Tumor-recruited M2 macrophages promote gastric and breast cancer metastasis via M2 macrophage-secreted CHI3L1 protein
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1756-8722
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Tumor-recruited M2 macrophages promote gastric and breast cancer metastasis via M2 macrophage-secreted CHI3L1 protein ; volume:10 ; number:1 ; day:1 ; month:2 ; year:2017 ; pages:1-13 ; date:12.2017
Journal of hematology & oncology ; 10, Heft 1 (1.2.2017), 1-13, 12.2017
- Urheber
-
Chen, Yulei
- Beteiligte Personen und Organisationen
-
Zhang, Siyuan
Wang, Qizhi
Zhang, Xiaobo
SpringerLink (Online service)
- DOI
-
10.1186/s13045-017-0408-0
- URN
-
urn:nbn:de:101:1-2018091512102685240753
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:31 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Chen, Yulei
- Zhang, Siyuan
- Wang, Qizhi
- Zhang, Xiaobo
- SpringerLink (Online service)
Ähnliche Objekte (12)
The characteristics of circRNA as competing endogenous RNA in pathogenesis of acute myeloid leukemia
An Emerging Role of Extracellular Traps in Chronic Rhinosinusitis
Multiplexed Ultrasound Imaging Using Spectral Analysis on Gas Vesicles
Expression of area-specific M2-macrophage phenotype by recruited rat monocytes in duct-ligation pancreatitis
M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma
M2-like tumor-associated macrophage-secreted CCL2 facilitates gallbladder cancer stemness and metastasis
Spatio-temporal analysis of the driving factors of urban land use expansion in China: A study of the Yangtze River Delta region
Full-color three-loop three-point form factors in ?? = 4 SYM
Carbon Nanomaterials Promote M1/M2 Macrophage Activation
M2
MSC-secreted TGF-β regulates lipopolysaccharide-stimulated macrophage M2-like polarization via the Akt/FoxO1 pathway
Wipptischanlage M2-22 und M2-23
The characteristics of circRNA as competing endogenous RNA in pathogenesis of acute myeloid leukemia
An Emerging Role of Extracellular Traps in Chronic Rhinosinusitis
Multiplexed Ultrasound Imaging Using Spectral Analysis on Gas Vesicles
Expression of area-specific M2-macrophage phenotype by recruited rat monocytes in duct-ligation pancreatitis
M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma
M2-like tumor-associated macrophage-secreted CCL2 facilitates gallbladder cancer stemness and metastasis
Spatio-temporal analysis of the driving factors of urban land use expansion in China: A study of the Yangtze River Delta region
Full-color three-loop three-point form factors in ?? = 4 SYM
Carbon Nanomaterials Promote M1/M2 Macrophage Activation
M2
MSC-secreted TGF-β regulates lipopolysaccharide-stimulated macrophage M2-like polarization via the Akt/FoxO1 pathway
Wipptischanlage M2-22 und M2-23
The characteristics of circRNA as competing endogenous RNA in pathogenesis of acute myeloid leukemia
An Emerging Role of Extracellular Traps in Chronic Rhinosinusitis
Multiplexed Ultrasound Imaging Using Spectral Analysis on Gas Vesicles
Expression of area-specific M2-macrophage phenotype by recruited rat monocytes in duct-ligation pancreatitis
M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma
M2-like tumor-associated macrophage-secreted CCL2 facilitates gallbladder cancer stemness and metastasis
Spatio-temporal analysis of the driving factors of urban land use expansion in China: A study of the Yangtze River Delta region
Full-color three-loop three-point form factors in ?? = 4 SYM
Carbon Nanomaterials Promote M1/M2 Macrophage Activation
M2
MSC-secreted TGF-β regulates lipopolysaccharide-stimulated macrophage M2-like polarization via the Akt/FoxO1 pathway