Epigenetic targeting of Mcl-1 Is synthetically lethal with Bcl-xL/Bcl-2 inhibition in model systems of glioblastoma
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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In: Cancers ; 12 (2020), 8. - Art.-Nr. 2137. - eISSN 2072-6694
- Keyword
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Krebs
Glioblastom
Apoptosis
- Event
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Veröffentlichung
- (where)
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Ulm
- (who)
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Universität Ulm
- (when)
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2020
- Creator
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Shang, Enyuan
Nguyen, Trang T. T.
Shu, Chang
Westhoff, Mike-Andrew
Karpel-Massler, Georg
Siegelin, Markus D.
- DOI
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10.18725/OPARU-44611
- URN
-
urn:nbn:de:bsz:289-oparu-44687-2
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:39 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Shang, Enyuan
- Nguyen, Trang T. T.
- Shu, Chang
- Westhoff, Mike-Andrew
- Karpel-Massler, Georg
- Siegelin, Markus D.
- Universität Ulm
Time of origin
- 2020
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The BCL-2 family in glioblastoma
Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms
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Selective Affimers Recognise the BCL‐2 Family Proteins BCL‐x L and MCL‐1 through Noncanonical Structural Motifs **
Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma
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