Host restriction factor SAMHD1 limits human T-cell leukemia virus (HTLV-1) infection of primary monocytes via the innate immune sensor STING
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1742-4690
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Host restriction factor SAMHD1 limits human T-cell leukemia virus (HTLV-1) infection of primary monocytes via the innate immune sensor STING ; volume:11 ; number:1 ; day:7 ; month:1 ; year:2014 ; pages:1 ; date:1.2014
Retrovirology ; 11, Heft 1 (7.1.2014), 1, 1.2014
- Creator
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Sze, Alexandre
- Contributor
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Belgnaoui, S Mehdi
Lin, Rongtuan
van Grevenynghe, Julien
Hiscott, John
SpringerLink (Online service)
- DOI
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10.1186/1742-4690-11-S1-O19
- URN
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urn:nbn:de:1111-2016080734828
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
- 14.08.2025, 10:55 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Sze, Alexandre
- Belgnaoui, S Mehdi
- Lin, Rongtuan
- van Grevenynghe, Julien
- Hiscott, John
- SpringerLink (Online service)
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Transcription-independent regulation of STING activation and innate immune responses by IRF8 in monocytes
HTLV-1 burden dependent on hijacking monocytes chemotaxis
O-GlcNAc of STING mediates antiviral innate immunity
The contribution of monocytes/macrophages to HTLV-1 infection and persistence
Anti-inflammatory monocytes—interplay of innate and adaptive immunity
Retroviral restriction factors: new mechanisms of innate immunity
HTLV-1 HBZ protein inhibits IRF3-mediated innate immune responses
Role of HTLV-1 p30 during infection of monocytes and dendritic cells
STING exerts antiviral innate immune response by activating pentose phosphate pathway
The roles of innate and acquired immune responses on HTLV-I infection
TLR9 and STING agonists synergistically induce innate and adaptive type‐II IFN
STING Suppresses Mitochondrial VDAC2 to Govern RCC Growth Independent of Innate Immunity
Transcription-independent regulation of STING activation and innate immune responses by IRF8 in monocytes
HTLV-1 burden dependent on hijacking monocytes chemotaxis
O-GlcNAc of STING mediates antiviral innate immunity
The contribution of monocytes/macrophages to HTLV-1 infection and persistence
Anti-inflammatory monocytes—interplay of innate and adaptive immunity
Retroviral restriction factors: new mechanisms of innate immunity
HTLV-1 HBZ protein inhibits IRF3-mediated innate immune responses
Role of HTLV-1 p30 during infection of monocytes and dendritic cells
STING exerts antiviral innate immune response by activating pentose phosphate pathway
The roles of innate and acquired immune responses on HTLV-I infection
TLR9 and STING agonists synergistically induce innate and adaptive type‐II IFN