PARP2 promotes Break Induced Replication-mediated telomere fragility in response to replication stress
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
-
PARP2 promotes Break Induced Replication-mediated telomere fragility in response to replication stress ; volume:15 ; number:1 ; day:2 ; month:4 ; year:2024 ; pages:1-18 ; date:12.2024
Nature Communications ; 15, Heft 1 (2.4.2024), 1-18, 12.2024
- Creator
-
Muoio, Daniela
Laspata, Natalie
Dannenberg, Rachel L.
Curry, Caroline
Darkoa-Larbi, Simone
Hedglin, Mark
Uttam, Shikhar
Fouquerel, Elise
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41467-024-47222-7
- URN
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urn:nbn:de:101:1-2406162120101.590874910673
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:48 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Muoio, Daniela
- Laspata, Natalie
- Dannenberg, Rachel L.
- Curry, Caroline
- Darkoa-Larbi, Simone
- Hedglin, Mark
- Uttam, Shikhar
- Fouquerel, Elise
- SpringerLink (Online service)
Other Objects (12)
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Deregulated DNA ADP-ribosylation impairs telomere replication
Break-induced replication promotes formation of lethal joint molecules dissolved by Srs2
Ssu72 phosphatase is a conserved telomere replication terminator
Identification of replication-dependent and replication-independent linker histone complexes: Tpr specifically promotes replication-dependent linker histone stability
Telomerase abrogates aneuploidy‐induced telomere replication stress, senescence and cell depletion
Alternative lengthening of human telomeres is a conservative DNA replication process with features of break‐induced replication
53BP1 deficiency leads to hyperrecombination using break-induced replication (BIR)
The bacterial replication origin BUS promotes nucleobase capture
Checkpoint effects and telomere amplification during DNA re-replication in fission yeast
Telomere-to-telomere human DNA replication timing profiles
PIF1 helicase promotes break‐induced replication in mammalian cells
Microcephalin 1/BRIT1-TRF2 interaction promotes telomere replication and repair, linking telomere dysfunction to primary microcephaly
Deregulated DNA ADP-ribosylation impairs telomere replication
Break-induced replication promotes formation of lethal joint molecules dissolved by Srs2
Ssu72 phosphatase is a conserved telomere replication terminator
Identification of replication-dependent and replication-independent linker histone complexes: Tpr specifically promotes replication-dependent linker histone stability
Telomerase abrogates aneuploidy‐induced telomere replication stress, senescence and cell depletion
Alternative lengthening of human telomeres is a conservative DNA replication process with features of break‐induced replication
53BP1 deficiency leads to hyperrecombination using break-induced replication (BIR)
The bacterial replication origin BUS promotes nucleobase capture
Checkpoint effects and telomere amplification during DNA re-replication in fission yeast
Telomere-to-telomere human DNA replication timing profiles
PIF1 helicase promotes break‐induced replication in mammalian cells
Microcephalin 1/BRIT1-TRF2 interaction promotes telomere replication and repair, linking telomere dysfunction to primary microcephaly
Deregulated DNA ADP-ribosylation impairs telomere replication
Break-induced replication promotes formation of lethal joint molecules dissolved by Srs2
Ssu72 phosphatase is a conserved telomere replication terminator
Identification of replication-dependent and replication-independent linker histone complexes: Tpr specifically promotes replication-dependent linker histone stability
Telomerase abrogates aneuploidy‐induced telomere replication stress, senescence and cell depletion
Alternative lengthening of human telomeres is a conservative DNA replication process with features of break‐induced replication
53BP1 deficiency leads to hyperrecombination using break-induced replication (BIR)
The bacterial replication origin BUS promotes nucleobase capture