Hypoxia promotes an inflammatory phenotype of fibroblasts in pancreatic cancer
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2157-9024
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Hypoxia promotes an inflammatory phenotype of fibroblasts in pancreatic cancer ; volume:11 ; number:1 ; day:15 ; month:9 ; year:2022 ; pages:1-9 ; date:12.2022
Oncogenesis ; 11, Heft 1 (15.9.2022), 1-9, 12.2022
- Urheber
-
Mello, Ashley M.
Ngodup, Tenzin
Lee, Yusoo
Donahue, Katelyn L.
Li, Jinju
Rao, Arvind
Carpenter, Eileen S.
Crawford, Howard C.
Pasca di Magliano, Marina
Lee, Kyoung Eun
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1038/s41389-022-00434-2
- URN
-
urn:nbn:de:101:1-2022120221113394823724
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:30 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Mello, Ashley M.
- Ngodup, Tenzin
- Lee, Yusoo
- Donahue, Katelyn L.
- Li, Jinju
- Rao, Arvind
- Carpenter, Eileen S.
- Crawford, Howard C.
- Pasca di Magliano, Marina
- Lee, Kyoung Eun
- SpringerLink (Online service)
Ähnliche Objekte (12)
Hypoxia-induced circRNF13 promotes the progression and glycolysis of pancreatic cancer
BNIP3+ fibroblasts associated with hypoxia and inflammation predict prognosis and immunotherapy response in pancreatic ductal adenocarcinoma
Cancer-associated fibroblasts in pancreatic ductal adenocarcinoma
CD9 promotes TβR2–TβR1 association driving the transition of human dermal fibroblasts to myofibroblast under hypoxia
Integrin-mediated tumor-stroma interfacial signaling promotes basal-like and immune exclusion phenotype in pancreatic cancer
Chronic hypoxia for the adaptation of extracellular vesicle phenotype
A Marfan syndrome gene expression phenotype in cultured skin fibroblasts
Transcriptional response to hypoxia of normal and rheumatoid arthritis synovial fibroblasts
Endogenously expressed IL-4Rα promotes the malignant phenotype of human pancreatic cancer in vitro and in vivo
Hypoxia-inducible factor-2α promotes tumor progression and has crosstalk with Wnt/β-catenin signaling in pancreatic cancer
Src kinase inhibition promotes the chondrocyte phenotype
Hypoxia Promotes Neutrophil Survival After Acute Myocardial Infarction
Hypoxia-induced circRNF13 promotes the progression and glycolysis of pancreatic cancer
BNIP3+ fibroblasts associated with hypoxia and inflammation predict prognosis and immunotherapy response in pancreatic ductal adenocarcinoma
Cancer-associated fibroblasts in pancreatic ductal adenocarcinoma
CD9 promotes TβR2–TβR1 association driving the transition of human dermal fibroblasts to myofibroblast under hypoxia
Integrin-mediated tumor-stroma interfacial signaling promotes basal-like and immune exclusion phenotype in pancreatic cancer
Chronic hypoxia for the adaptation of extracellular vesicle phenotype
A Marfan syndrome gene expression phenotype in cultured skin fibroblasts
Transcriptional response to hypoxia of normal and rheumatoid arthritis synovial fibroblasts
Endogenously expressed IL-4Rα promotes the malignant phenotype of human pancreatic cancer in vitro and in vivo
Hypoxia-inducible factor-2α promotes tumor progression and has crosstalk with Wnt/β-catenin signaling in pancreatic cancer
Src kinase inhibition promotes the chondrocyte phenotype
Hypoxia Promotes Neutrophil Survival After Acute Myocardial Infarction
Hypoxia-induced circRNF13 promotes the progression and glycolysis of pancreatic cancer
BNIP3+ fibroblasts associated with hypoxia and inflammation predict prognosis and immunotherapy response in pancreatic ductal adenocarcinoma
Cancer-associated fibroblasts in pancreatic ductal adenocarcinoma
CD9 promotes TβR2–TβR1 association driving the transition of human dermal fibroblasts to myofibroblast under hypoxia
Integrin-mediated tumor-stroma interfacial signaling promotes basal-like and immune exclusion phenotype in pancreatic cancer
Chronic hypoxia for the adaptation of extracellular vesicle phenotype
A Marfan syndrome gene expression phenotype in cultured skin fibroblasts
Transcriptional response to hypoxia of normal and rheumatoid arthritis synovial fibroblasts
Endogenously expressed IL-4Rα promotes the malignant phenotype of human pancreatic cancer in vitro and in vivo
Hypoxia-inducible factor-2α promotes tumor progression and has crosstalk with Wnt/β-catenin signaling in pancreatic cancer
Src kinase inhibition promotes the chondrocyte phenotype