Glucocorticoid receptor in astrocytes regulates midbrain dopamine neurodegeneration through connexin hemichannel activity

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1476-5403

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Glucocorticoid receptor in astrocytes regulates midbrain dopamine neurodegeneration through connexin hemichannel activity ; day:13 ; month:7 ; year:2018 ; pages:1-17
Cell death and differentiation ; (13.7.2018), 1-17

Creator: Maatouk, Layal

Contributor: Yi, Chenju
Carrillo-de Sauvage, Maria-Angeles
Compagnion, Anne-Claire
Hunot, Stéphane
Ezan, Pascal
Hirsch, Etienne C.
Koulakoff, Annette
Pfrieger, Frank W.
Tronche, François
Leybaert, Luc
Giaume, Christian
Vyas, Sheela
SpringerLink (Online service)

DOI: 10.1038/s41418-018-0150-3

URN: urn:nbn:de:101:1-2018091723401622857020

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 2025-08-15T07:25:17+0200

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Associated

Maatouk, Layal
Yi, Chenju
Carrillo-de Sauvage, Maria-Angeles
Compagnion, Anne-Claire
Hunot, Stéphane
Ezan, Pascal
Hirsch, Etienne C.
Koulakoff, Annette
Pfrieger, Frank W.
Tronche, François
Leybaert, Luc
Giaume, Christian
Vyas, Sheela
SpringerLink (Online service)

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Glucocorticoid receptor in astrocytes regulates midbrain dopamine neurodegeneration through connexin hemichannel activity

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Other Objects (12)

Astrocytes contribute to toll-like receptor 2-mediated neurodegeneration and alpha-synuclein pathology in a human midbrain Parkinson’s model

TLR9 activation via microglial glucocorticoid receptors contributes to degeneration of midbrain dopamine neurons

Glucocorticoid receptor signaling in astrocytes is required for aversive memory formation

Ventral midbrain astrocytes display unique physiological features and sensitivity to dopamine D2 receptor signaling

Transient IKK2 activation in astrocytes initiates selective non-cell-autonomous neurodegeneration

Author Correction: TLR9 activation via microglial glucocorticoid receptors contributes to degeneration of midbrain dopamine neurons

Expression von Connexin 37, Connexin 40 und Connexin 43 unter laminarem und turbulentem Shear-Stress

Midbrain cysticercal cyst

The overexpression of TDP-43 in astrocytes causes neurodegeneration via a PTP1B-mediated inflammatory response

Deficiency of Perry syndrome-associated p150Glued in midbrain dopaminergic neurons leads to progressive neurodegeneration and endoplasmic reticulum abnormalities

Pineal and midbrain lesions

Temperatursensitivität von Connexin 26

Astrocytes contribute to toll-like receptor 2-mediated neurodegeneration and alpha-synuclein pathology in a human midbrain Parkinson’s model

TLR9 activation via microglial glucocorticoid receptors contributes to degeneration of midbrain dopamine neurons

Glucocorticoid receptor signaling in astrocytes is required for aversive memory formation

Ventral midbrain astrocytes display unique physiological features and sensitivity to dopamine D2 receptor signaling

Transient IKK2 activation in astrocytes initiates selective non-cell-autonomous neurodegeneration

Author Correction: TLR9 activation via microglial glucocorticoid receptors contributes to degeneration of midbrain dopamine neurons

Expression von Connexin 37, Connexin 40 und Connexin 43 unter laminarem und turbulentem Shear-Stress

Midbrain cysticercal cyst

The overexpression of TDP-43 in astrocytes causes neurodegeneration via a PTP1B-mediated inflammatory response

Deficiency of Perry syndrome-associated p150Glued in midbrain dopaminergic neurons leads to progressive neurodegeneration and endoplasmic reticulum abnormalities

Pineal and midbrain lesions

Temperatursensitivität von Connexin 26

Astrocytes contribute to toll-like receptor 2-mediated neurodegeneration and alpha-synuclein pathology in a human midbrain Parkinson’s model

TLR9 activation via microglial glucocorticoid receptors contributes to degeneration of midbrain dopamine neurons

Glucocorticoid receptor signaling in astrocytes is required for aversive memory formation

Ventral midbrain astrocytes display unique physiological features and sensitivity to dopamine D2 receptor signaling

Transient IKK2 activation in astrocytes initiates selective non-cell-autonomous neurodegeneration

Author Correction: TLR9 activation via microglial glucocorticoid receptors contributes to degeneration of midbrain dopamine neurons

Expression von Connexin 37, Connexin 40 und Connexin 43 unter laminarem und turbulentem Shear-Stress

Midbrain cysticercal cyst

The overexpression of TDP-43 in astrocytes causes neurodegeneration via a PTP1B-mediated inflammatory response

Deficiency of Perry syndrome-associated p150Glued in midbrain dopaminergic neurons leads to progressive neurodegeneration and endoplasmic reticulum abnormalities

Pineal and midbrain lesions

Temperatursensitivität von Connexin 26

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