Overexpression of V-ATPase B2 attenuates lung injury/fibrosis by stabilizing lysosomal membrane permeabilization and increasing collagen degradation

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2092-6413

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Overexpression of V-ATPase B2 attenuates lung injury/fibrosis by stabilizing lysosomal membrane permeabilization and increasing collagen degradation ; volume:54 ; number:5 ; day:27 ; month:5 ; year:2022 ; pages:662-672 ; date:5.2022
Experimental & molecular medicine ; 54, Heft 5 (27.5.2022), 662-672, 5.2022

Creator: Lee, Jong-Uk
Hong, Jisu
Shin, Hyesun
Ryu, Chnag-Beom
Park, Sung-Woo
Jeong, Sung Hwan

Contributor: SpringerLink (Online service)

DOI: 10.1038/s12276-022-00776-2

URN: urn:nbn:de:101:1-2022080221340289377129

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:36 AM CEST

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Lee, Jong-Uk
Hong, Jisu
Shin, Hyesun
Ryu, Chnag-Beom
Park, Sung-Woo
Jeong, Sung Hwan
SpringerLink (Online service)

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Hochschulschrift

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Direct control of lysosomal catabolic activity by mTORC1 through regulation of V-ATPase assembly

The V-ATPase complex component RNAseK is required for lysosomal hydrolase delivery and autophagosome degradation

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Leucine/glutamine and v-ATPase/lysosomal acidification via mTORC1 activation are required for position-dependent regeneration

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Hochschulschrift

mTORC1 regulates lysosomal catabolic activity by preventing V-ATPase assembly

Defective Lamtor5 Leads to Autoimmunity by Deregulating v‐ATPase and Lysosomal Acidification

NDST3 deacetylates α‐tubulin and suppresses V‐ATPase assembly and lysosomal acidification

The emerging roles of vacuolar-type ATPase-dependent Lysosomal acidification in neurodegenerative diseases

STAT3 associates with vacuolar H+-ATPase and regulates cytosolic and lysosomal pH

Antipsychotics possess anti-glioblastoma activity by disrupting lysosomal function and inhibiting oncogenic signaling by stabilizing PTEN

Beta-estradiol attenuates hypoxic pulmonary hypertension by stabilizing the expression of p27kip1in rats

Direct control of lysosomal catabolic activity by mTORC1 through regulation of V-ATPase assembly

The V-ATPase complex component RNAseK is required for lysosomal hydrolase delivery and autophagosome degradation

Overexpression of Nrf2 attenuates Carmustine-induced cytotoxicity in U87MG human glioma cells

Leucine/glutamine and v-ATPase/lysosomal acidification via mTORC1 activation are required for position-dependent regeneration

Guanidine‐Derived Polymeric Nanoinhibitors Target the Lysosomal V‐ATPase and Activate AMPK Pathway to Ameliorate Liver Lipid Accumulation

Hochschulschrift

mTORC1 regulates lysosomal catabolic activity by preventing V-ATPase assembly

Defective Lamtor5 Leads to Autoimmunity by Deregulating v‐ATPase and Lysosomal Acidification

NDST3 deacetylates α‐tubulin and suppresses V‐ATPase assembly and lysosomal acidification

The emerging roles of vacuolar-type ATPase-dependent Lysosomal acidification in neurodegenerative diseases

STAT3 associates with vacuolar H+-ATPase and regulates cytosolic and lysosomal pH

Antipsychotics possess anti-glioblastoma activity by disrupting lysosomal function and inhibiting oncogenic signaling by stabilizing PTEN

Beta-estradiol attenuates hypoxic pulmonary hypertension by stabilizing the expression of p27kip1in rats

Direct control of lysosomal catabolic activity by mTORC1 through regulation of V-ATPase assembly

The V-ATPase complex component RNAseK is required for lysosomal hydrolase delivery and autophagosome degradation

Overexpression of Nrf2 attenuates Carmustine-induced cytotoxicity in U87MG human glioma cells

Leucine/glutamine and v-ATPase/lysosomal acidification via mTORC1 activation are required for position-dependent regeneration

Guanidine‐Derived Polymeric Nanoinhibitors Target the Lysosomal V‐ATPase and Activate AMPK Pathway to Ameliorate Liver Lipid Accumulation

