Hochschulschrift

S100A1 from Damaged Cardiomyocytes Elicits a Chemoattractant Cardiac Fibroblast Phenotype

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Notes: Heidelberg, Universität Heidelberg, Dissertation, 2017

Keyword: Heart cells
Myocardial infarction
Phenotype

Event: Veröffentlichung

(where): Heidelberg

(who): Universitätsbibliothek Heidelberg

(when): 2017

Creator: Didrihsone, Ieva

Contributor: Rappold, Gudrun

DOI: 10.11588/heidok.00022835

URN: urn:nbn:de:bsz:16-heidok-228353

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:54 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Object type

Hochschulschrift

Associated

Didrihsone, Ieva
Rappold, Gudrun
Universitätsbibliothek Heidelberg

Time of origin

2017

Other Objects (12)

Hochschulschrift

S100A1 from damaged cardiomyocytes elicits a chemoattractant cardiac fibroblast phenotype

SNTA1-deficient human cardiomyocytes demonstrate hypertrophic phenotype and calcium handling disorder

Incomplete cellular reprogramming of colorectal cancer cells elicits an epithelial/mesenchymal hybrid phenotype

Arbeitspapier

Pricing Damaged Goods

Artikel

Pricing Damaged Goods

Investigation of the cellular Fabry disease phenotype using human iPSC-derived cardiomyocytes

The transcription factor scleraxis is a critical regulator of cardiac fibroblast phenotype

A positive mechanobiological feedback loop controls bistable switching of cardiac fibroblast phenotype

Exogenous glycosaminoglycans coat damaged bladder surfaces in experimentally damaged mouse bladder

Hochschulschrift

S100A1 from damaged cardiomyocytes elicits a chemoattractant cardiac fibroblast phenotype

SNTA1-deficient human cardiomyocytes demonstrate hypertrophic phenotype and calcium handling disorder

Incomplete cellular reprogramming of colorectal cancer cells elicits an epithelial/mesenchymal hybrid phenotype

Arbeitspapier

Pricing Damaged Goods

Artikel

Pricing Damaged Goods

Investigation of the cellular Fabry disease phenotype using human iPSC-derived cardiomyocytes

The transcription factor scleraxis is a critical regulator of cardiac fibroblast phenotype

A positive mechanobiological feedback loop controls bistable switching of cardiac fibroblast phenotype

Exogenous glycosaminoglycans coat damaged bladder surfaces in experimentally damaged mouse bladder

Hochschulschrift

S100A1 from damaged cardiomyocytes elicits a chemoattractant cardiac fibroblast phenotype

SNTA1-deficient human cardiomyocytes demonstrate hypertrophic phenotype and calcium handling disorder

Incomplete cellular reprogramming of colorectal cancer cells elicits an epithelial/mesenchymal hybrid phenotype

Arbeitspapier

Pricing Damaged Goods

Artikel

Pricing Damaged Goods

Investigation of the cellular Fabry disease phenotype using human iPSC-derived cardiomyocytes

The transcription factor scleraxis is a critical regulator of cardiac fibroblast phenotype

A positive mechanobiological feedback loop controls bistable switching of cardiac fibroblast phenotype

Exogenous glycosaminoglycans coat damaged bladder surfaces in experimentally damaged mouse bladder

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

S100A1 from Damaged Cardiomyocytes Elicits a Chemoattractant Cardiac Fibroblast Phenotype

Object Details

Classification and Topics

Contributors, Places and Time

Further information

Data provider

Object type

Associated

Time of origin

Other Objects (12)

S100A1 from damaged cardiomyocytes elicits a chemoattractant cardiac fibroblast phenotype

SNTA1-deficient human cardiomyocytes demonstrate hypertrophic phenotype and calcium handling disorder

Incomplete cellular reprogramming of colorectal cancer cells elicits an epithelial/mesenchymal hybrid phenotype

Pricing Damaged Goods

Pricing Damaged Goods

Pricing Damaged Goods

Pricing Damaged Goods

Pricing Damaged Goods

Investigation of the cellular Fabry disease phenotype using human iPSC-derived cardiomyocytes

The transcription factor scleraxis is a critical regulator of cardiac fibroblast phenotype

A positive mechanobiological feedback loop controls bistable switching of cardiac fibroblast phenotype

Exogenous glycosaminoglycans coat damaged bladder surfaces in experimentally damaged mouse bladder

S100A1 from damaged cardiomyocytes elicits a chemoattractant cardiac fibroblast phenotype

SNTA1-deficient human cardiomyocytes demonstrate hypertrophic phenotype and calcium handling disorder

Incomplete cellular reprogramming of colorectal cancer cells elicits an epithelial/mesenchymal hybrid phenotype

Pricing Damaged Goods

Pricing Damaged Goods

Pricing Damaged Goods

Pricing Damaged Goods

Pricing Damaged Goods

Investigation of the cellular Fabry disease phenotype using human iPSC-derived cardiomyocytes

The transcription factor scleraxis is a critical regulator of cardiac fibroblast phenotype

A positive mechanobiological feedback loop controls bistable switching of cardiac fibroblast phenotype

Exogenous glycosaminoglycans coat damaged bladder surfaces in experimentally damaged mouse bladder

S100A1 from damaged cardiomyocytes elicits a chemoattractant cardiac fibroblast phenotype

SNTA1-deficient human cardiomyocytes demonstrate hypertrophic phenotype and calcium handling disorder

Incomplete cellular reprogramming of colorectal cancer cells elicits an epithelial/mesenchymal hybrid phenotype

Pricing Damaged Goods

Pricing Damaged Goods

Pricing Damaged Goods

Pricing Damaged Goods

Pricing Damaged Goods

Investigation of the cellular Fabry disease phenotype using human iPSC-derived cardiomyocytes

The transcription factor scleraxis is a critical regulator of cardiac fibroblast phenotype

A positive mechanobiological feedback loop controls bistable switching of cardiac fibroblast phenotype

Exogenous glycosaminoglycans coat damaged bladder surfaces in experimentally damaged mouse bladder

Related objects

Reset password