3D collagen migration patterns reveal a SMAD3-dependent and TGF-β1-independent mechanism of recruitment for tumour-associated fibroblasts in lung adenocarcinoma
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1532-1827
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
3D collagen migration patterns reveal a SMAD3-dependent and TGF-β1-independent mechanism of recruitment for tumour-associated fibroblasts in lung adenocarcinoma ; day:26 ; month:12 ; year:2022 ; pages:1-15
British journal of cancer ; (26.12.2022), 1-15
- Creator
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Juste-Lanas, Yago
Díaz-Valdivia, Natalia
Llorente, Alejandro
Ikemori, Rafael
Bernardo, Alejandro
Arshakyan, Marselina
Borau, Carlos
Ramírez, Josep
Ruffinelli, José Carlos
Nadal, Ernest
Reguart, Noemí
García-Aznar, José M.
Alcaraz, Jordi
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s41416-022-02093-x
- URN
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urn:nbn:de:101:1-2023030721182664061945
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:43 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Juste-Lanas, Yago
- Díaz-Valdivia, Natalia
- Llorente, Alejandro
- Ikemori, Rafael
- Bernardo, Alejandro
- Arshakyan, Marselina
- Borau, Carlos
- Ramírez, Josep
- Ruffinelli, José Carlos
- Nadal, Ernest
- Reguart, Noemí
- García-Aznar, José M.
- Alcaraz, Jordi
- SpringerLink (Online service)
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The expression of TGF-β1, Smad3, phospho-Smad3 and Smad7 is correlated with the development and invasion of nonfunctioning pituitary adenomas
circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activation of MMPs in pancreatic cancer
Serum TGF-β1 and SMAD3 levels are closely associated with coronary artery disease
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YAP/TAZ regulates TGF-β/Smad3 signaling by induction of Smad7 via AP-1 in human skin dermal fibroblasts
Smad3 Promotes Cancer‐Associated Fibroblasts Generation via Macrophage–Myofibroblast Transition
The expression of TGF-β1, Smad3, phospho-Smad3 and Smad7 is correlated with the development and invasion of nonfunctioning pituitary adenomas
circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activation of MMPs in pancreatic cancer
Serum TGF-β1 and SMAD3 levels are closely associated with coronary artery disease
Alltrans retinoic acid recruits Smad3 to drive TGFbeta signaling in lung fibroblasts : new insights for the management of bronchopulmonary dysplasia with retinoids
NFAT3 and TGF-β/SMAD3 regulate the expression of miR-140 in osteoarthritis
circRIP2 accelerates bladder cancer progression via miR-1305/Tgf-β2/smad3 pathway
Hepatocellular carcinoma cells remodel the pro-metastatic tumour microenvironment through recruitment and activation of fibroblasts via paracrine Egfl7 signaling
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