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Overexpression of V-ATPase B2 attenuates lung injury/fibrosis by stabilizing lysosomal membrane permeabilization and increasing collagen degradation

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Other Objects (12)

mTORC1 regulates lysosomal catabolic activity by preventing V-ATPase assembly

Defective Lamtor5 Leads to Autoimmunity by Deregulating v‐ATPase and Lysosomal Acidification

NDST3 deacetylates α‐tubulin and suppresses V‐ATPase assembly and lysosomal acidification

The emerging roles of vacuolar-type ATPase-dependent Lysosomal acidification in neurodegenerative diseases

STAT3 associates with vacuolar H+-ATPase and regulates cytosolic and lysosomal pH

Antipsychotics possess anti-glioblastoma activity by disrupting lysosomal function and inhibiting oncogenic signaling by stabilizing PTEN

Beta-estradiol attenuates hypoxic pulmonary hypertension by stabilizing the expression of p27kip1in rats

Direct control of lysosomal catabolic activity by mTORC1 through regulation of V-ATPase assembly

The V-ATPase complex component RNAseK is required for lysosomal hydrolase delivery and autophagosome degradation

Overexpression of Nrf2 attenuates Carmustine-induced cytotoxicity in U87MG human glioma cells

Leucine/glutamine and v-ATPase/lysosomal acidification via mTORC1 activation are required for position-dependent regeneration

Guanidine‐Derived Polymeric Nanoinhibitors Target the Lysosomal V‐ATPase and Activate AMPK Pathway to Ameliorate Liver Lipid Accumulation

mTORC1 regulates lysosomal catabolic activity by preventing V-ATPase assembly

Defective Lamtor5 Leads to Autoimmunity by Deregulating v‐ATPase and Lysosomal Acidification

NDST3 deacetylates α‐tubulin and suppresses V‐ATPase assembly and lysosomal acidification

The emerging roles of vacuolar-type ATPase-dependent Lysosomal acidification in neurodegenerative diseases

STAT3 associates with vacuolar H+-ATPase and regulates cytosolic and lysosomal pH

Antipsychotics possess anti-glioblastoma activity by disrupting lysosomal function and inhibiting oncogenic signaling by stabilizing PTEN

Beta-estradiol attenuates hypoxic pulmonary hypertension by stabilizing the expression of p27kip1in rats

Direct control of lysosomal catabolic activity by mTORC1 through regulation of V-ATPase assembly

The V-ATPase complex component RNAseK is required for lysosomal hydrolase delivery and autophagosome degradation

Overexpression of Nrf2 attenuates Carmustine-induced cytotoxicity in U87MG human glioma cells

Leucine/glutamine and v-ATPase/lysosomal acidification via mTORC1 activation are required for position-dependent regeneration

Guanidine‐Derived Polymeric Nanoinhibitors Target the Lysosomal V‐ATPase and Activate AMPK Pathway to Ameliorate Liver Lipid Accumulation

mTORC1 regulates lysosomal catabolic activity by preventing V-ATPase assembly

Defective Lamtor5 Leads to Autoimmunity by Deregulating v‐ATPase and Lysosomal Acidification

NDST3 deacetylates α‐tubulin and suppresses V‐ATPase assembly and lysosomal acidification

The emerging roles of vacuolar-type ATPase-dependent Lysosomal acidification in neurodegenerative diseases

STAT3 associates with vacuolar H+-ATPase and regulates cytosolic and lysosomal pH

Antipsychotics possess anti-glioblastoma activity by disrupting lysosomal function and inhibiting oncogenic signaling by stabilizing PTEN

Beta-estradiol attenuates hypoxic pulmonary hypertension by stabilizing the expression of p27kip1in rats

Direct control of lysosomal catabolic activity by mTORC1 through regulation of V-ATPase assembly

The V-ATPase complex component RNAseK is required for lysosomal hydrolase delivery and autophagosome degradation

Overexpression of Nrf2 attenuates Carmustine-induced cytotoxicity in U87MG human glioma cells

Leucine/glutamine and v-ATPase/lysosomal acidification via mTORC1 activation are required for position-dependent regeneration

Guanidine‐Derived Polymeric Nanoinhibitors Target the Lysosomal V‐ATPase and Activate AMPK Pathway to Ameliorate Liver Lipid Accumulation

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