MCRS1 overexpression, which is specifically inhibited by miR-129*, promotes the epithelial-mesenchymal transition and metastasis in non-small cell lung cancer

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1476-4598

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: MCRS1 overexpression, which is specifically inhibited by miR-129*, promotes the epithelial-mesenchymal transition and metastasis in non-small cell lung cancer ; volume:13 ; number:1 ; day:6 ; month:11 ; year:2014 ; pages:1-15 ; date:12.2014
Molecular cancer ; 13, Heft 1 (6.11.2014), 1-15, 12.2014

Creator: Liu, Min-Xia
Zhou, Ke-Cheng
Cao, Yi

Contributor: SpringerLink (Online service)

DOI: 10.1186/1476-4598-13-245

URN: urn:nbn:de:101:1-2021082319542066678801

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:51 AM CEST

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Liu, Min-Xia
Zhou, Ke-Cheng
Cao, Yi
SpringerLink (Online service)

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MCRS1 overexpression, which is specifically inhibited by miR-129*, promotes the epithelial-mesenchymal transition and metastasis in non-small cell lung cancer

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Other Objects (12)

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HK3 overexpression associated with epithelial-mesenchymal transition in colorectal cancer

Cell surface IL‐1α trafficking is specifically inhibited by interferon‐γ, and associates with the membrane via IL‐1R2 and GPI anchors

Apelin inhibited epithelial−mesenchymal transition of podocytes in diabetic mice through downregulating immunoproteasome subunits β5i

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ALKBH5 inhibited autophagy of epithelial ovarian cancer through miR-7 and BCL-2

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HK3 overexpression associated with epithelial-mesenchymal transition in colorectal cancer

Cell surface IL‐1α trafficking is specifically inhibited by interferon‐γ, and associates with the membrane via IL‐1R2 and GPI anchors

Apelin inhibited epithelial−mesenchymal transition of podocytes in diabetic mice through downregulating immunoproteasome subunits β5i

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Correction to: Thymoquinone inhibited vasculogenic capacity and promoted mesenchymal-epithelial transition of human breast cancer stem cells

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