DYRK1A antagonists rescue degeneration and behavioural deficits of in vivo models based on amyloid-β, Tau and DYRK1A neurotoxicity
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2045-2322
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
DYRK1A antagonists rescue degeneration and behavioural deficits of in vivo models based on amyloid-β, Tau and DYRK1A neurotoxicity ; volume:12 ; number:1 ; day:23 ; month:9 ; year:2022 ; pages:1-14 ; date:12.2022
Scientific reports ; 12, Heft 1 (23.9.2022), 1-14, 12.2022
- Urheber
-
Zhu, Bangfu
Parsons, Tom
Foley, Christopher
Shaw, Yeng
Dunckley, Travis
Hulme, Christopher
Hodge, James J. L.
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1038/s41598-022-19967-y
- URN
-
urn:nbn:de:101:1-2022121010002658237844
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
- 15.08.2025, 07:25 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Zhu, Bangfu
- Parsons, Tom
- Foley, Christopher
- Shaw, Yeng
- Dunckley, Travis
- Hulme, Christopher
- Hodge, James J. L.
- SpringerLink (Online service)
Ähnliche Objekte (12)
Effects of anle138b treatment on amyloid-β neurotoxicity
Attenuated amyloid-β aggregation and neurotoxicity owing to methionine oxidation
Iron overload contributes to general anaesthesia-induced neurotoxicity and cognitive deficits
Early and Late Pathomechanisms in Alzheimer’s Disease: From Zinc to Amyloid-β Neurotoxicity
FcgRIIb mediates amyloid-β neurotoxicity and memory impairment in a model of Alzheimer’s disease
Amyloid-β slows cilia movement along the ventricle, impairs fluid flow, and exacerbates its neurotoxicity in explant culture
Chinese herbal extract reduces Amyloid-β induced neurotoxicity through inhibiting NF-κB signaling pathway in neuronal cells
Selective neurotoxicity
Early cerebral amyloid-β accumulation and hypermetabolism are associated with subtle cognitive deficits before accelerated cerebral atrophy
Functional network structure supports resilience to memory deficits in cognitively normal older adults with amyloid-β pathology
Neurotoxicity of NMDA antagonists: a glutamatergic theory of schizophrenia based on selective impairment of local inhibitory feedback circuits
NMDA receptor antagonists reduce amyloid-β deposition by modulating calpain-1 signaling and autophagy, rescuing cognitive impairment in 5XFAD mice
Effects of anle138b treatment on amyloid-β neurotoxicity
Attenuated amyloid-β aggregation and neurotoxicity owing to methionine oxidation
Iron overload contributes to general anaesthesia-induced neurotoxicity and cognitive deficits
Early and Late Pathomechanisms in Alzheimer’s Disease: From Zinc to Amyloid-β Neurotoxicity
FcgRIIb mediates amyloid-β neurotoxicity and memory impairment in a model of Alzheimer’s disease
Amyloid-β slows cilia movement along the ventricle, impairs fluid flow, and exacerbates its neurotoxicity in explant culture
Chinese herbal extract reduces Amyloid-β induced neurotoxicity through inhibiting NF-κB signaling pathway in neuronal cells
Selective neurotoxicity
Early cerebral amyloid-β accumulation and hypermetabolism are associated with subtle cognitive deficits before accelerated cerebral atrophy
Functional network structure supports resilience to memory deficits in cognitively normal older adults with amyloid-β pathology
Neurotoxicity of NMDA antagonists: a glutamatergic theory of schizophrenia based on selective impairment of local inhibitory feedback circuits
NMDA receptor antagonists reduce amyloid-β deposition by modulating calpain-1 signaling and autophagy, rescuing cognitive impairment in 5XFAD mice
Effects of anle138b treatment on amyloid-β neurotoxicity
Attenuated amyloid-β aggregation and neurotoxicity owing to methionine oxidation
Iron overload contributes to general anaesthesia-induced neurotoxicity and cognitive deficits
Early and Late Pathomechanisms in Alzheimer’s Disease: From Zinc to Amyloid-β Neurotoxicity
FcgRIIb mediates amyloid-β neurotoxicity and memory impairment in a model of Alzheimer’s disease
Amyloid-β slows cilia movement along the ventricle, impairs fluid flow, and exacerbates its neurotoxicity in explant culture
Chinese herbal extract reduces Amyloid-β induced neurotoxicity through inhibiting NF-κB signaling pathway in neuronal cells
Selective neurotoxicity
Early cerebral amyloid-β accumulation and hypermetabolism are associated with subtle cognitive deficits before accelerated cerebral atrophy
Functional network structure supports resilience to memory deficits in cognitively normal older adults with amyloid-β pathology
Neurotoxicity of NMDA antagonists: a glutamatergic theory of schizophrenia based on selective impairment of local inhibitory feedback circuits