SOX17 restrains proliferation and tumor formation by down-regulating activity of the Wnt/β-catenin signaling pathway via trans-suppressing β-catenin in cervical cancer

zu Verbundenen Objekten

Standort
Deutsche Nationalbibliothek Frankfurt am Main
ISSN
2041-4889
Umfang
Online-Ressource
Sprache
Englisch
Anmerkungen
online resource.

Erschienen in
SOX17 restrains proliferation and tumor formation by down-regulating activity of the Wnt/β-catenin signaling pathway via trans-suppressing β-catenin in cervical cancer ; volume:9 ; number:7 ; day:3 ; month:7 ; year:2018 ; pages:1-16 ; date:7.2018
Cell death & disease ; 9, Heft 7 (3.7.2018), 1-16, 7.2018

Urheber
Li, Lu
Beteiligte Personen und Organisationen
Yang, Wen-Ting
Zheng, Peng-Sheng
Liu, Xiao-Fang
SpringerLink (Online service)

DOI
10.1038/s41419-018-0782-8
URN
urn:nbn:de:101:1-2018090400121339653511
Rechteinformation
Der Zugriff auf das Objekt ist unbeschränkt möglich.
Letzte Aktualisierung
15.08.2025, 07:29 MESZ

Datenpartner

Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Original beim Datenpartner anzeigen

Beteiligte

  • Li, Lu
  • Yang, Wen-Ting
  • Zheng, Peng-Sheng
  • Liu, Xiao-Fang
  • SpringerLink (Online service)

Ähnliche Objekte (12)

β-Catenin [Beta-Catenin] in der kolorektalen Karzinogenese

Hochschulschrift

β-Catenin [Beta-Catenin] in der kolorektalen Karzinogenese

β-Catenin [Beta-Catenin] in der kolorektalen Karzinogenese

Hochschulschrift

β-Catenin [Beta-Catenin] in der kolorektalen Karzinogenese

KCTD9 inhibits the Wnt/β-catenin pathway by decreasing the level of β-catenin in colorectal cancer

KCTD9 inhibits the Wnt/β-catenin pathway by decreasing the level of β-catenin in colorectal cancer

CREPT facilitates colorectal cancer growth through inducing Wnt/β-catenin pathway by enhancing p300-mediated β-catenin acetylation

CREPT facilitates colorectal cancer growth through inducing Wnt/β-catenin pathway by enhancing p300-mediated β-catenin acetylation

Comprehensive analysis of β-catenin target genes in colorectal carcinoma cell lines with deregulated Wnt/β-catenin signaling

Comprehensive analysis of β-catenin target genes in colorectal carcinoma cell lines with deregulated Wnt/β-catenin signaling

Interaction between Wnt/β-catenin and RAS-ERK pathways and an anti-cancer strategy via degradations of β-catenin and RAS by targeting the Wnt/β-catenin pathway

Interaction between Wnt/β-catenin and RAS-ERK pathways and an anti-cancer strategy via degradations of β-catenin and RAS by targeting the Wnt/β-catenin pathway

SPAG5 promotes hepatocellular carcinoma progression by downregulating SCARA5 through modifying β-catenin degradation

SPAG5 promotes hepatocellular carcinoma progression by downregulating SCARA5 through modifying β-catenin degradation

Pathogenetic Analysis of Sinonasal Teratocarcinosarcomas Reveal Actionable β-catenin Overexpression and a β-catenin Mutation

Pathogenetic Analysis of Sinonasal Teratocarcinosarcomas Reveal Actionable β-catenin Overexpression and a β-catenin Mutation

Interaktionspartner des Tumorsuppressorproteins APC : Tubulin, β-Catenin [Beta-Catenin] und DNA

Hochschulschrift

Interaktionspartner des Tumorsuppressorproteins APC : Tubulin, β-Catenin [Beta-Catenin] und DNA

Kerntransport des Signalmoleküls β-Catenin [Beta-Catenin] in der Wnt-Transduktionskaskade

Hochschulschrift

Kerntransport des Signalmoleküls β-Catenin [Beta-Catenin] in der Wnt-Transduktionskaskade

Aktivierung von β-Catenin [Beta-Catenin] durch Genmutation in kultivierten Hepatomzellen

Hochschulschrift

Aktivierung von β-Catenin [Beta-Catenin] durch Genmutation in kultivierten Hepatomzellen

Cepharanthine suppresses APC-mutant colorectal cancers by down-regulating the expression of β-catenin

Cepharanthine suppresses APC-mutant colorectal cancers by down-regulating the expression of β-catenin

Verbundene Objekte