Proteotoxic stress-induced apoptosis in cancer cells: understanding the susceptibility and enhancing the potency

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2058-7716

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Proteotoxic stress-induced apoptosis in cancer cells: understanding the susceptibility and enhancing the potency ; volume:8 ; number:1 ; day:4 ; month:10 ; year:2022 ; pages:1-12 ; date:12.2022
Cell death discovery ; 8, Heft 1 (4.10.2022), 1-12, 12.2022

Creator: Iuliano, Luca
Dalla, Emiliano
Picco, Raffaella
Mallavarapu, Showmeya
Minisini, Martina
Malavasi, Eleonora
Brancolini, Claudio

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41420-022-01202-2

URN: urn:nbn:de:101:1-2022122409262136020118

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:30 AM CEST

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Associated

Iuliano, Luca
Dalla, Emiliano
Picco, Raffaella
Mallavarapu, Showmeya
Minisini, Martina
Malavasi, Eleonora
Brancolini, Claudio
SpringerLink (Online service)

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Proteotoxic stress-induced apoptosis in cancer cells: understanding the susceptibility and enhancing the potency

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Other Objects (12)

(+)-Catechin protects dermal fibroblasts against oxidative stress-induced apoptosis

Stress-induced sleep-like inactivity modulates stress susceptibility in mice

Combination drug therapy prevents CIAKI by suppressing ER stress-induced apoptosis

CRTH2 promotes endoplasmic reticulum stress‐induced cardiomyocyte apoptosis through m‐calpain

The Role of Stress-Induced Changes of Homer1 Expression in Stress Susceptibility

IL17F Expression as an Early Sign of Oxidative Stress-Induced Cytotoxicity/Apoptosis

Cudraflavone B induces human glioblastoma cells apoptosis via ER stress-induced autophagy

p,p’-DDT induces testicular oxidative stress-induced apoptosis in adult rats

GKN2 promotes oxidative stress-induced gastric cancer cell apoptosis via the Hsc70 pathway

Bcl-2 regulates store-operated Ca2+ entry to modulate ER stress-induced apoptosis

Cellular apoptosis susceptibility (CSE1L/CAS) protein in cancer metastasis and chemotherapeutic drug-induced apoptosis

3′,4′-dihydroxyflavonol ameliorates endoplasmic reticulum stress-induced apoptosis and endothelial dysfunction in mice

(+)-Catechin protects dermal fibroblasts against oxidative stress-induced apoptosis

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Combination drug therapy prevents CIAKI by suppressing ER stress-induced apoptosis

CRTH2 promotes endoplasmic reticulum stress‐induced cardiomyocyte apoptosis through m‐calpain

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IL17F Expression as an Early Sign of Oxidative Stress-Induced Cytotoxicity/Apoptosis

Cudraflavone B induces human glioblastoma cells apoptosis via ER stress-induced autophagy

p,p’-DDT induces testicular oxidative stress-induced apoptosis in adult rats

GKN2 promotes oxidative stress-induced gastric cancer cell apoptosis via the Hsc70 pathway

Bcl-2 regulates store-operated Ca2+ entry to modulate ER stress-induced apoptosis

Cellular apoptosis susceptibility (CSE1L/CAS) protein in cancer metastasis and chemotherapeutic drug-induced apoptosis

3′,4′-dihydroxyflavonol ameliorates endoplasmic reticulum stress-induced apoptosis and endothelial dysfunction in mice

(+)-Catechin protects dermal fibroblasts against oxidative stress-induced apoptosis

Stress-induced sleep-like inactivity modulates stress susceptibility in mice

Combination drug therapy prevents CIAKI by suppressing ER stress-induced apoptosis

CRTH2 promotes endoplasmic reticulum stress‐induced cardiomyocyte apoptosis through m‐calpain

The Role of Stress-Induced Changes of Homer1 Expression in Stress Susceptibility

IL17F Expression as an Early Sign of Oxidative Stress-Induced Cytotoxicity/Apoptosis

Cudraflavone B induces human glioblastoma cells apoptosis via ER stress-induced autophagy

p,p’-DDT induces testicular oxidative stress-induced apoptosis in adult rats

GKN2 promotes oxidative stress-induced gastric cancer cell apoptosis via the Hsc70 pathway

Bcl-2 regulates store-operated Ca2+ entry to modulate ER stress-induced apoptosis

Cellular apoptosis susceptibility (CSE1L/CAS) protein in cancer metastasis and chemotherapeutic drug-induced apoptosis

3′,4′-dihydroxyflavonol ameliorates endoplasmic reticulum stress-induced apoptosis and endothelial dysfunction in mice

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