The MYCN inhibitor BGA002 restores the retinoic acid response leading to differentiation or apoptosis by the mTOR block in MYCN-amplified neuroblastoma
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1756-9966
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
The MYCN inhibitor BGA002 restores the retinoic acid response leading to differentiation or apoptosis by the mTOR block in MYCN-amplified neuroblastoma ; volume:41 ; number:1 ; day:30 ; month:4 ; year:2022 ; pages:1-19 ; date:12.2022
Journal of experimental & clinical cancer research ; 41, Heft 1 (30.4.2022), 1-19, 12.2022
- Creator
-
Lampis, Silvia
Raieli, Salvatore
Montemurro, Luca
Bartolucci, Damiano
Amadesi, Camilla
Bortolotti, Sonia
Angelucci, Silvia
Scardovi, Anna Lisa
Nieddu, Giammario
Cerisoli, Lucia
Paganelli, Francesca
Valente, Sabrina
Fischer, Matthias
Martelli, Alberto Maria
Pasquinelli, Gianandrea
Pession, Andrea
Hrelia, Patrizia
Tonelli, Roberto
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1186/s13046-022-02367-5
- URN
-
urn:nbn:de:101:1-2022071011034089968947
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:30 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Lampis, Silvia
- Raieli, Salvatore
- Montemurro, Luca
- Bartolucci, Damiano
- Amadesi, Camilla
- Bortolotti, Sonia
- Angelucci, Silvia
- Scardovi, Anna Lisa
- Nieddu, Giammario
- Cerisoli, Lucia
- Paganelli, Francesca
- Valente, Sabrina
- Fischer, Matthias
- Martelli, Alberto Maria
- Pasquinelli, Gianandrea
- Pession, Andrea
- Hrelia, Patrizia
- Tonelli, Roberto
- SpringerLink (Online service)
Other Objects (12)
SMAD9-MYCN positive feedback loop represents a unique dependency for MYCN-amplified neuroblastoma
MTHFD1 regulates the NADPH redox homeostasis in MYCN-amplified neuroblastoma
Inhibitors of ribosome biogenesis repress the growth of MYCN-amplified neuroblastoma
The long non-coding RNA MYCNOS-01 regulates MYCN protein levels and affects growth of MYCN-amplified rhabdomyosarcoma and neuroblastoma cells
Aurora-A prevents transcription-replication conflicts in MYCN-amplified neuroblastoma
LRP8‐mediated selenocysteine uptake is a targetable vulnerability in MYCN‐amplified neuroblastoma
Conditional expression of retrovirally delivered anti-MYCN shRNA as an in vitro model system to study neuronal differentiation in MYCN-amplified neuroblastoma
Inhibition of autophagy potentiates the efficacy of Gli inhibitor GANT-61 in MYCN-amplified neuroblastoma cells
Retinoic acid and TGF-β signalling cooperate to overcome MYCN-induced retinoid resistance
Retinoic acid and TGF-β signalling cooperate to overcome MYCN-induced retinoid resistance
Inhibition von Aurora-A als neue Therapiestrategie gegen MYCN-amplifizierte Neuroblastome : = Inhibition of Aurora A as a novel therapeutic strategy against MYCN-amplified neuroblastoma
MYCN dependency of MYCN amplified neuroblastoma cell lines analyzed in relation to their interaction with BET proteins and in a novel orthotopic mouse model
SMAD9-MYCN positive feedback loop represents a unique dependency for MYCN-amplified neuroblastoma
MTHFD1 regulates the NADPH redox homeostasis in MYCN-amplified neuroblastoma
Inhibitors of ribosome biogenesis repress the growth of MYCN-amplified neuroblastoma
The long non-coding RNA MYCNOS-01 regulates MYCN protein levels and affects growth of MYCN-amplified rhabdomyosarcoma and neuroblastoma cells
Aurora-A prevents transcription-replication conflicts in MYCN-amplified neuroblastoma
LRP8‐mediated selenocysteine uptake is a targetable vulnerability in MYCN‐amplified neuroblastoma
Conditional expression of retrovirally delivered anti-MYCN shRNA as an in vitro model system to study neuronal differentiation in MYCN-amplified neuroblastoma
Inhibition of autophagy potentiates the efficacy of Gli inhibitor GANT-61 in MYCN-amplified neuroblastoma cells
Retinoic acid and TGF-β signalling cooperate to overcome MYCN-induced retinoid resistance
Retinoic acid and TGF-β signalling cooperate to overcome MYCN-induced retinoid resistance
Inhibition von Aurora-A als neue Therapiestrategie gegen MYCN-amplifizierte Neuroblastome : = Inhibition of Aurora A as a novel therapeutic strategy against MYCN-amplified neuroblastoma
MYCN dependency of MYCN amplified neuroblastoma cell lines analyzed in relation to their interaction with BET proteins and in a novel orthotopic mouse model
SMAD9-MYCN positive feedback loop represents a unique dependency for MYCN-amplified neuroblastoma
MTHFD1 regulates the NADPH redox homeostasis in MYCN-amplified neuroblastoma
Inhibitors of ribosome biogenesis repress the growth of MYCN-amplified neuroblastoma
The long non-coding RNA MYCNOS-01 regulates MYCN protein levels and affects growth of MYCN-amplified rhabdomyosarcoma and neuroblastoma cells
Aurora-A prevents transcription-replication conflicts in MYCN-amplified neuroblastoma
LRP8‐mediated selenocysteine uptake is a targetable vulnerability in MYCN‐amplified neuroblastoma
Conditional expression of retrovirally delivered anti-MYCN shRNA as an in vitro model system to study neuronal differentiation in MYCN-amplified neuroblastoma
Inhibition of autophagy potentiates the efficacy of Gli inhibitor GANT-61 in MYCN-amplified neuroblastoma cells
Retinoic acid and TGF-β signalling cooperate to overcome MYCN-induced retinoid resistance
Retinoic acid and TGF-β signalling cooperate to overcome MYCN-induced retinoid resistance
Inhibition von Aurora-A als neue Therapiestrategie gegen MYCN-amplifizierte Neuroblastome : = Inhibition of Aurora A as a novel therapeutic strategy against MYCN-amplified neuroblastoma