MiR-150-5p contributes to unexplained recurrent spontaneous abortion by targeting VEGFA and downregulating the PI3K/AKT/mTOR signaling pathway
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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MiR-150-5p contributes to unexplained recurrent spontaneous abortion by targeting VEGFA and downregulating the PI3K/AKT/mTOR signaling pathway ; volume:41 ; number:1 ; day:3 ; month:11 ; year:2023 ; pages:63-77 ; date:1.2024
Journal of assisted reproduction and genetics ; 41, Heft 1 (3.11.2023), 63-77, 1.2024
- Creator
-
Liao, Wenyan
Deng, Xin
Chen, Guodong
Yang, Juanli
Li, Yi
Li, Li
Zhong, Lili
Tao, Guangwei
Hou, Jiafeng
Li, Mujun
Ding, Chengming
- Contributor
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SpringerLink (Online service)
- DOI
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10.1007/s10815-023-02959-w
- URN
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urn:nbn:de:101:1-2024032721071167405260
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:46 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Liao, Wenyan
- Deng, Xin
- Chen, Guodong
- Yang, Juanli
- Li, Yi
- Li, Li
- Zhong, Lili
- Tao, Guangwei
- Hou, Jiafeng
- Li, Mujun
- Ding, Chengming
- SpringerLink (Online service)
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Knockdown of TFRC suppressed the progression of nasopharyngeal carcinoma by downregulating the PI3K/Akt/mTOR pathway
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Analysis of gene expression difference and biological process in chorionic villi of unexplained recurrent spontaneous abortion
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Cyanidin-3-glucoside suppresses the progression of lung adenocarcinoma by downregulating TP53I3 and inhibiting PI3K/AKT/mTOR pathway
MAX deficiency impairs human endometrial decidualization through down-regulating OSR2 in women with recurrent spontaneous abortion
Activation of SGK1/ENaC Signaling Pathway Improves the Level of Decidualization in Unexplained Recurrent Spontaneous Abortion
KLF4 regulates trophoblast function and associates with unexplained recurrent spontaneous abortion
miR-6086 inhibits ovarian cancer angiogenesis by downregulating the OC2/VEGFA/EGFL6 axis
AMPK-mTOR pathway modulates glycolysis reprogramming in unexplained recurrent spontaneous abortion
A missense variant in NCF1 is associated with susceptibility to unexplained recurrent spontaneous abortion
Knockdown of TFRC suppressed the progression of nasopharyngeal carcinoma by downregulating the PI3K/Akt/mTOR pathway
Bargaining unexplained
LncRNA landscape and associated ceRNA network in placental villus of unexplained recurrent spontaneous abortion
Analysis of gene expression difference and biological process in chorionic villi of unexplained recurrent spontaneous abortion
Morphine promotes non-small cell lung cancer progression by downregulating E-cadherin via the PI3K/AKT/mTOR pathway
Cyanidin-3-glucoside suppresses the progression of lung adenocarcinoma by downregulating TP53I3 and inhibiting PI3K/AKT/mTOR pathway
MAX deficiency impairs human endometrial decidualization through down-regulating OSR2 in women with recurrent spontaneous abortion
Activation of SGK1/ENaC Signaling Pathway Improves the Level of Decidualization in Unexplained Recurrent Spontaneous Abortion
KLF4 regulates trophoblast function and associates with unexplained recurrent spontaneous abortion
miR-6086 inhibits ovarian cancer angiogenesis by downregulating the OC2/VEGFA/EGFL6 axis
AMPK-mTOR pathway modulates glycolysis reprogramming in unexplained recurrent spontaneous abortion
A missense variant in NCF1 is associated with susceptibility to unexplained recurrent spontaneous abortion
Knockdown of TFRC suppressed the progression of nasopharyngeal carcinoma by downregulating the PI3K/Akt/mTOR pathway
Bargaining unexplained
LncRNA landscape and associated ceRNA network in placental villus of unexplained recurrent spontaneous abortion
Analysis of gene expression difference and biological process in chorionic villi of unexplained recurrent spontaneous abortion
Morphine promotes non-small cell lung cancer progression by downregulating E-cadherin via the PI3K/AKT/mTOR pathway
Cyanidin-3-glucoside suppresses the progression of lung adenocarcinoma by downregulating TP53I3 and inhibiting PI3K/AKT/mTOR pathway
MAX deficiency impairs human endometrial decidualization through down-regulating OSR2 in women with recurrent spontaneous abortion