Genomic retargeting of p53 and CTCF is associated with transcriptional changes during oncogenic HRas-induced transformation
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2399-3642
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Genomic retargeting of p53 and CTCF is associated with transcriptional changes during oncogenic HRas-induced transformation ; volume:3 ; number:1 ; day:25 ; month:11 ; year:2020 ; pages:1-13 ; date:12.2020
Communications biology ; 3, Heft 1 (25.11.2020), 1-13, 12.2020
- Creator
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Schwartz, Michal
Portugez, Avital Sarusi
Attia, Bracha Zukerman
Tannenbaum, Miriam
Cohen, Leslie
Loza, Olga
Chase, Emily
Turman, Yousef
Kaplan, Tommy
Salah, Zaidoun
Hakim, Ofir
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s42003-020-01398-y
- URN
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urn:nbn:de:101:1-2020123020311016159199
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
26.01.2025, 11:21 AM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Schwartz, Michal
- Portugez, Avital Sarusi
- Attia, Bracha Zukerman
- Tannenbaum, Miriam
- Cohen, Leslie
- Loza, Olga
- Chase, Emily
- Turman, Yousef
- Kaplan, Tommy
- Salah, Zaidoun
- Hakim, Ofir
- SpringerLink (Online service)
Other Objects (12)
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BORIS/CTCFL epigenetically reprograms clustered CTCF binding sites into alternative transcriptional start sites
Oncogenic c-Myc induces replication stress by increasing cohesins chromatin occupancy in a CTCF-dependent manner
Defining the relative and combined contribution of CTCF and CTCFL to genomic regulation
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Constitutive Photomorphogensis Protein1 (COP1) mediated p53 pathway and its oncogenic role
Oncogenic p53 triggers amyloid aggregation of p63 and p73 liquid droplets
Id4 dependent acetylation restores mutant-p53 transcriptional activity
Cancer-specific CTCF binding facilitates oncogenic transcriptional dysregulation
Predicting CTCF-mediated chromatin interactions by integrating genomic and epigenomic features
CTCF binding site classes exhibit distinct evolutionary, genomic, epigenomic and transcriptomic features
Auxin-inducible degron 2 system deciphers functions of CTCF domains in transcriptional regulation
BORIS/CTCFL epigenetically reprograms clustered CTCF binding sites into alternative transcriptional start sites
Oncogenic c-Myc induces replication stress by increasing cohesins chromatin occupancy in a CTCF-dependent manner
Defining the relative and combined contribution of CTCF and CTCFL to genomic regulation
CTCF and EGR1 suppress breast cancer cell migration through transcriptional control of Nm23-H1
CTCF and cellular heterogeneity
Constitutive Photomorphogensis Protein1 (COP1) mediated p53 pathway and its oncogenic role
Oncogenic p53 triggers amyloid aggregation of p63 and p73 liquid droplets
Id4 dependent acetylation restores mutant-p53 transcriptional activity
Cancer-specific CTCF binding facilitates oncogenic transcriptional dysregulation
Predicting CTCF-mediated chromatin interactions by integrating genomic and epigenomic features
CTCF binding site classes exhibit distinct evolutionary, genomic, epigenomic and transcriptomic features
Auxin-inducible degron 2 system deciphers functions of CTCF domains in transcriptional regulation
BORIS/CTCFL epigenetically reprograms clustered CTCF binding sites into alternative transcriptional start sites
Oncogenic c-Myc induces replication stress by increasing cohesins chromatin occupancy in a CTCF-dependent manner
Defining the relative and combined contribution of CTCF and CTCFL to genomic regulation
CTCF and EGR1 suppress breast cancer cell migration through transcriptional control of Nm23-H1
CTCF and cellular heterogeneity
Constitutive Photomorphogensis Protein1 (COP1) mediated p53 pathway and its oncogenic role
Oncogenic p53 triggers amyloid aggregation of p63 and p73 liquid droplets