The non-canonical inflammasome activators Caspase-4 and Caspase-5 are differentially regulated during immunosuppression-associated organ damage

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource, 13 Seiten

Language: Englisch

Bibliographic citation: In: Frontiers in Immunology(14) - ISSN 1664-3224
In: Copyright © 2023 Ghait, Duduskar, Rooney, Häfner, Reng, Göhrig, Reuken, Bloos, Bauer, Sponholz, Bruns and Rubio, Lausanne

Event: Veröffentlichung

(where): Jena

(who): Friedrich-Schiller-Universität Jena

(when): 2023

Creator: Ghait, Mohamed
Duduskar, Shivalee N.
Rooney, Michael
Häfner, Norman
Reng, Laura
Göhrig, Bianca
Reuken, Philipp Alexander
Bloos, Frank
Bauer, Michael
Sponholz, Christoph
Bruns, Tony
Rubio, Ignacio

URN: urn:nbn:de:gbv:27-dbt-20240418-140336-005

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:59 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Ghait, Mohamed
Duduskar, Shivalee N.
Rooney, Michael
Häfner, Norman
Reng, Laura
Göhrig, Bianca
Reuken, Philipp Alexander
Bloos, Frank
Bauer, Michael
Sponholz, Christoph
Bruns, Tony
Rubio, Ignacio
Friedrich-Schiller-Universität Jena

Time of origin

2023

Other Objects (12)

The non-canonical inflammasome activators Caspase-4 and Caspase-5 are differentially regulated during immunosuppression-associated organ damage

Cytoplasmic innate immune sensing by the caspase-4 non-canonical inflammasome promotes cellular senescence

Activation of the NLRP1B inflammasome by caspase-8

Caspase-9, caspase-3 and caspase-7 have distinct roles during intrinsic apoptosis

Dimer‐specific immunoprecipitation of active caspase‐2 identifies TRAF proteins as novel activators

Caspase-3 and caspase-8 expression in breast cancer: caspase-3 is associated with survival

Role of caspase-11 non-canonical inflammasomes in retinal ischemia/reperfusion injury

S1PR1 regulates NDV-induced IL-1β expression via NLRP3/caspase-1 inflammasome

Structure optimization of new tumor-selective Passerini α-acyloxy carboxamides as Caspase-3/7 activators

Prdx4 limits caspase‐1 activation and restricts inflammasome‐mediated signaling by extracellular vesicles

AIP1 Regulates Ocular Angiogenesis Via NLRP12‐ASC‐Caspase‐8 Inflammasome‐Mediated Endothelial Pyroptosis

Caspase-independent cell death

The non-canonical inflammasome activators Caspase-4 and Caspase-5 are differentially regulated during immunosuppression-associated organ damage

Cytoplasmic innate immune sensing by the caspase-4 non-canonical inflammasome promotes cellular senescence

Activation of the NLRP1B inflammasome by caspase-8

Caspase-9, caspase-3 and caspase-7 have distinct roles during intrinsic apoptosis

Dimer‐specific immunoprecipitation of active caspase‐2 identifies TRAF proteins as novel activators

Caspase-3 and caspase-8 expression in breast cancer: caspase-3 is associated with survival

Role of caspase-11 non-canonical inflammasomes in retinal ischemia/reperfusion injury

S1PR1 regulates NDV-induced IL-1β expression via NLRP3/caspase-1 inflammasome

Structure optimization of new tumor-selective Passerini α-acyloxy carboxamides as Caspase-3/7 activators

Prdx4 limits caspase‐1 activation and restricts inflammasome‐mediated signaling by extracellular vesicles

AIP1 Regulates Ocular Angiogenesis Via NLRP12‐ASC‐Caspase‐8 Inflammasome‐Mediated Endothelial Pyroptosis

Caspase-independent cell death

The non-canonical inflammasome activators Caspase-4 and Caspase-5 are differentially regulated during immunosuppression-associated organ damage

Cytoplasmic innate immune sensing by the caspase-4 non-canonical inflammasome promotes cellular senescence

Activation of the NLRP1B inflammasome by caspase-8

Caspase-9, caspase-3 and caspase-7 have distinct roles during intrinsic apoptosis

Dimer‐specific immunoprecipitation of active caspase‐2 identifies TRAF proteins as novel activators

Caspase-3 and caspase-8 expression in breast cancer: caspase-3 is associated with survival

Role of caspase-11 non-canonical inflammasomes in retinal ischemia/reperfusion injury

