Fascin-1 expression is associated with neuroendocrine prostate cancer and directly suppressed by androgen receptor
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
-
Fascin-1 expression is associated with neuroendocrine prostate cancer and directly suppressed by androgen receptor ; volume:129 ; number:12 ; day:24 ; month:10 ; year:2023 ; pages:1903-1914 ; date:12.2023
British journal of cancer ; 129, Heft 12 (24.10.2023), 1903-1914, 12.2023
- Creator
-
Turpin, Anthony
Delliaux, Carine
Parent, Pauline
Chevalier, Hortense
Escudero-Iriarte, Carmen
Bonardi, Franck
Vanpouille, Nathalie
Flourens, Anne
Querol, Jessica
Carnot, Aurélien
Leroy, Xavier
Herranz, Nicolás
Lanel, Tristan
Villers, Arnauld
Olivier, Jonathan
Touzet, Hélène
Launoit, Yvan de
Tian, Tian V.
Duterque-Coquillaud, Martine
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s41416-023-02449-x
- URN
-
urn:nbn:de:101:1-2024022308102255897445
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 11:02 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Turpin, Anthony
- Delliaux, Carine
- Parent, Pauline
- Chevalier, Hortense
- Escudero-Iriarte, Carmen
- Bonardi, Franck
- Vanpouille, Nathalie
- Flourens, Anne
- Querol, Jessica
- Carnot, Aurélien
- Leroy, Xavier
- Herranz, Nicolás
- Lanel, Tristan
- Villers, Arnauld
- Olivier, Jonathan
- Touzet, Hélène
- Launoit, Yvan de
- Tian, Tian V.
- Duterque-Coquillaud, Martine
- SpringerLink (Online service)
Other Objects (12)
Androgen deprivation restores ARHGEF2 to promote neuroendocrine differentiation of prostate cancer
Correction: Androgen deprivation restores ARHGEF2 to promote neuroendocrine differentiation of prostate cancer
AIRE promotes androgen-independent prostate cancer by directly regulating IL-6 and modulating tumor microenvironment
Loss of stromal androgen receptor leads to suppressed prostate tumourigenesis via modulation of pro‐inflammatory cytokines/chemokines
Androgen deprivation promotes neuroendocrine differentiation and angiogenesis through CREB-EZH2-TSP1 pathway in prostate cancers
Androgen deprivation induces neuroendocrine phenotypes in prostate cancer cells through CREB1/EZH2-mediated downregulation of REST
Hedgehog/Gli supports androgen signaling in androgen deprived and androgen independent prostate cancer cells
TRIM50 suppressed hepatocarcinoma progression through directly targeting SNAIL for ubiquitous degradation
Androgen receptor profiling predicts prostate cancer outcome
Duration of androgen deprivation therapy with maximum androgen blockade for localized prostate cancer
Suppressed androgen receptor expression promotes M2 macrophage reprogramming through the STAT3/SOCS3 pathway
Osteoclasts directly influence castration-resistant prostate cancer cells
Androgen deprivation restores ARHGEF2 to promote neuroendocrine differentiation of prostate cancer
Correction: Androgen deprivation restores ARHGEF2 to promote neuroendocrine differentiation of prostate cancer
AIRE promotes androgen-independent prostate cancer by directly regulating IL-6 and modulating tumor microenvironment
Loss of stromal androgen receptor leads to suppressed prostate tumourigenesis via modulation of pro‐inflammatory cytokines/chemokines
Androgen deprivation promotes neuroendocrine differentiation and angiogenesis through CREB-EZH2-TSP1 pathway in prostate cancers
Androgen deprivation induces neuroendocrine phenotypes in prostate cancer cells through CREB1/EZH2-mediated downregulation of REST
Hedgehog/Gli supports androgen signaling in androgen deprived and androgen independent prostate cancer cells
TRIM50 suppressed hepatocarcinoma progression through directly targeting SNAIL for ubiquitous degradation
Androgen receptor profiling predicts prostate cancer outcome
Duration of androgen deprivation therapy with maximum androgen blockade for localized prostate cancer
Suppressed androgen receptor expression promotes M2 macrophage reprogramming through the STAT3/SOCS3 pathway
Osteoclasts directly influence castration-resistant prostate cancer cells
Androgen deprivation restores ARHGEF2 to promote neuroendocrine differentiation of prostate cancer
Correction: Androgen deprivation restores ARHGEF2 to promote neuroendocrine differentiation of prostate cancer
AIRE promotes androgen-independent prostate cancer by directly regulating IL-6 and modulating tumor microenvironment
Loss of stromal androgen receptor leads to suppressed prostate tumourigenesis via modulation of pro‐inflammatory cytokines/chemokines
Androgen deprivation promotes neuroendocrine differentiation and angiogenesis through CREB-EZH2-TSP1 pathway in prostate cancers
Androgen deprivation induces neuroendocrine phenotypes in prostate cancer cells through CREB1/EZH2-mediated downregulation of REST
Hedgehog/Gli supports androgen signaling in androgen deprived and androgen independent prostate cancer cells
TRIM50 suppressed hepatocarcinoma progression through directly targeting SNAIL for ubiquitous degradation
Androgen receptor profiling predicts prostate cancer outcome
Duration of androgen deprivation therapy with maximum androgen blockade for localized prostate cancer
Suppressed androgen receptor expression promotes M2 macrophage reprogramming through the STAT3/SOCS3 pathway