Hochschulschrift
Novel chromatin interactions by structural rearrangements and aberrant enhancer functions drive oncogenic programs in unfavorable neuroblastoma
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Maße
-
30 cm
- Umfang
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XVI, 152 Blätter
- Sprache
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Englisch
- Anmerkungen
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Illustrationen, Diagramme
Ruperto Carola University Heidelberg, Dissertation, 2018
- Klassifikation
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Medizin, Gesundheit
Biowissenschaften, Biologie
- Inhaltsverzeichnis
- Rechteinformation
-
Bei diesem Objekt liegt nur das Inhaltsverzeichnis digital vor. Der Zugriff darauf ist unbeschränkt möglich.
- Letzte Aktualisierung
-
11.06.2025, 13:41 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
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Objekttyp
- Hochschulschrift
Beteiligte
Entstanden
- [2018]
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An oncogenic enhancer promotes melanoma progression via regulating ETV4 expression
Shared enhancer gene regulatory networks between wound and oncogenic programs
Arginine deiminase and other antiangiogenic agents inhibit unfavorable neuroblastoma growth: potentiation by irradiation
Absence of polysialylated NCAM is an unfavorable prognostic phenotype for advanced stage neuroblastoma
Recurrent DNMT3B rearrangements are associated with unfavorable outcome in dicentric (9;20)-positive pediatric BCP-ALL
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A local tumor microenvironment acquired super-enhancer induces an oncogenic driver in colorectal carcinoma
Enhancer engagement sustains oncogenic transformation and progression of B-cell precursor acute lymphoblastic leukemia
Isoform dependent oncogenic or tumor suppressive properties of the transcription factor MEIS2 in neuroblastoma
Novel chromatin interactions by structural rearrangements and aberrant enhancer functions drive oncogenic programs in unfavorable neuroblastoma
Insights into the formation of oncogenic chromosome rearrangements
Enhancer hijacking determines extrachromosomal circular MYCN amplicon architecture in neuroblastoma
An oncogenic enhancer promotes melanoma progression via regulating ETV4 expression
Shared enhancer gene regulatory networks between wound and oncogenic programs
Arginine deiminase and other antiangiogenic agents inhibit unfavorable neuroblastoma growth: potentiation by irradiation
Absence of polysialylated NCAM is an unfavorable prognostic phenotype for advanced stage neuroblastoma
Recurrent DNMT3B rearrangements are associated with unfavorable outcome in dicentric (9;20)-positive pediatric BCP-ALL
Age dependence of tumor genetics in unfavorable neuroblastoma: arrayCGH profiles of 34 consecutive cases, using a Swedish 25-year neuroblastoma cohort for validation
A local tumor microenvironment acquired super-enhancer induces an oncogenic driver in colorectal carcinoma
Enhancer engagement sustains oncogenic transformation and progression of B-cell precursor acute lymphoblastic leukemia