Hochschulschrift
A novel role of S100B in neuronal trace metal homeostasis associated with dysregulation of the autism-associated SHANK pathway
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Maße
-
30 cm
- Umfang
-
XIX, 166 Blätter
- Sprache
-
Englisch
- Anmerkungen
-
Illustrationen, Diagramme
Universität Ulm, Dissertation, 2018
- Schlagwort
-
Autismus
S-100-Protein
Homöostase
Zink
- Ereignis
-
Veröffentlichung
- (wo)
-
Ulm
- (wann)
-
2018
- Urheber
- Beteiligte Personen und Organisationen
- Inhaltsverzeichnis
- Rechteinformation
-
Bei diesem Objekt liegt nur das Inhaltsverzeichnis digital vor. Der Zugriff darauf ist unbeschränkt möglich.
- Letzte Aktualisierung
-
11.06.2025, 14:02 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Objekttyp
- Hochschulschrift
Beteiligte
Entstanden
- 2018
Ähnliche Objekte (12)
Loss of COMMD1 and copper overload disrupt zinc homeostasis and influence an autism-associated pathway at glutamatergic synapses
Activity regulating signals in neurons associated with energy homeostasis
Immune dysregulation associated with co-occurring germline CBL and SH2B3 variants
Dissecting hormonal and transient effects in Shankopathies associated with autism spectrum disorder
Hyperglycaemia is associated with male infertility and activin dysregulation in Type 1 Diabetes
Structure-Based Discovery of Mouse Trace Amine-Associated Receptor 5 Antagonists
Human trace amine-associated receptor TAAR5 can be activated by trimethylamine
Characterization of the structural and functional impact of autism-associated SHANK3 missense mutations
Investigating the impact of autism-associated SHANK2 genetic defects on early neuronal differentiation
Interferon-driven immune dysregulation in common variable immunodeficiency-associated villous atrophy and norovirus infection
GPCR-specific autoantibody signatures are associated with physiological and pathological immune homeostasis
Deletion of the autism-associated protein SHANK3 abolishes structural synaptic plasticity after brain trauma
Loss of COMMD1 and copper overload disrupt zinc homeostasis and influence an autism-associated pathway at glutamatergic synapses
Activity regulating signals in neurons associated with energy homeostasis
Immune dysregulation associated with co-occurring germline CBL and SH2B3 variants
Dissecting hormonal and transient effects in Shankopathies associated with autism spectrum disorder
Hyperglycaemia is associated with male infertility and activin dysregulation in Type 1 Diabetes
Structure-Based Discovery of Mouse Trace Amine-Associated Receptor 5 Antagonists
Human trace amine-associated receptor TAAR5 can be activated by trimethylamine
Characterization of the structural and functional impact of autism-associated SHANK3 missense mutations
Investigating the impact of autism-associated SHANK2 genetic defects on early neuronal differentiation
Interferon-driven immune dysregulation in common variable immunodeficiency-associated villous atrophy and norovirus infection
GPCR-specific autoantibody signatures are associated with physiological and pathological immune homeostasis
Deletion of the autism-associated protein SHANK3 abolishes structural synaptic plasticity after brain trauma
Loss of COMMD1 and copper overload disrupt zinc homeostasis and influence an autism-associated pathway at glutamatergic synapses
Activity regulating signals in neurons associated with energy homeostasis
Immune dysregulation associated with co-occurring germline CBL and SH2B3 variants
Dissecting hormonal and transient effects in Shankopathies associated with autism spectrum disorder
Hyperglycaemia is associated with male infertility and activin dysregulation in Type 1 Diabetes
Structure-Based Discovery of Mouse Trace Amine-Associated Receptor 5 Antagonists
Human trace amine-associated receptor TAAR5 can be activated by trimethylamine
Characterization of the structural and functional impact of autism-associated SHANK3 missense mutations
Investigating the impact of autism-associated SHANK2 genetic defects on early neuronal differentiation
Interferon-driven immune dysregulation in common variable immunodeficiency-associated villous atrophy and norovirus infection
GPCR-specific autoantibody signatures are associated with physiological and pathological immune homeostasis