S1PR1 regulates NDV-induced IL-1β expression via NLRP3/caspase-1 inflammasome

Structure optimization of new tumor-selective Passerini α-acyloxy carboxamides as Caspase-3/7 activators

Prdx4 limits caspase‐1 activation and restricts inflammasome‐mediated signaling by extracellular vesicles

AIP1 Regulates Ocular Angiogenesis Via NLRP12‐ASC‐Caspase‐8 Inflammasome‐Mediated Endothelial Pyroptosis

Caspase-independent cell death

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

The non-canonical inflammasome activators Caspase-4 and Caspase-5 are differentially regulated during immunosuppression-associated organ damage

Object Details

References and Relationships

Contributors, Places and Time

Further information

Data provider

Associated

Time of origin

Other Objects (12)

The non-canonical inflammasome activators Caspase-4 and Caspase-5 are differentially regulated during immunosuppression-associated organ damage

Cytoplasmic innate immune sensing by the caspase-4 non-canonical inflammasome promotes cellular senescence

Activation of the NLRP1B inflammasome by caspase-8

Caspase-9, caspase-3 and caspase-7 have distinct roles during intrinsic apoptosis

Dimer‐specific immunoprecipitation of active caspase‐2 identifies TRAF proteins as novel activators

Caspase-3 and caspase-8 expression in breast cancer: caspase-3 is associated with survival

Role of caspase-11 non-canonical inflammasomes in retinal ischemia/reperfusion injury

S1PR1 regulates NDV-induced IL-1β expression via NLRP3/caspase-1 inflammasome

Structure optimization of new tumor-selective Passerini α-acyloxy carboxamides as Caspase-3/7 activators

Prdx4 limits caspase‐1 activation and restricts inflammasome‐mediated signaling by extracellular vesicles

AIP1 Regulates Ocular Angiogenesis Via NLRP12‐ASC‐Caspase‐8 Inflammasome‐Mediated Endothelial Pyroptosis

Caspase-independent cell death

The non-canonical inflammasome activators Caspase-4 and Caspase-5 are differentially regulated during immunosuppression-associated organ damage

Cytoplasmic innate immune sensing by the caspase-4 non-canonical inflammasome promotes cellular senescence

Activation of the NLRP1B inflammasome by caspase-8

Caspase-9, caspase-3 and caspase-7 have distinct roles during intrinsic apoptosis

Dimer‐specific immunoprecipitation of active caspase‐2 identifies TRAF proteins as novel activators

Caspase-3 and caspase-8 expression in breast cancer: caspase-3 is associated with survival

Role of caspase-11 non-canonical inflammasomes in retinal ischemia/reperfusion injury

S1PR1 regulates NDV-induced IL-1β expression via NLRP3/caspase-1 inflammasome

Structure optimization of new tumor-selective Passerini α-acyloxy carboxamides as Caspase-3/7 activators

Prdx4 limits caspase‐1 activation and restricts inflammasome‐mediated signaling by extracellular vesicles

AIP1 Regulates Ocular Angiogenesis Via NLRP12‐ASC‐Caspase‐8 Inflammasome‐Mediated Endothelial Pyroptosis

Caspase-independent cell death

The non-canonical inflammasome activators Caspase-4 and Caspase-5 are differentially regulated during immunosuppression-associated organ damage

Cytoplasmic innate immune sensing by the caspase-4 non-canonical inflammasome promotes cellular senescence

Activation of the NLRP1B inflammasome by caspase-8

Caspase-9, caspase-3 and caspase-7 have distinct roles during intrinsic apoptosis

Dimer‐specific immunoprecipitation of active caspase‐2 identifies TRAF proteins as novel activators

Caspase-3 and caspase-8 expression in breast cancer: caspase-3 is associated with survival

Role of caspase-11 non-canonical inflammasomes in retinal ischemia/reperfusion injury

S1PR1 regulates NDV-induced IL-1β expression via NLRP3/caspase-1 inflammasome

Structure optimization of new tumor-selective Passerini α-acyloxy carboxamides as Caspase-3/7 activators

Prdx4 limits caspase‐1 activation and restricts inflammasome‐mediated signaling by extracellular vesicles

AIP1 Regulates Ocular Angiogenesis Via NLRP12‐ASC‐Caspase‐8 Inflammasome‐Mediated Endothelial Pyroptosis

Caspase-independent cell death

Related objects

Reset password