed in pancreatic β-cells and promote glucose-stimulated insulin secretion
Nutrient-stimulated insulin secretion in mouse islets is critically dependent on intracellular pH
High-dose clevudine impairs mitochondrial function and glucose-stimulated insulin secretion in INS-1E cells