Phenotyping of cancer-associated somatic mutations in the BCL2 transmembrane domain
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Phenotyping of cancer-associated somatic mutations in the BCL2 transmembrane domain ; volume:13 ; number:1 ; day:26 ; month:4 ; year:2024 ; pages:1-10 ; date:12.2024
Oncogenesis ; 13, Heft 1 (26.4.2024), 1-10, 12.2024
- Creator
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Leiva, Diego
Lucendo, Estefanía
García-Jareño, Alicia Belén
Sancho, Mónica
Orzáez, Mar
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41389-024-00516-3
- URN
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urn:nbn:de:101:1-2407092121381.861255819970
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:53 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Leiva, Diego
- Lucendo, Estefanía
- García-Jareño, Alicia Belén
- Sancho, Mónica
- Orzáez, Mar
- SpringerLink (Online service)
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Targeted inhibition of DHODH is synergistic with BCL2 blockade in HGBCL with concurrent MYC and BCL2 rearrangement
A BCL2 promoter polymorphism rs2279115 is not associated with BCL2 protein expression or patient survival in breast cancer patients
GNA13 regulates BCL2 expression and the sensitivity of GCB-DLBCL cells to BCL2 inhibitors in a palmitoylation-dependent manner
Hydrophobic pulses predict transmembrane helix irregularities and channel transmembrane units
Computational study on novel natural inhibitors targeting BCL2
Viral Bcl2s’ transmembrane domain interact with host Bcl2 proteins to control cellular apoptosis
Feature selection algorithm based on dual correlation filters for cancer-associated somatic variants
Somatic copy number gains in MYC, BCL2, and BCL6 identifies a subset of aggressive alternative-DH/TH DLBCL patients
Quantitative analysis of tumor-specific BCL2 expression in DLBCL: refinement of prognostic relevance of BCL2
Bcl2 inhibition of mitochondrial DNA repair
CD23-positive, BCL2-Rearrangement-negative Keimzentrumslymphome
BCL2 genotypes and prostate cancer survival
Targeted inhibition of DHODH is synergistic with BCL2 blockade in HGBCL with concurrent MYC and BCL2 rearrangement
A BCL2 promoter polymorphism rs2279115 is not associated with BCL2 protein expression or patient survival in breast cancer patients
GNA13 regulates BCL2 expression and the sensitivity of GCB-DLBCL cells to BCL2 inhibitors in a palmitoylation-dependent manner
Hydrophobic pulses predict transmembrane helix irregularities and channel transmembrane units
Computational study on novel natural inhibitors targeting BCL2
Viral Bcl2s’ transmembrane domain interact with host Bcl2 proteins to control cellular apoptosis
Feature selection algorithm based on dual correlation filters for cancer-associated somatic variants
Somatic copy number gains in MYC, BCL2, and BCL6 identifies a subset of aggressive alternative-DH/TH DLBCL patients
Quantitative analysis of tumor-specific BCL2 expression in DLBCL: refinement of prognostic relevance of BCL2
Bcl2 inhibition of mitochondrial DNA repair
CD23-positive, BCL2-Rearrangement-negative Keimzentrumslymphome
BCL2 genotypes and prostate cancer survival
Targeted inhibition of DHODH is synergistic with BCL2 blockade in HGBCL with concurrent MYC and BCL2 rearrangement
A BCL2 promoter polymorphism rs2279115 is not associated with BCL2 protein expression or patient survival in breast cancer patients
GNA13 regulates BCL2 expression and the sensitivity of GCB-DLBCL cells to BCL2 inhibitors in a palmitoylation-dependent manner
Hydrophobic pulses predict transmembrane helix irregularities and channel transmembrane units