Gene expression and functional deficits underlie TREM2-knockout microglia responses in human models of Alzheimer’s disease
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2041-1723
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Gene expression and functional deficits underlie TREM2-knockout microglia responses in human models of Alzheimer’s disease ; volume:11 ; number:1 ; day:23 ; month:10 ; year:2020 ; pages:1-17 ; date:12.2020
Nature Communications ; 11, Heft 1 (23.10.2020), 1-17, 12.2020
- Creator
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McQuade, Amanda
Kang, You Jung
Hasselmann, Jonathan
Jairaman, Amit
Sotelo, Alexandra
Coburn, Morgan
Shabestari, Sepideh Kiani
Chadarevian, Jean Paul
Fote, Gianna
Tu, Christina H.
Danhash, Emma
Silva, Jorge
Martinez, Eric
Cotman, Carl
Prieto, G. Aleph
Thompson, Leslie M.
Steffan, Joan S.
Smith, Ian
Davtyan, Hayk
Cahalan, Michael
Cho, Hansang
Blurton-Jones, Mathew
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41467-020-19227-5
- URN
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urn:nbn:de:101:1-2020120419542019083627
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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24.11.2028, 12:57 AM CET
Data provider
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Associated
- McQuade, Amanda
- Kang, You Jung
- Hasselmann, Jonathan
- Jairaman, Amit
- Sotelo, Alexandra
- Coburn, Morgan
- Shabestari, Sepideh Kiani
- Chadarevian, Jean Paul
- Fote, Gianna
- Tu, Christina H.
- Danhash, Emma
- Silva, Jorge
- Martinez, Eric
- Cotman, Carl
- Prieto, G. Aleph
- Thompson, Leslie M.
- Steffan, Joan S.
- Smith, Ian
- Davtyan, Hayk
- Cahalan, Michael
- Cho, Hansang
- Blurton-Jones, Mathew
- SpringerLink (Online service)
Other Objects (12)
Author Correction: Gene expression and functional deficits underlie TREM2-knockout microglia responses in human models of Alzheimer’s disease
TREM2 dependent and independent functions of microglia in Alzheimer’s disease
TREM2 regulates innate immunity in Alzheimer’s disease
Soluble TREM2 ameliorates tau phosphorylation and cognitive deficits through activating transgelin-2 in Alzheimer’s disease
TREM2 gene expression associations with Alzheimer’s disease neuropathology are region-specific: implications for cortical versus subcortical microglia
Cerebrospinal fluid soluble TREM2 in aging and Alzheimer’s disease
Microglia‐synapse engulfment via PtdSer‐TREM2 ameliorates neuronal hyperactivity in Alzheimer's disease models
New insights into the role of TREM2 in Alzheimer’s disease
Intermittent hypoxia training enhances Aβ endocytosis by plaque associated microglia via VPS35-dependent TREM2 recycling in murine Alzheimer’s disease
Occupational-like organophosphate exposure disrupts microglia and accelerates deficits in a rat model of Alzheimer’s disease
TREM2 upregulation correlates with 5-hydroxymethycytosine enrichment in Alzheimer’s disease hippocampus
Pinpointing Brain TREM2 Levels in Two Mouse Models of Alzheimer’s Disease
Author Correction: Gene expression and functional deficits underlie TREM2-knockout microglia responses in human models of Alzheimer’s disease
TREM2 dependent and independent functions of microglia in Alzheimer’s disease
TREM2 regulates innate immunity in Alzheimer’s disease
Soluble TREM2 ameliorates tau phosphorylation and cognitive deficits through activating transgelin-2 in Alzheimer’s disease
TREM2 gene expression associations with Alzheimer’s disease neuropathology are region-specific: implications for cortical versus subcortical microglia
Cerebrospinal fluid soluble TREM2 in aging and Alzheimer’s disease
Microglia‐synapse engulfment via PtdSer‐TREM2 ameliorates neuronal hyperactivity in Alzheimer's disease models
New insights into the role of TREM2 in Alzheimer’s disease
Intermittent hypoxia training enhances Aβ endocytosis by plaque associated microglia via VPS35-dependent TREM2 recycling in murine Alzheimer’s disease
Occupational-like organophosphate exposure disrupts microglia and accelerates deficits in a rat model of Alzheimer’s disease
TREM2 upregulation correlates with 5-hydroxymethycytosine enrichment in Alzheimer’s disease hippocampus
Pinpointing Brain TREM2 Levels in Two Mouse Models of Alzheimer’s Disease
Author Correction: Gene expression and functional deficits underlie TREM2-knockout microglia responses in human models of Alzheimer’s disease
TREM2 dependent and independent functions of microglia in Alzheimer’s disease
TREM2 regulates innate immunity in Alzheimer’s disease
Soluble TREM2 ameliorates tau phosphorylation and cognitive deficits through activating transgelin-2 in Alzheimer’s disease
TREM2 gene expression associations with Alzheimer’s disease neuropathology are region-specific: implications for cortical versus subcortical microglia
Cerebrospinal fluid soluble TREM2 in aging and Alzheimer’s disease
Microglia‐synapse engulfment via PtdSer‐TREM2 ameliorates neuronal hyperactivity in Alzheimer's disease models
New insights into the role of TREM2 in Alzheimer’s disease
Intermittent hypoxia training enhances Aβ endocytosis by plaque associated microglia via VPS35-dependent TREM2 recycling in murine Alzheimer’s disease
Occupational-like organophosphate exposure disrupts microglia and accelerates deficits in a rat model of Alzheimer’s disease
TREM2 upregulation correlates with 5-hydroxymethycytosine enrichment in Alzheimer’s disease hippocampus