Potentiation of glycine-gated NR1/NR3A NMDA receptors relieves Ca2+-dependent outward rectification

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: In: Frontiers in molecular neuroscience, 3.2010, Nr. 6 ; doi:10.3389/fnmol.2010.00006

Keyword: Biopharmazie
Pharmakodynamik
Pharmakokinetik

Event: Veröffentlichung

(where): Frankfurt am Main

(who): Universitätsbibliothek Johann Christian Senckenberg

(when): 2010

Creator: Madry, Christian
Betz, Heinrich
Geiger, Jörg R. P.
Laube, Bodo

URN: urn:nbn:de:hebis:30-83861

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:33 AM CEST

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Madry, Christian
Betz, Heinrich
Geiger, Jörg R. P.
Laube, Bodo
Universitätsbibliothek Johann Christian Senckenberg

Time of origin

2010

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Potentiation of glycine-gated NR1/NR3A NMDA receptors relieves Ca2+-dependent outward rectification

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Other Objects (12)

NMDA Receptor Dependent Long-term Potentiation in Chronic Pain

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Relieves de Xochicalco.

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Rectification.

Rectification.

Rectification.

Rectification.

Rectification.

Rectification.

Rectification.

NMDA Receptor Dependent Long-term Potentiation in Chronic Pain

Outward rectification of voltage-gated K+ channels evolved at least twice in life history

Relieves de Xochicalco.

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NMDA Receptor Dependent Long-term Potentiation in Chronic Pain

Outward rectification of voltage-gated K+ channels evolved at least twice in life history

Relieves de Xochicalco.

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Rectification.

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Rectification.

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