Oxidative toxicity in diabetes and Alzheimer’s disease: mechanisms behind ROS/ RNS generation
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1423-0127
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Oxidative toxicity in diabetes and Alzheimer’s disease: mechanisms behind ROS/ RNS generation ; volume:24 ; number:1 ; day:19 ; month:9 ; year:2017 ; pages:1-10 ; date:12.2017
Journal of biomedical science ; 24, Heft 1 (19.9.2017), 1-10, 12.2017
- Keyword
-
Endokrinologie
- Creator
-
Ahmad, Waqar
- Contributor
-
Ijaz, Bushra
Shabbiri, Khadija
Ahmed, Fayyaz
Rehman, Sidra
SpringerLink (Online service)
- DOI
-
10.1186/s12929-017-0379-z
- URN
-
urn:nbn:de:1111-2017111816115
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:34 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Ahmad, Waqar
- Ijaz, Bushra
- Shabbiri, Khadija
- Ahmed, Fayyaz
- Rehman, Sidra
- SpringerLink (Online service)
Other Objects (12)
Enhanced ROS-generation in lymphocytes from Alzheimer’s patients
Transient dynamics of Aβ contribute to toxicity in Alzheimer’s disease
Aβ42 and ROS dual-targeted multifunctional nanocomposite for combination therapy of Alzheimer’s disease
Composite mathematical modeling of calcium signaling behind neuronal cell death in Alzheimer’s disease
Otophylloside B Protects Against Aβ Toxicity in Caenorhabditis elegans Models of Alzheimer’s Disease
Amyloid accelerates tau propagation and toxicity in a model of early Alzheimer’s disease
Alzheimer’s Disease
Galectin-3 promotes Aβ oligomerization and Aβ toxicity in a mouse model of Alzheimer’s disease
Alzheimer
Alzheimer
Generalvollmacht: Eduard Alzheimer für Karl Alzheimer
Treatments in Alzheimer’s disease
Enhanced ROS-generation in lymphocytes from Alzheimer’s patients
Transient dynamics of Aβ contribute to toxicity in Alzheimer’s disease
Aβ42 and ROS dual-targeted multifunctional nanocomposite for combination therapy of Alzheimer’s disease
Composite mathematical modeling of calcium signaling behind neuronal cell death in Alzheimer’s disease
Otophylloside B Protects Against Aβ Toxicity in Caenorhabditis elegans Models of Alzheimer’s Disease
Amyloid accelerates tau propagation and toxicity in a model of early Alzheimer’s disease
Alzheimer’s Disease
Galectin-3 promotes Aβ oligomerization and Aβ toxicity in a mouse model of Alzheimer’s disease
Alzheimer
Alzheimer
Generalvollmacht: Eduard Alzheimer für Karl Alzheimer
Treatments in Alzheimer’s disease
Enhanced ROS-generation in lymphocytes from Alzheimer’s patients
Transient dynamics of Aβ contribute to toxicity in Alzheimer’s disease
Aβ42 and ROS dual-targeted multifunctional nanocomposite for combination therapy of Alzheimer’s disease
Composite mathematical modeling of calcium signaling behind neuronal cell death in Alzheimer’s disease
Otophylloside B Protects Against Aβ Toxicity in Caenorhabditis elegans Models of Alzheimer’s Disease
Amyloid accelerates tau propagation and toxicity in a model of early Alzheimer’s disease
Alzheimer’s Disease
Galectin-3 promotes Aβ oligomerization and Aβ toxicity in a mouse model of Alzheimer’s disease
Alzheimer
Alzheimer
Generalvollmacht: Eduard Alzheimer für Karl Alzheimer