Functional networks are impaired by elevated tau-protein but reversible in a regulatable Alzheimer’s disease mouse model
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1750-1326
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Functional networks are impaired by elevated tau-protein but reversible in a regulatable Alzheimer’s disease mouse model ; volume:14 ; number:1 ; day:27 ; month:3 ; year:2019 ; pages:1-13 ; date:12.2019
Molecular neurodegeneration ; 14, Heft 1 (27.3.2019), 1-13, 12.2019
- Urheber
-
Green, Claudia
Sydow, Astrid
Vogel, Stefanie
Anglada-Huguet, Marta
Wiedermann, Dirk
Mandelkow, Eckhard
Mandelkow, Eva-Maria
Hoehn, Mathias
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s13024-019-0316-6
- URN
-
urn:nbn:de:101:1-2019041823114228369537
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:59 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Green, Claudia
- Sydow, Astrid
- Vogel, Stefanie
- Anglada-Huguet, Marta
- Wiedermann, Dirk
- Mandelkow, Eckhard
- Mandelkow, Eva-Maria
- Hoehn, Mathias
- SpringerLink (Online service)
Ähnliche Objekte (12)
P53 aggregation, interactions with tau, and impaired DNA damage response in Alzheimer’s disease
Tau Biology and Tau-Directed Therapies for Alzheimer’s Disease
Elevated CSF GAP-43 is associated with accelerated tau accumulation and spread in Alzheimer’s disease
Emotional prosody recognition is impaired in Alzheimer’s disease
The UFMylation pathway is impaired in Alzheimer’s disease
Inhibiting tau-induced elevated nSMase2 activity and ceramides is therapeutic in an Alzheimer’s disease mouse model
Neurons derived from sporadic Alzheimer’s disease iPSCs reveal elevated TAU hyperphosphorylation, increased amyloid levels, and GSK3B activation
Tau-focused therapy and tau transmission: Implications for Alzheimer’s Disease and related tauopathies
Plasma N-terminal containing tau fragments (NTA-tau): a biomarker of tau deposition in Alzheimer’s Disease
APOE effects on regional tau in preclinical Alzheimer’s disease
Specific serum antibody binding to phosphorylated and non-phosphorylated tau in non-cognitively impaired, mildly cognitively impaired, and Alzheimer’s disease subjects: an exploratory study
Impaired Functional Criticality of Human Brain during Alzheimer’s Disease Progression
P53 aggregation, interactions with tau, and impaired DNA damage response in Alzheimer’s disease
Tau Biology and Tau-Directed Therapies for Alzheimer’s Disease
Elevated CSF GAP-43 is associated with accelerated tau accumulation and spread in Alzheimer’s disease
Emotional prosody recognition is impaired in Alzheimer’s disease
The UFMylation pathway is impaired in Alzheimer’s disease
Inhibiting tau-induced elevated nSMase2 activity and ceramides is therapeutic in an Alzheimer’s disease mouse model
Neurons derived from sporadic Alzheimer’s disease iPSCs reveal elevated TAU hyperphosphorylation, increased amyloid levels, and GSK3B activation
Tau-focused therapy and tau transmission: Implications for Alzheimer’s Disease and related tauopathies
Plasma N-terminal containing tau fragments (NTA-tau): a biomarker of tau deposition in Alzheimer’s Disease
APOE effects on regional tau in preclinical Alzheimer’s disease
Specific serum antibody binding to phosphorylated and non-phosphorylated tau in non-cognitively impaired, mildly cognitively impaired, and Alzheimer’s disease subjects: an exploratory study
Impaired Functional Criticality of Human Brain during Alzheimer’s Disease Progression
P53 aggregation, interactions with tau, and impaired DNA damage response in Alzheimer’s disease
Tau Biology and Tau-Directed Therapies for Alzheimer’s Disease
Elevated CSF GAP-43 is associated with accelerated tau accumulation and spread in Alzheimer’s disease
Emotional prosody recognition is impaired in Alzheimer’s disease
The UFMylation pathway is impaired in Alzheimer’s disease
Inhibiting tau-induced elevated nSMase2 activity and ceramides is therapeutic in an Alzheimer’s disease mouse model
Neurons derived from sporadic Alzheimer’s disease iPSCs reveal elevated TAU hyperphosphorylation, increased amyloid levels, and GSK3B activation
Tau-focused therapy and tau transmission: Implications for Alzheimer’s Disease and related tauopathies
Plasma N-terminal containing tau fragments (NTA-tau): a biomarker of tau deposition in Alzheimer’s Disease
APOE effects on regional tau in preclinical Alzheimer’s disease
Specific serum antibody binding to phosphorylated and non-phosphorylated tau in non-cognitively impaired, mildly cognitively impaired, and Alzheimer’s disease subjects: an exploratory study