mTOR contributes to endothelium-dependent vasorelaxation by promoting eNOS expression and preventing eNOS uncoupling
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2399-3642
- Extent
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Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
mTOR contributes to endothelium-dependent vasorelaxation by promoting eNOS expression and preventing eNOS uncoupling ; volume:5 ; number:1 ; day:22 ; month:7 ; year:2022 ; pages:1-14 ; date:12.2022
Communications biology ; 5, Heft 1 (22.7.2022), 1-14, 12.2022
- Creator
-
Wang, Yiying
Li, Qiannan
Zhang, Zhiyang
Peng, Kai
Zhang, Dai-Min
Yang, Qianlu
Passerini, Anthony G.
Simon, Scott I.
Sun, ChongXiu
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s42003-022-03653-w
- URN
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urn:nbn:de:101:1-2022100319511474030187
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
- 15.08.2025, 7:20 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Wang, Yiying
- Li, Qiannan
- Zhang, Zhiyang
- Peng, Kai
- Zhang, Dai-Min
- Yang, Qianlu
- Passerini, Anthony G.
- Simon, Scott I.
- Sun, ChongXiu
- SpringerLink (Online service)
Other Objects (12)
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Stachydrine protects eNOS uncoupling and ameliorates endothelial dysfunction induced by homocysteine
Endothelial dysfunction due to eNOS uncoupling: molecular mechanisms as potential therapeutic targets
Endothelial eNOS and red blood cell eNOS independently regulate blood pressure and nitric oxide metabolites
Erythrozytäre eNOS : NO-Synthese und Aktivität
Enos
Auswirkungen akuter körperlicher Belastung auf die erythrozytäre eNOS
Paternal eNOS deficiency in mice affects glucose homeostasis and liver glycogen in male offspring without inheritance of eNOS deficiency itself
Inflammatorische Leukozyten vermitteln Angiotensin II-induzierte eNOS Entkopplung und iNOS Aktivität
Patriarchenzyklus : Enos
Patriarchenzyklus : Enos
Frequency of eNOS polymorphisms in the Colombian general population
Molecular mechanisms underlying pharmacological stimulation of eNOS expression and eNOS activity
Stachydrine protects eNOS uncoupling and ameliorates endothelial dysfunction induced by homocysteine
Endothelial dysfunction due to eNOS uncoupling: molecular mechanisms as potential therapeutic targets
Endothelial eNOS and red blood cell eNOS independently regulate blood pressure and nitric oxide metabolites
Erythrozytäre eNOS : NO-Synthese und Aktivität
Enos
Auswirkungen akuter körperlicher Belastung auf die erythrozytäre eNOS
Paternal eNOS deficiency in mice affects glucose homeostasis and liver glycogen in male offspring without inheritance of eNOS deficiency itself
Inflammatorische Leukozyten vermitteln Angiotensin II-induzierte eNOS Entkopplung und iNOS Aktivität
Patriarchenzyklus : Enos
Patriarchenzyklus : Enos
Frequency of eNOS polymorphisms in the Colombian general population
Molecular mechanisms underlying pharmacological stimulation of eNOS expression and eNOS activity
Stachydrine protects eNOS uncoupling and ameliorates endothelial dysfunction induced by homocysteine
Endothelial dysfunction due to eNOS uncoupling: molecular mechanisms as potential therapeutic targets
Endothelial eNOS and red blood cell eNOS independently regulate blood pressure and nitric oxide metabolites
Erythrozytäre eNOS : NO-Synthese und Aktivität
Enos
Auswirkungen akuter körperlicher Belastung auf die erythrozytäre eNOS
Paternal eNOS deficiency in mice affects glucose homeostasis and liver glycogen in male offspring without inheritance of eNOS deficiency itself
Inflammatorische Leukozyten vermitteln Angiotensin II-induzierte eNOS Entkopplung und iNOS Aktivität
Patriarchenzyklus : Enos
Patriarchenzyklus : Enos