Nucleolar stress in C9orf72 and sporadic ALS spinal motor neurons precedes TDP-43 mislocalization

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2051-5960

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Nucleolar stress in C9orf72 and sporadic ALS spinal motor neurons precedes TDP-43 mislocalization ; volume:9 ; number:1 ; day:15 ; month:2 ; year:2021 ; pages:1-16 ; date:12.2021
Acta Neuropathologica Communications ; 9, Heft 1 (15.2.2021), 1-16, 12.2021

Creator: Aladesuyi Arogundade, Olubankole
Nguyen, Sandra
Leung, Ringo
Wainio, Danielle
Rodriguez, Maria
Ravits, John

Contributor: SpringerLink (Online service)

DOI: 10.1186/s40478-021-01125-6

URN: urn:nbn:de:101:1-2021040500185556465739

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 11:03 AM CEST

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Aladesuyi Arogundade, Olubankole
Nguyen, Sandra
Leung, Ringo
Wainio, Danielle
Rodriguez, Maria
Ravits, John
SpringerLink (Online service)

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Cytoskeletal rearrangement precedes nucleolar remodeling during adipogenesis

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Nucleolar stress in C9orf72 and sporadic ALS spinal motor neurons precedes TDP-43 mislocalization

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Other Objects (12)

Bidirectional nucleolar dysfunction in C9orf72 frontotemporal lobar degeneration

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Phosphorylated TDP-43 (pTDP-43) aggregates in the axial skeletal muscle of patients with sporadic and familial amyotrophic lateral sclerosis

Poly-GA triggers TDP-43 pathology by inhibiting the proteasome and nucleocytoplasmic import in C9orf72 ALS/FTD

TDP-43 in sporadic ALS : pathological changes of an RNA-binding protein and consequences on RNA metabolism = TDP-43 in der sporadischen ALS

Heterogeneity of cerebral TDP-43 pathology in sporadic amyotrophic lateral sclerosis: Evidence for clinico-pathologic subtypes

Serum total TDP-43 levels are decreased in frontotemporal dementia patients with C9orf72 repeat expansion or concomitant motoneuron disease phenotype

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Phosphorylated TDP-43 (pTDP-43) aggregates in the axial skeletal muscle of patients with sporadic and familial amyotrophic lateral sclerosis

Poly-GA triggers TDP-43 pathology by inhibiting the proteasome and nucleocytoplasmic import in C9orf72 ALS/FTD

TDP-43 in sporadic ALS : pathological changes of an RNA-binding protein and consequences on RNA metabolism = TDP-43 in der sporadischen ALS

Heterogeneity of cerebral TDP-43 pathology in sporadic amyotrophic lateral sclerosis: Evidence for clinico-pathologic subtypes

Serum total TDP-43 levels are decreased in frontotemporal dementia patients with C9orf72 repeat expansion or concomitant motoneuron disease phenotype

A robust evaluation of TDP-43, poly GP, cellular pathology and behavior in an AAV-C9ORF72 (G4C2)66 mouse model

Nucleolar epigenomics by dynamic molecular combing and nucleolar matrix analysis

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Bidirectional nucleolar dysfunction in C9orf72 frontotemporal lobar degeneration

Cytoskeletal rearrangement precedes nucleolar remodeling during adipogenesis

Aberrant deposition of stress granule-resident proteins linked to C9orf72-associated TDP-43 proteinopathy

Phosphorylated TDP-43 (pTDP-43) aggregates in the axial skeletal muscle of patients with sporadic and familial amyotrophic lateral sclerosis

Poly-GA triggers TDP-43 pathology by inhibiting the proteasome and nucleocytoplasmic import in C9orf72 ALS/FTD

TDP-43 in sporadic ALS : pathological changes of an RNA-binding protein and consequences on RNA metabolism = TDP-43 in der sporadischen ALS

Heterogeneity of cerebral TDP-43 pathology in sporadic amyotrophic lateral sclerosis: Evidence for clinico-pathologic subtypes

Serum total TDP-43 levels are decreased in frontotemporal dementia patients with C9orf72 repeat expansion or concomitant motoneuron disease phenotype

A robust evaluation of TDP-43, poly GP, cellular pathology and behavior in an AAV-C9ORF72 (G4C2)66 mouse model

Nucleolar epigenomics by dynamic molecular combing and nucleolar matrix analysis

Nucleolar epigenomics by dynamic molecular combing and nucleolar matrix analysis

Tau and TDP-43 synergy: a novel therapeutic target for sporadic late-onset Alzheimer’s disease

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