Metformin ameliorates scleroderma via inhibiting Th17 cells and reducing mTOR-STAT3 signaling in skin fibroblasts

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1479-5876

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Metformin ameliorates scleroderma via inhibiting Th17 cells and reducing mTOR-STAT3 signaling in skin fibroblasts ; volume:19 ; number:1 ; day:4 ; month:5 ; year:2021 ; pages:1-10 ; date:12.2021
Journal of translational medicine ; 19, Heft 1 (4.5.2021), 1-10, 12.2021

Creator: Moon, Jeonghyeon
Lee, Seon-yeong
Choi, Jeong Won
Lee, A Ram
Yoo, Jin Hee
Moon, Su-Jin
Park, Sung-Hwan
Cho, Mi-La

Contributor: SpringerLink (Online service)

DOI: 10.1186/s12967-021-02860-z

URN: urn:nbn:de:101:1-2021102818501906258091

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:21 AM CEST

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Associated

Moon, Jeonghyeon
Lee, Seon-yeong
Choi, Jeong Won
Lee, A Ram
Yoo, Jin Hee
Moon, Su-Jin
Park, Sung-Hwan
Cho, Mi-La
SpringerLink (Online service)

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Correction to: Metformin ameliorates scleroderma via inhibiting Th17 cells and reducing mTOR-STAT3 signaling in skin fibroblasts

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Hepatocyte growth factor ameliorates dermal sclerosis in the tight-skin mouse model of scleroderma

GRIM19 ameliorates acute graft-versus-host disease (GVHD) by modulating Th17 and Treg cell balance through down-regulation of STAT3 and NF-AT activation

Aging weakens Th17 cell pathogenicity and ameliorates experimental autoimmune uveitis in mice

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Treg17 cells are programmed by Stat3 to suppress Th17 responses in systemic lupus

Hectd3 promotes pathogenic Th17 lineage through Stat3 activation and Malt1 signaling in neuroinflammation

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ROCK2, but not ROCK1 interacts with phosphorylated STAT3 and co-occupies TH17/TFH gene promoters in TH17-activated human T cells

STAT3 blockade ameliorates LPS-induced kidney injury through macrophage-driven inflammation

Valproic acid ameliorates coxsackievirus-B3-induced viral myocarditis by modulating Th17/Treg imbalance

Correction to: Metformin ameliorates scleroderma via inhibiting Th17 cells and reducing mTOR-STAT3 signaling in skin fibroblasts

Metformin ameliorates valve interstitial cell calcification by promoting autophagic flux

Ssu72 attenuates autoimmune arthritis via targeting of STAT3 signaling and Th17 activation

Hepatocyte growth factor ameliorates dermal sclerosis in the tight-skin mouse model of scleroderma

GRIM19 ameliorates acute graft-versus-host disease (GVHD) by modulating Th17 and Treg cell balance through down-regulation of STAT3 and NF-AT activation

Aging weakens Th17 cell pathogenicity and ameliorates experimental autoimmune uveitis in mice

Hochschulschrift

Treg17 cells are programmed by Stat3 to suppress Th17 responses in systemic lupus

Hectd3 promotes pathogenic Th17 lineage through Stat3 activation and Malt1 signaling in neuroinflammation

Phototherapy in Scleroderma

ROCK2, but not ROCK1 interacts with phosphorylated STAT3 and co-occupies TH17/TFH gene promoters in TH17-activated human T cells

STAT3 blockade ameliorates LPS-induced kidney injury through macrophage-driven inflammation

Valproic acid ameliorates coxsackievirus-B3-induced viral myocarditis by modulating Th17/Treg imbalance

Correction to: Metformin ameliorates scleroderma via inhibiting Th17 cells and reducing mTOR-STAT3 signaling in skin fibroblasts

Metformin ameliorates valve interstitial cell calcification by promoting autophagic flux

Ssu72 attenuates autoimmune arthritis via targeting of STAT3 signaling and Th17 activation

Hepatocyte growth factor ameliorates dermal sclerosis in the tight-skin mouse model of scleroderma

GRIM19 ameliorates acute graft-versus-host disease (GVHD) by modulating Th17 and Treg cell balance through down-regulation of STAT3 and NF-AT activation

Aging weakens Th17 cell pathogenicity and ameliorates experimental autoimmune uveitis in mice

Hochschulschrift

Treg17 cells are programmed by Stat3 to suppress Th17 responses in systemic lupus

Hectd3 promotes pathogenic Th17 lineage through Stat3 activation and Malt1 signaling in neuroinflammation

