Neuron–astrocyte metabolic coupling facilitates spinal plasticity and maintenance of inflammatory pain

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: Neuron–astrocyte metabolic coupling facilitates spinal plasticity and maintenance of inflammatory pain ; volume:6 ; number:3 ; day:5 ; month:3 ; year:2024 ; pages:494-513 ; date:3.2024
Nature metabolism ; 6, Heft 3 (5.3.2024), 494-513, 3.2024

Creator: Marty-Lombardi, Sebastián
Lu, Shiying
Ambroziak, Wojciech
Schrenk-Siemens, Katrin
Wang, Jialin
DePaoli-Roach, Anna A.
Hertle, Anna M.
Wende, Hagen
Tappe-Theodor, Anke
Simonetti, Manuela
Bading, Hilmar
Okun, Jürgen G.
Kuner, Rohini
Fleming, Thomas
Siemens, Jan

Contributor: SpringerLink (Online service)

DOI: 10.1038/s42255-024-01001-2

URN: urn:nbn:de:101:1-2405232118489.914627490758

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:44 AM CEST

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Neuron–astrocyte metabolic coupling facilitates spinal plasticity and maintenance of inflammatory pain

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Other Objects (12)

PKMζ is essential for spinal plasticity underlying the maintenance of persistent pain

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Weak gene–gene interaction facilitates the evolution of gene expression plasticity

Translational switch for long term maintenance of synaptic plasticity

Sec61β facilitates the maintenance of endoplasmic reticulum homeostasis by associating microtubules

Maladaptive plasticity facilitates evolution of thermal tolerance during an experimental range shift

PKMζ is essential for spinal plasticity underlying the maintenance of persistent pain

RGS5 augments astrocyte activation and facilitates neuroinflammation via TNF signaling

Induction and maintenance of synaptic plasticity

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Viral plasticity facilitates host diversity in challenging environments

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Notch Signaling Mediates Astrocyte Abnormality in Spinal Muscular Atrophy Model Systems

Injury-specific factors in the cerebrospinal fluid regulate astrocyte plasticity in the human brain

Weak gene–gene interaction facilitates the evolution of gene expression plasticity

Translational switch for long term maintenance of synaptic plasticity

Sec61β facilitates the maintenance of endoplasmic reticulum homeostasis by associating microtubules

Maladaptive plasticity facilitates evolution of thermal tolerance during an experimental range shift

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Weak gene–gene interaction facilitates the evolution of gene expression plasticity

Translational switch for long term maintenance of synaptic plasticity

Sec61β facilitates the maintenance of endoplasmic reticulum homeostasis by associating microtubules

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