The combination of serum insulin, osteopontin, and hepatocyte growth factor predicts time to castration-resistant progression in androgen dependent metastatic prostate cancer- an exploratory study
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2407
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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The combination of serum insulin, osteopontin, and hepatocyte growth factor predicts time to castration-resistant progression in androgen dependent metastatic prostate cancer- an exploratory study ; volume:16 ; number:1 ; day:6 ; month:9 ; year:2016 ; pages:1-10 ; date:12.2016
BMC cancer ; 16, Heft 1 (6.9.2016), 1-10, 12.2016
- Creator
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Dayyani, Farshid
- Contributor
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Zurita, Amado J.
Nogueras-González, Graciela M.
Slack, Rebecca
Millikan, Randall E.
Araujo, John C.
Gallick, Gary E.
Logothetis, Christopher J.
Corn, Paul G.
SpringerLink (Online service)
- DOI
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10.1186/s12885-016-2723-1
- URN
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urn:nbn:de:1111-2016100616218
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.04.2250, 8:58 PM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Dayyani, Farshid
- Zurita, Amado J.
- Nogueras-González, Graciela M.
- Slack, Rebecca
- Millikan, Randall E.
- Araujo, John C.
- Gallick, Gary E.
- Logothetis, Christopher J.
- Corn, Paul G.
- SpringerLink (Online service)
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Clinical characteristics of patients with metastatic castration-resistant prostate cancer after treatment with combined androgen blockade
Patient-derived castration-resistant prostate cancer model revealed CTBP2 upregulation mediated by OCT1 and androgen receptor
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Combination of PARP Inhibitors and Androgen Receptor Pathway Inhibitors in Metastatic Castration-Resistant Prostate Cancer
Orphan nuclear receptor TLX contributes to androgen insensitivity in castration-resistant prostate cancer via its repression of androgen receptor transcription
Clinical characteristics of patients with metastatic castration-resistant prostate cancer after treatment with combined androgen blockade
Patient-derived castration-resistant prostate cancer model revealed CTBP2 upregulation mediated by OCT1 and androgen receptor
Targeting molecular resistance in castration-resistant prostate cancer
Evolving Personalized Therapy for Castration-Resistant Prostate Cancer
Osteoclasts directly influence castration-resistant prostate cancer cells
Bipolar androgen therapy plus nivolumab for patients with metastatic castration-resistant prostate cancer: the COMBAT phase II trial
The natural compound atraric acid suppresses androgen-regulated neo-angiogenesis of castration-resistant prostate cancer through angiopoietin 2
Effect of simvastatin on castration-resistant prostate cancer cells
Ureter metastatic castration-resistant prostate cancer: a case report
Analysis of regulation of angiogenesis by androgen receptor ligands in castration-resistant prostate cancer
Combination of PARP Inhibitors and Androgen Receptor Pathway Inhibitors in Metastatic Castration-Resistant Prostate Cancer
Orphan nuclear receptor TLX contributes to androgen insensitivity in castration-resistant prostate cancer via its repression of androgen receptor transcription
Clinical characteristics of patients with metastatic castration-resistant prostate cancer after treatment with combined androgen blockade
Patient-derived castration-resistant prostate cancer model revealed CTBP2 upregulation mediated by OCT1 and androgen receptor
Targeting molecular resistance in castration-resistant prostate cancer
Evolving Personalized Therapy for Castration-Resistant Prostate Cancer
Osteoclasts directly influence castration-resistant prostate cancer cells
Bipolar androgen therapy plus nivolumab for patients with metastatic castration-resistant prostate cancer: the COMBAT phase II trial
The natural compound atraric acid suppresses androgen-regulated neo-angiogenesis of castration-resistant prostate cancer through angiopoietin 2
Effect of simvastatin on castration-resistant prostate cancer cells