Phototherapy in Scleroderma

ROCK2, but not ROCK1 interacts with phosphorylated STAT3 and co-occupies TH17/TFH gene promoters in TH17-activated human T cells

STAT3 blockade ameliorates LPS-induced kidney injury through macrophage-driven inflammation

Valproic acid ameliorates coxsackievirus-B3-induced viral myocarditis by modulating Th17/Treg imbalance

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Metformin ameliorates scleroderma via inhibiting Th17 cells and reducing mTOR-STAT3 signaling in skin fibroblasts

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Other Objects (12)

Correction to: Metformin ameliorates scleroderma via inhibiting Th17 cells and reducing mTOR-STAT3 signaling in skin fibroblasts

Metformin ameliorates valve interstitial cell calcification by promoting autophagic flux

Ssu72 attenuates autoimmune arthritis via targeting of STAT3 signaling and Th17 activation

Hepatocyte growth factor ameliorates dermal sclerosis in the tight-skin mouse model of scleroderma

GRIM19 ameliorates acute graft-versus-host disease (GVHD) by modulating Th17 and Treg cell balance through down-regulation of STAT3 and NF-AT activation

Aging weakens Th17 cell pathogenicity and ameliorates experimental autoimmune uveitis in mice

Treg17 cells are programmed by Stat3 to suppress Th17 responses in systemic lupus

Hectd3 promotes pathogenic Th17 lineage through Stat3 activation and Malt1 signaling in neuroinflammation

Phototherapy in Scleroderma

ROCK2, but not ROCK1 interacts with phosphorylated STAT3 and co-occupies TH17/TFH gene promoters in TH17-activated human T cells

STAT3 blockade ameliorates LPS-induced kidney injury through macrophage-driven inflammation

Valproic acid ameliorates coxsackievirus-B3-induced viral myocarditis by modulating Th17/Treg imbalance

Correction to: Metformin ameliorates scleroderma via inhibiting Th17 cells and reducing mTOR-STAT3 signaling in skin fibroblasts

Metformin ameliorates valve interstitial cell calcification by promoting autophagic flux

Ssu72 attenuates autoimmune arthritis via targeting of STAT3 signaling and Th17 activation

Hepatocyte growth factor ameliorates dermal sclerosis in the tight-skin mouse model of scleroderma

GRIM19 ameliorates acute graft-versus-host disease (GVHD) by modulating Th17 and Treg cell balance through down-regulation of STAT3 and NF-AT activation

Aging weakens Th17 cell pathogenicity and ameliorates experimental autoimmune uveitis in mice

Treg17 cells are programmed by Stat3 to suppress Th17 responses in systemic lupus

Hectd3 promotes pathogenic Th17 lineage through Stat3 activation and Malt1 signaling in neuroinflammation

Phototherapy in Scleroderma

ROCK2, but not ROCK1 interacts with phosphorylated STAT3 and co-occupies TH17/TFH gene promoters in TH17-activated human T cells

STAT3 blockade ameliorates LPS-induced kidney injury through macrophage-driven inflammation

Valproic acid ameliorates coxsackievirus-B3-induced viral myocarditis by modulating Th17/Treg imbalance

Correction to: Metformin ameliorates scleroderma via inhibiting Th17 cells and reducing mTOR-STAT3 signaling in skin fibroblasts

Metformin ameliorates valve interstitial cell calcification by promoting autophagic flux

Ssu72 attenuates autoimmune arthritis via targeting of STAT3 signaling and Th17 activation

Hepatocyte growth factor ameliorates dermal sclerosis in the tight-skin mouse model of scleroderma

GRIM19 ameliorates acute graft-versus-host disease (GVHD) by modulating Th17 and Treg cell balance through down-regulation of STAT3 and NF-AT activation

Aging weakens Th17 cell pathogenicity and ameliorates experimental autoimmune uveitis in mice

Treg17 cells are programmed by Stat3 to suppress Th17 responses in systemic lupus

Hectd3 promotes pathogenic Th17 lineage through Stat3 activation and Malt1 signaling in neuroinflammation

Phototherapy in Scleroderma

ROCK2, but not ROCK1 interacts with phosphorylated STAT3 and co-occupies TH17/TFH gene promoters in TH17-activated human T cells

STAT3 blockade ameliorates LPS-induced kidney injury through macrophage-driven inflammation

Valproic acid ameliorates coxsackievirus-B3-induced viral myocarditis by modulating Th17/Treg imbalance

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