Langerhans cells transport Leishmania major from the infected skin to the draining lymph node for presentation to antigen-specific T cells
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Erschienen in
-
In: European journal of immunology (1993) 23, 7, 1595 - 1601
- Schlagwort
-
Immunologie
Infektion
Infektiologie
- Ereignis
-
Veröffentlichung
- (wo)
-
Würzburg
- (wer)
-
Universität Würzburg
- (wann)
-
2010
- Urheber
-
Moll, Heidrun
Fuchs, Harald
Blank, Christine
Röllinghoff, Martin
- URN
-
urn:nbn:de:bvb:20-opus-46023
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:57 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Moll, Heidrun
- Fuchs, Harald
- Blank, Christine
- Röllinghoff, Martin
- Universität Würzburg
Entstanden
- 2010
Ähnliche Objekte (12)
Experimental cutaneous leishmaniasis: Langerhans cells internalize Leishmania major and induce an antigen-specific T-cell response.
Epidermal Langerhans cells are critical for immunoregulation of cutaneous leishmaniasis
Murine epidermal Langerhans cells are potent stimulators of an antigen-specific T cell response to Leishmania major, the cause of cutaneous leishmaniasis
Development of helper T cell subsets: a central role for interleukin 12
Resistance to murine cutaneous leishmaniasis is mediated by Th1 cells, but disease-promoting CD4+ cells are different from Th2 cells
Mechanism of dendritic cell-based vaccination against Leishmania major
L3T4+ T cells promoting susceptibility to murine cutaneous leishmaniasis express the surface marker Ly-24 (Pgp-1)
Parasitism of epidermal Langerhans cells in experimental cutaneous leishmaniasis with Leishmania major
T-cell reactivity to purified lipophosphoglycan from Leishmania major: A model for analysis of the cellular immune response to microbial carbohydrates.
Analysis of variables associated with the promotion of resistance, and its abrogation, in T cell-reconstituted nude mice infected with Leishmania major
Signaling via Interleukin-4 Receptor alpha chain during dendritic cell–mediated vaccination is required to induce protective immunity against Leishmania major in susceptible BALB/c mice
Resistance to cutaneous leishmaniasis in nude mice injected with L3T4+ T cells but not with Ly-2+ T cells
Experimental cutaneous leishmaniasis: Langerhans cells internalize Leishmania major and induce an antigen-specific T-cell response.
Epidermal Langerhans cells are critical for immunoregulation of cutaneous leishmaniasis
Murine epidermal Langerhans cells are potent stimulators of an antigen-specific T cell response to Leishmania major, the cause of cutaneous leishmaniasis
Development of helper T cell subsets: a central role for interleukin 12
Resistance to murine cutaneous leishmaniasis is mediated by Th1 cells, but disease-promoting CD4+ cells are different from Th2 cells
Mechanism of dendritic cell-based vaccination against Leishmania major
L3T4+ T cells promoting susceptibility to murine cutaneous leishmaniasis express the surface marker Ly-24 (Pgp-1)
Parasitism of epidermal Langerhans cells in experimental cutaneous leishmaniasis with Leishmania major
T-cell reactivity to purified lipophosphoglycan from Leishmania major: A model for analysis of the cellular immune response to microbial carbohydrates.
Analysis of variables associated with the promotion of resistance, and its abrogation, in T cell-reconstituted nude mice infected with Leishmania major
Signaling via Interleukin-4 Receptor alpha chain during dendritic cell–mediated vaccination is required to induce protective immunity against Leishmania major in susceptible BALB/c mice
Resistance to cutaneous leishmaniasis in nude mice injected with L3T4+ T cells but not with Ly-2+ T cells
Experimental cutaneous leishmaniasis: Langerhans cells internalize Leishmania major and induce an antigen-specific T-cell response.
Epidermal Langerhans cells are critical for immunoregulation of cutaneous leishmaniasis
Murine epidermal Langerhans cells are potent stimulators of an antigen-specific T cell response to Leishmania major, the cause of cutaneous leishmaniasis
Development of helper T cell subsets: a central role for interleukin 12
Resistance to murine cutaneous leishmaniasis is mediated by Th1 cells, but disease-promoting CD4+ cells are different from Th2 cells
Mechanism of dendritic cell-based vaccination against Leishmania major
L3T4+ T cells promoting susceptibility to murine cutaneous leishmaniasis express the surface marker Ly-24 (Pgp-1)
Parasitism of epidermal Langerhans cells in experimental cutaneous leishmaniasis with Leishmania major
T-cell reactivity to purified lipophosphoglycan from Leishmania major: A model for analysis of the cellular immune response to microbial carbohydrates.
Analysis of variables associated with the promotion of resistance, and its abrogation, in T cell-reconstituted nude mice infected with Leishmania major
Signaling via Interleukin-4 Receptor alpha chain during dendritic cell–mediated vaccination is required to induce protective immunity against Leishmania major in susceptible BALB/c mice