Gene expression and functional deficits underlie TREM2-knockout microglia responses in human models of Alzheimer’s disease
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2041-1723
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
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online resource.
- Erschienen in
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Gene expression and functional deficits underlie TREM2-knockout microglia responses in human models of Alzheimer’s disease ; volume:11 ; number:1 ; day:23 ; month:10 ; year:2020 ; pages:1-17 ; date:12.2020
Nature Communications ; 11, Heft 1 (23.10.2020), 1-17, 12.2020
- Urheber
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McQuade, Amanda
Kang, You Jung
Hasselmann, Jonathan
Jairaman, Amit
Sotelo, Alexandra
Coburn, Morgan
Shabestari, Sepideh Kiani
Chadarevian, Jean Paul
Fote, Gianna
Tu, Christina H.
Danhash, Emma
Silva, Jorge
Martinez, Eric
Cotman, Carl
Prieto, G. Aleph
Thompson, Leslie M.
Steffan, Joan S.
Smith, Ian
Davtyan, Hayk
Cahalan, Michael
Cho, Hansang
Blurton-Jones, Mathew
- Beteiligte Personen und Organisationen
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SpringerLink (Online service)
- DOI
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10.1038/s41467-020-19227-5
- URN
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urn:nbn:de:101:1-2020120419542019083627
- Rechteinformation
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
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15.08.2025, 07:25 MESZ
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Beteiligte
- McQuade, Amanda
- Kang, You Jung
- Hasselmann, Jonathan
- Jairaman, Amit
- Sotelo, Alexandra
- Coburn, Morgan
- Shabestari, Sepideh Kiani
- Chadarevian, Jean Paul
- Fote, Gianna
- Tu, Christina H.
- Danhash, Emma
- Silva, Jorge
- Martinez, Eric
- Cotman, Carl
- Prieto, G. Aleph
- Thompson, Leslie M.
- Steffan, Joan S.
- Smith, Ian
- Davtyan, Hayk
- Cahalan, Michael
- Cho, Hansang
- Blurton-Jones, Mathew
- SpringerLink (Online service)
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TREM2 regulates innate immunity in Alzheimer’s disease
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Microglia‐synapse engulfment via PtdSer‐TREM2 ameliorates neuronal hyperactivity in Alzheimer's disease models
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Occupational-like organophosphate exposure disrupts microglia and accelerates deficits in a rat model of Alzheimer’s disease
TREM2 upregulation correlates with 5-hydroxymethycytosine enrichment in Alzheimer’s disease hippocampus