IGFBP5 is released by senescent cells and is internalized by healthy cells, promoting their senescence through interaction with retinoic receptors
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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IGFBP5 is released by senescent cells and is internalized by healthy cells, promoting their senescence through interaction with retinoic receptors ; volume:22 ; number:1 ; day:13 ; month:2 ; year:2024 ; pages:1-17 ; date:12.2024
Cell communication and signaling ; 22, Heft 1 (13.2.2024), 1-17, 12.2024
- Creator
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Alessio, Nicola
Aprile, Domenico
Peluso, Gianfranco
Mazzone, Valeria
Patrone, Deanira
Bernardo, Giovanni Di
Galderisi, Umberto
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12964-024-01469-1
- URN
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urn:nbn:de:101:1-2405162123284.698148588933
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:55 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Alessio, Nicola
- Aprile, Domenico
- Peluso, Gianfranco
- Mazzone, Valeria
- Patrone, Deanira
- Bernardo, Giovanni Di
- Galderisi, Umberto
- SpringerLink (Online service)
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Single senescent cell sequencing reveals heterogeneity in senescent cells induced by telomere erosion
The efficacy of chemotherapy is limited by intratumoral senescent cells expressing PD-L2
Apoptosis resistance of senescent cells is an intrinsic barrier for senolysis induced by cardiac glycosides
Metformin inhibition of neuroblastoma cell proliferation is differently modulated by cell differentiation induced by retinoic acid or overexpression of NDM29 non-coding RNA
The senescent mesothelial matrix accentuates colonization by ovarian cancer cells
The mTORC1-autophagy pathway is a target for senescent cell elimination
Senolytic Therapy Enabled by Senescent Cell‐Sensitive Biomimetic Melanin Nano‐Senolytics
Biogenesis of Pro-senescent Microparticles by Endothelial Colony Forming Cells from Premature Neonates is driven by SIRT1-Dependent Epigenetic Regulation of MKK6
The angiogenic factor midkine is regulated by dexamethasone and retinoic acid during alveolarization and in alveolar epithelial cells
Targeting senescent cells with NKG2D-CAR T cells
Aneuploid senescent cells activate NF‐κB to promote their immune clearance by NK cells
Rejuvenation of Senescent Bone Marrow Mesenchymal Stromal Cells by Pulsed Triboelectric Stimulation
Single senescent cell sequencing reveals heterogeneity in senescent cells induced by telomere erosion
The efficacy of chemotherapy is limited by intratumoral senescent cells expressing PD-L2
Apoptosis resistance of senescent cells is an intrinsic barrier for senolysis induced by cardiac glycosides
Metformin inhibition of neuroblastoma cell proliferation is differently modulated by cell differentiation induced by retinoic acid or overexpression of NDM29 non-coding RNA
The senescent mesothelial matrix accentuates colonization by ovarian cancer cells
The mTORC1-autophagy pathway is a target for senescent cell elimination
Senolytic Therapy Enabled by Senescent Cell‐Sensitive Biomimetic Melanin Nano‐Senolytics
Biogenesis of Pro-senescent Microparticles by Endothelial Colony Forming Cells from Premature Neonates is driven by SIRT1-Dependent Epigenetic Regulation of MKK6
The angiogenic factor midkine is regulated by dexamethasone and retinoic acid during alveolarization and in alveolar epithelial cells
Targeting senescent cells with NKG2D-CAR T cells
Aneuploid senescent cells activate NF‐κB to promote their immune clearance by NK cells