Nitration of chemokine CXCL8 acts as a natural mechanism to limit acute inflammation
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1420-9071
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Nitration of chemokine CXCL8 acts as a natural mechanism to limit acute inflammation ; volume:80 ; number:1 ; day:9 ; month:1 ; year:2023 ; pages:1-14 ; date:1.2023
Cellular and molecular life sciences ; 80, Heft 1 (9.1.2023), 1-14, 1.2023
- Creator
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Thompson, Sarah
Pang, Chong Yun
Sepuru, Krishna Mohan
Cambier, Seppe
Hellyer, Thomas P.
Scott, Jonathan
Simpson, A. John
Proost, Paul
Kirby, John A.
Rajarathnam, Krishna
Sheerin, Neil S.
Ali, Simi
- Contributor
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SpringerLink (Online service)
- DOI
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10.1007/s00018-022-04663-x
- URN
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urn:nbn:de:101:1-2023033122072881223067
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 11:01 AM CEST
Data provider
This object is provided by:
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Associated
- Thompson, Sarah
- Pang, Chong Yun
- Sepuru, Krishna Mohan
- Cambier, Seppe
- Hellyer, Thomas P.
- Scott, Jonathan
- Simpson, A. John
- Proost, Paul
- Kirby, John A.
- Rajarathnam, Krishna
- Sheerin, Neil S.
- Ali, Simi
- SpringerLink (Online service)
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Designed CXCR4 mimic acts as a soluble chemokine receptor that blocks atherogenic inflammation by agonist-specific targeting
Heterocomplexes between the atypical chemokine MIF and the CXC-motif chemokine CXCL4L1 regulate inflammation and thrombus formation
G protein coupled receptors in inflammation - from kinin to chemokine
Epigenetic regulation of CC-chemokine ligand 2 in nonresolving inflammation
Methylmercury toxic mechanism related to protein degradation and chemokine transcription
P23 acts as functional RBP in the macrophage inflammation response
P23 Acts as Functional RBP in the Macrophage Inflammation Response
CB1 receptors on keratinocytes modulate proinflammatory chemokine secretion and attenuate contact allergic inflammation
The chemokine receptor CCR10 promotes inflammation-driven hepatocarcinogenesis via PI3K/Akt pathway activation
Soluble epoxide hydrolase limits mechanical hyperalgesia during inflammation
IL-10 constrains sphingolipid metabolism to limit inflammation
Tax1BP1 limits hepatic inflammation and reduces experimental hepatocarcinogenesis
Designed CXCR4 mimic acts as a soluble chemokine receptor that blocks atherogenic inflammation by agonist-specific targeting
Heterocomplexes between the atypical chemokine MIF and the CXC-motif chemokine CXCL4L1 regulate inflammation and thrombus formation
G protein coupled receptors in inflammation - from kinin to chemokine
Epigenetic regulation of CC-chemokine ligand 2 in nonresolving inflammation
Methylmercury toxic mechanism related to protein degradation and chemokine transcription
P23 acts as functional RBP in the macrophage inflammation response
P23 Acts as Functional RBP in the Macrophage Inflammation Response
CB1 receptors on keratinocytes modulate proinflammatory chemokine secretion and attenuate contact allergic inflammation
The chemokine receptor CCR10 promotes inflammation-driven hepatocarcinogenesis via PI3K/Akt pathway activation
Soluble epoxide hydrolase limits mechanical hyperalgesia during inflammation
IL-10 constrains sphingolipid metabolism to limit inflammation