Protective functions of myricetin in LPS-induced cardiomyocytes H9c2 cells injury by regulation of MALAT1
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2047-783X
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Protective functions of myricetin in LPS-induced cardiomyocytes H9c2 cells injury by regulation of MALAT1 ; volume:24 ; number:1 ; day:26 ; month:4 ; year:2019 ; pages:1-9 ; date:12.2019
European journal of medical research ; 24, Heft 1 (26.4.2019), 1-9, 12.2019
- Creator
-
Sun, Jinliang
Sun, Jianhui
Zhou, Xuezhong
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1186/s40001-019-0378-5
- URN
-
urn:nbn:de:101:1-2019051921572702318662
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:57 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Sun, Jinliang
- Sun, Jianhui
- Zhou, Xuezhong
- SpringerLink (Online service)
Other Objects (12)
Retraction Note: Protective functions of myricetin in LPS-induced cardiomyocytes H9c2 cells injury by regulation of MALAT1
3,3′-Diindolylmethane protects cardiomyocytes from LPS-induced inflammatory response and apoptosis
Impacts of long noncoding RNA MALAT1 on LPS-induced periodontitis via modulating miR-155/SIRT1 axis
Protective effect of oxytocin on LPS-induced acute lung injury in mice
Protective Effect of Phillyrin on Lethal LPS-Induced Neutrophil Inflammation in Zebrafish
N-Acetylcysteine Suppresses LPS-Induced Pathological Angiogenesis
Sex-differences in LPS-induced neonatal lung injury
The protective effect of procyanidin against LPS-induced acute gut injury by the regulations of oxidative state
Carvacrol alleviates LPS-induced myocardial dysfunction by inhibiting the TLR4/MyD88/NF-κB and NLRP3 inflammasome in cardiomyocytes
Superior protective effects of PGE2 priming mesenchymal stem cells against LPS-induced acute lung injury (ALI) through macrophage immunomodulation
IRAK-1 is a mediator of LPS-induced pulmonary inflammation
Suppression of LPS-induced tau hyperphosphorylation by serum amyloid A
Retraction Note: Protective functions of myricetin in LPS-induced cardiomyocytes H9c2 cells injury by regulation of MALAT1
3,3′-Diindolylmethane protects cardiomyocytes from LPS-induced inflammatory response and apoptosis
Impacts of long noncoding RNA MALAT1 on LPS-induced periodontitis via modulating miR-155/SIRT1 axis
Protective effect of oxytocin on LPS-induced acute lung injury in mice
Protective Effect of Phillyrin on Lethal LPS-Induced Neutrophil Inflammation in Zebrafish
N-Acetylcysteine Suppresses LPS-Induced Pathological Angiogenesis
Sex-differences in LPS-induced neonatal lung injury
The protective effect of procyanidin against LPS-induced acute gut injury by the regulations of oxidative state
Carvacrol alleviates LPS-induced myocardial dysfunction by inhibiting the TLR4/MyD88/NF-κB and NLRP3 inflammasome in cardiomyocytes
Superior protective effects of PGE2 priming mesenchymal stem cells against LPS-induced acute lung injury (ALI) through macrophage immunomodulation
IRAK-1 is a mediator of LPS-induced pulmonary inflammation
Suppression of LPS-induced tau hyperphosphorylation by serum amyloid A
Retraction Note: Protective functions of myricetin in LPS-induced cardiomyocytes H9c2 cells injury by regulation of MALAT1
3,3′-Diindolylmethane protects cardiomyocytes from LPS-induced inflammatory response and apoptosis
Impacts of long noncoding RNA MALAT1 on LPS-induced periodontitis via modulating miR-155/SIRT1 axis
Protective effect of oxytocin on LPS-induced acute lung injury in mice
Protective Effect of Phillyrin on Lethal LPS-Induced Neutrophil Inflammation in Zebrafish
N-Acetylcysteine Suppresses LPS-Induced Pathological Angiogenesis
Sex-differences in LPS-induced neonatal lung injury
The protective effect of procyanidin against LPS-induced acute gut injury by the regulations of oxidative state
Carvacrol alleviates LPS-induced myocardial dysfunction by inhibiting the TLR4/MyD88/NF-κB and NLRP3 inflammasome in cardiomyocytes
Superior protective effects of PGE2 priming mesenchymal stem cells against LPS-induced acute lung injury (ALI) through macrophage immunomodulation
IRAK-1 is a mediator of LPS-induced pulmonary inflammation