Regulation of senescence escape by the cdk4–EZH2–AP2M1 pathway in response to chemotherapy

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2041-4889

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Regulation of senescence escape by the cdk4–EZH2–AP2M1 pathway in response to chemotherapy ; volume:9 ; number:2 ; day:7 ; month:2 ; year:2018 ; pages:1-15 ; date:2.2018
Cell death & disease ; 9, Heft 2 (7.2.2018), 1-15, 2.2018

Creator: Duff, Mélanie Le

Contributor: Gouju, Julien
Jonchère, Barbara
Guillon, Jordan
Toutain, Bertrand
Boissard, Alice
Henry, Cécile
Guette, Catherine
Lelièvre, Eric
Coqueret, Olivier
SpringerLink (Online service)

DOI: 10.1038/s41419-017-0209-y

URN: urn:nbn:de:101:1-2018090802214265673933

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:26 AM CEST

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Associated

Duff, Mélanie Le
Gouju, Julien
Jonchère, Barbara
Guillon, Jordan
Toutain, Bertrand
Boissard, Alice
Henry, Cécile
Guette, Catherine
Lelièvre, Eric
Coqueret, Olivier
SpringerLink (Online service)

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Regulation of senescence escape by the cdk4–EZH2–AP2M1 pathway in response to chemotherapy

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Other Objects (12)

Regulation of senescence escape by TSP1 and CD47 following chemotherapy treatment

EZH2 inhibition sensitizes retinoic acid-driven senescence in synovial sarcoma

Combined inhibition of EZH2 and CDK4/6 perturbs endoplasmic reticulum-mitochondrial homeostasis and increases antitumor activity against glioblastoma

Cellular senescence in the response of HR+ breast cancer to radiotherapy and CDK4/6 inhibitors

PDLIM7 and CDH18 regulate the turnover of MDM2 during CDK4/6 inhibitor therapy-induced senescence

Regulation of CDK4

SIRT1 upregulation promotes epithelial-mesenchymal transition by inducing senescence escape in endometriosis

Loss of fragile WWOX gene leads to senescence escape and genome instability

Inhibition of EZH2 alleviates SAHA-induced senescence-associated secretion phenotype in small cell lung cancer cells

Immortalization of primary myoblasts from the DMD pig - hTERT/CDK4 prevent cellular senescence in primary porcine myoblasts

EZH2-H3K27me3-mediated silencing of mir-139-5p inhibits cellular senescence in hepatocellular carcinoma by activating TOP2A

EZH2 Protein Expression and Tumor Response to Neoadjuvant Chemotherapy in Locally Advanced Breast Cancer

Regulation of senescence escape by TSP1 and CD47 following chemotherapy treatment

EZH2 inhibition sensitizes retinoic acid-driven senescence in synovial sarcoma

Combined inhibition of EZH2 and CDK4/6 perturbs endoplasmic reticulum-mitochondrial homeostasis and increases antitumor activity against glioblastoma

Cellular senescence in the response of HR+ breast cancer to radiotherapy and CDK4/6 inhibitors

PDLIM7 and CDH18 regulate the turnover of MDM2 during CDK4/6 inhibitor therapy-induced senescence

Regulation of CDK4

SIRT1 upregulation promotes epithelial-mesenchymal transition by inducing senescence escape in endometriosis

Loss of fragile WWOX gene leads to senescence escape and genome instability

Inhibition of EZH2 alleviates SAHA-induced senescence-associated secretion phenotype in small cell lung cancer cells

Immortalization of primary myoblasts from the DMD pig - hTERT/CDK4 prevent cellular senescence in primary porcine myoblasts

EZH2-H3K27me3-mediated silencing of mir-139-5p inhibits cellular senescence in hepatocellular carcinoma by activating TOP2A

EZH2 Protein Expression and Tumor Response to Neoadjuvant Chemotherapy in Locally Advanced Breast Cancer

Regulation of senescence escape by TSP1 and CD47 following chemotherapy treatment

EZH2 inhibition sensitizes retinoic acid-driven senescence in synovial sarcoma

Combined inhibition of EZH2 and CDK4/6 perturbs endoplasmic reticulum-mitochondrial homeostasis and increases antitumor activity against glioblastoma

Cellular senescence in the response of HR+ breast cancer to radiotherapy and CDK4/6 inhibitors

PDLIM7 and CDH18 regulate the turnover of MDM2 during CDK4/6 inhibitor therapy-induced senescence

Regulation of CDK4

SIRT1 upregulation promotes epithelial-mesenchymal transition by inducing senescence escape in endometriosis

Loss of fragile WWOX gene leads to senescence escape and genome instability

Inhibition of EZH2 alleviates SAHA-induced senescence-associated secretion phenotype in small cell lung cancer cells

Immortalization of primary myoblasts from the DMD pig - hTERT/CDK4 prevent cellular senescence in primary porcine myoblasts

EZH2-H3K27me3-mediated silencing of mir-139-5p inhibits cellular senescence in hepatocellular carcinoma by activating TOP2A

EZH2 Protein Expression and Tumor Response to Neoadjuvant Chemotherapy in Locally Advanced Breast Cancer

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