Early deficits in an in vitro striatal microcircuit model carrying the Parkinson’s GBA-N370S mutation

to related objects

Location
Deutsche Nationalbibliothek Frankfurt am Main
Extent
1 Online-Ressource.
Language
Englisch

Bibliographic citation
Early deficits in an in vitro striatal microcircuit model carrying the Parkinson’s GBA-N370S mutation ; volume:10 ; number:1 ; day:12 ; month:4 ; year:2024 ; pages:1-15 ; date:12.2024
npj Parkinson's Disease ; 10, Heft 1 (12.4.2024), 1-15, 12.2024

Creator
Do, Quyen B.
Noor, Humaira
Marquez-Gomez, Ricardo
Cramb, Kaitlyn M. L.
Ng, Bryan
Abbey, Ajantha
Ibarra-Aizpurua, Naroa
Caiazza, Maria Claudia
Sharifi, Parnaz
Lang, Charmaine
Beccano-Kelly, Dayne
Baleriola, Jimena
Bengoa-Vergniory, Nora
Wade-Martins, Richard
Contributor
SpringerLink (Online service)

DOI
10.1038/s41531-024-00694-2
URN
urn:nbn:de:101:1-2406252123015.781164916966
Rights
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
Last update
14.08.2025, 10:51 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Show original at data provider

Associated

  • Do, Quyen B.
  • Noor, Humaira
  • Marquez-Gomez, Ricardo
  • Cramb, Kaitlyn M. L.
  • Ng, Bryan
  • Abbey, Ajantha
  • Ibarra-Aizpurua, Naroa
  • Caiazza, Maria Claudia
  • Sharifi, Parnaz
  • Lang, Charmaine
  • Beccano-Kelly, Dayne
  • Baleriola, Jimena
  • Bengoa-Vergniory, Nora
  • Wade-Martins, Richard
  • SpringerLink (Online service)

Other Objects (12)

Dual-Task Performance in GBA Parkinson’s Disease

Dual-Task Performance in GBA Parkinson’s Disease

GBA-associated Parkinson: does personalized medicine work in Parkinson's disease?

Hochschulschrift

GBA-associated Parkinson: does personalized medicine work in Parkinson's disease?

GBA mutations and Parkinson's disease in Brazil

GBA mutations and Parkinson's disease in Brazil

Dual-Task Performance in GBA Parkinson's Disease

Dual-Task Performance in GBA Parkinson's Disease

GBA-associated Parkinson’s disease in Hungary: clinical features and genetic insights

GBA-associated Parkinson’s disease in Hungary: clinical features and genetic insights

Parkinson’s disease patients with heterozygous GBA-mutation : longitudinal phenotyping of motor and non-motor symptoms – more rapid progression compared to Parkinson’s disease patients without GBA-mutation

Parkinson’s disease patients with heterozygous GBA-mutation : longitudinal phenotyping of motor and non-motor symptoms – more rapid progression compared to Parkinson’s disease patients without GBA-mutation

Autonomic failure in Parkinson’s disease is associated with striatal dopamine deficiencies

Autonomic failure in Parkinson’s disease is associated with striatal dopamine deficiencies

Local striatal volume and motor reserve in drug-naïve Parkinson’s disease

Local striatal volume and motor reserve in drug-naïve Parkinson’s disease

Transcriptome deregulation of peripheral monocytes and whole blood in GBA-related Parkinson’s disease

Transcriptome deregulation of peripheral monocytes and whole blood in GBA-related Parkinson’s disease

Modulating brain networks associated with cognitive deficits in Parkinson’s disease

Modulating brain networks associated with cognitive deficits in Parkinson’s disease

Motor skill learning, retention, and control deficits in Parkinson´s disease

Motor skill learning, retention, and control deficits in Parkinson´s disease

Putative second hit rare genetic variants in families with seemingly GBA-associated Parkinson’s disease

Putative second hit rare genetic variants in families with seemingly GBA-associated Parkinson’s disease

Related objects